Cardiovascular magnetic resonance-derived left ventricular
hemodynamic force among patients with systemic lupus erythematosus
Yangzhen Hou1,2, Hui Zhou1, Jing Luo1, Ji Yang1, Huiting Zhang3, and Xiaoming Bi4 1Department of Radiology, Xiangya Hospital Central South University, Changsha, Hunan, China., Changsha, China, 2Department of Radiology, Xiangya Hospital Central South University, Changsha, Hunan,, China., Changsha, China, 3Scientific Marketing, Siemens Healthineers Ltd., Wuhan, China, Wuhan, China, 4MR Collabration, Siemens Healthineers, Los Angeles, USA, Los Angeles, CA, United States
Synopsis
Keywords: Heart Failure, Cardiovascular
Motivation: Cardiovascular involvement in systemic lupus erythematosus (SLE) patients is common and serious; early and accurate identification of the cardiac dysfunction is crucial.
Goal(s): Our goal was to investigate the potential role of hemodynamic force (HDF) in early recognizing impaired cardiac function in SLE.
Approach: Left ventricular (LV)HDF and strain analysis were performed based on long-axis cine imaging using feature-tracking in 36 SLE patients and 34 healthy volunteers.
Results: The force ratio between transverse and longitudinal direction was larger in SLE patients with no difference of strain analysis, indicating that the orientation of HDF altered in LV.
Impact: This
study demonstrated that HDF analysis can early detect systolic dysfunction in SLE in
advance of significant decrease of LV EF, which may contribute to the early
diagnosis of cardiac involvement and prognosis improvement of patients.
INTRODUCTION
Cardiovascular
disease is a major cause of morbidity/mortality in patients with systemic lupus erythematosus (SLE)1. Since left ventricular (LV) ejection fraction (EF) can be preserved in the early
stages of SLE, LV dysfunction should be assessed with more sensitive
techniques. LV strain analysis and hemodynamic force (HDF) may be valuable in detecting subtle changes in
LV mechanics; the value of HDF in SLE is unknown. Therefore, the aim of our
study is to evaluate LV strain and HDF from cardiovascular magnetic resonance
(CMR) cines in SLE patients.
METHODS
In
this retrospective study, 36 SLE patients (20 patients with preserved EF (pEF)
who were defined as LV EF ≥ 55%) and 34 healthy volunteers underwent CMR
imaging. LV HDF was computed from a formulation with myocardial movement and
velocity of the reconstructed 3D-LV (derived from long-axis cines using feature-tracking)2. To facilitate comparison between patients with
different LV sizes, HDF is converted to percentage numbers by dividing the
blood density and the acceleration of gravity. HDF assessment included the
“apical-basal” (longitudinal) and “lateral-septal” (transverse) forces during
systole and diastole, respectively, “apical-basal” force during the entire
heartbeat and the systolic peak. The force ratio was calculated between
transverse force and longitudinal force. LV global longitudinal strain (GLS) and global circumferential strain
(GCS) were also measured using CMR feature tracking technique.
RESULTS
Compared to healthy volunteers, the
force ratio during systole was higher in SLE patients (0.19±0.08% vs. 0.13±0.04%,
P< 0.001); and the systolic function was impaired depicted by (i) lower LV EF
(48.50±14.43% vs. 61.74±5.69%, P< 0.001) and (ii) lower systolic force (17.71±7.46%
vs. 23.13±5.64%, P< 0.01) and systolic
peak (33.98±14.89% vs. 44.62±10.76%, P< 0.01) and (iii) lower GCS (17.97±5.92%
vs. 22.79±3.30%, P< 0.001) and GLS (18.29±6.09% vs. 22.43±3.03%, P< 0.01).
In pEF SLE patients, the force ratio during systole was still higher (0.19±0.08%
vs. 0.13±0.04%, P< 0.001) while no significant difference in strain analysis
was found.
DISCUSSION
HDF
is the global force of cardiac motion acquired by integrating the
intraventricular pressure gradient over the entire ventricle and thus may
detect subtle heart injury before a significant decline of EF. In the spatial
reference system, HDF is decomposed into three inter-perpendicular vectors in
the left ventricle3. As the longitudinal force is nearly consistent
with the blood flow during ejection, the force ratio can represent the extent of
HDF deviating from the functional direction. In this study, we demonstrated
that the LV HDF orientation was altered in pEF SLE patients when there was no
obvious abnormality of LV strain analysis.
CONCLUSION
LV
systolic function impairment can be demonstrated by the alteration of HDF ratio
in pEF SLE patients, HDF could therefore potentially be used as an early marker
for LV functional assessment.
Acknowledgements
none
References
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Figures
The time curve of hemodynamic force in apical-basal
and lateral-septal directions.
Comparison of LV EF, strain and hemodynamic force parameters in SLE patients and healthy controls.
Quantification of left ventricular hemodynamic force
using cine CMR