Chia-Chi Yang1, Archana Malagi1, Yuheng Huang2,3, Ghazal Yoosefian2, Xinheng Zhang2,3, Xinming Guan2, Anthony Christodoulou1,4, Debiao Li1, Hui Han5, Rohan Dharmakumar2, and Hsin-Jung Yang1
1Biomedical Imaging Research Institude, Cedars-Sinai Medical Center, Los Angeles, CA, United States, 2krannert cardiovascular research center, Indiana University School of Medicine, Indianapolis, IN, United States, 3Bioengineering, UCLA, Los Angeles, CA, United States, 4Department of Radiological Sciences, David Geffen School of Medicine, UCLA, Los Angeles, CA, United States, 5Radiology, Weill Cornell Medicine, New York, NY, United States
Synopsis
Keywords: Heart Failure, Heart, myocardial volume oxygenation consumption
Motivation: Whole-heart myocardial oxygen consumption (MVO2) is the central factor that determines cardiac function and is a sign of heart diseases.
Goal(s): We proposed a high-resolution, free breathing, cardiac-phase resolved sequence to quantify MVO2 in the beating hearts.
Approach: Healthy pigs were scanned at 3T. Coronary sinus images were acquired with a continuous, free-breathing, Radial T2Prep-IR sequence with flow compensation and water excitation and a 2D phase contrast sequence to quantify the MVO2. Invasive ground truth was also measured to verify the accuracy of our estimation.
Results: The proposed method measured comparable SbO2,OEF,MBF and MVO2 values to the invasive ground truth.
Impact: The proposed
free-breathing, motion-resolved cardiac MR Oximetry technique has the potential
to non-invasively measure accurate myocardial
oxygen consumption without using ionizing radiation and exogenous contrast agents.
Background
Cardiac energy consumption is the central factor
that determines cardiac function and is the hallmark sign of many heart
diseases(e.g., heart failure and different forms of cardiomyopathy). The gold
standard for staging cardiac energetic changes is based on whole-heart
myocardial volume oxygen consumption (MvO2)1 measured by invasive
catheterization. A noninvasive MvO2
assessment is critical for the management of the aforementioned chronic
diseases and for improving patient outcomes. Because more than 90% of the coronary
blood is drained through the coronary sinus (CS), MR oximetry and flowmetry of
the CS blood has the potential to non-invasively quantify MvO2
without ionizing radiation and exogenous contrast agent.(Fig. 1) While CS flow
measurements have been established in clinical studies using phase contrast MRI2,
current CMR oximetry techniques are not suitable for imaging the CS blood due
to several limitations, including i) insufficient image resolution and spatial
determination of the CS due to its collapsed anatomical footprint during
typical diastolic acquisitions (the CS collapses by over 50% during diastole), ii)
unreliable image quality from the rapid CS movement throughout the respiratory
and cardiac cycles, and iii) inaccurate measurements of MR Oximetry due to
blood oxygenation related artifacts(e.g., B0, B1, and flow) 2. To overcome these challenges, we developed a
high-resolution, cardiac-phase resolved, and confounder-mitigated MR oximetry
sequence in the presence of respiratory motion to quantify MvO2 in
the beating hearts. We tested the technique in healthy pigs and validated it
against invasive catheterized measurements.Methods
Under institutional approval, healthy pigs (N=3) were studied with a 3T
clinical scanner (Biograph mMR, Siemens) and validated against invasive
catheterization measurements. During image acquisition, a continuous Radial GRE
Prep-IR sequence with flow compensation and
water excitation(Fig. 2B) was prescribed at the heart’s mid-slice and the cross-section
of the CS to acquire images of the arterial and venous blood. (TE/TR=3.3/5.8ms, FA=5°, FOV=270mm, Voxel=1.3*1.3*2.7mm3,
BW=1093 Hz/pixel, T2prep duration = 0,30,60,90,110,120ms). The images were then reconstructed by a blood oxygenation3 formulated Low-Rank Tensor (LRT) model4 to generate
motion-resolved blood oxygen saturation(SbO2) maps(Fig. 2C and D). Details
of the sequence diagram, image reconstruction framework and blood oxygenation
model are illustrated in Fig. 2. Notably, a real-time signal can be
reconstructed from the continuously acquired sequence and be used to determine
the initial parameters for the L-M model. HCT is measured with blood drawn
right before the scan. For MBF derivation, a 2D phase contrast sequence was
also used at the CS to attain its blood flow velocity(TE/TR=2.9/41.2ms,FA=20°,FOV=270mm,Voxel=1.3*1.3*6mm3,Bandwidth=445Hz/pixel,
). MBF was calculated using Cvi42 and The of the hearts were
estimated. Following the CMR studies, animals were sent to the Cath lab to
measure the invasive ground truth of the LV, RV, CS blood oxygen saturation
level, and MBF.Results
Representative systolic oximetry images of a healthy pig are shown in
Fig. 3. The signal progressions of LV, RV and CS blood and the corresponding apparent
T2 measured under different refocusing time(t180) are reported in Fig
3B-C. Significant difference in T2 signal progression is shown between the
arterial and venous blood. Fig.3D shows
the maps derived from the voxel-wise L-M model
fitting. Significantly lower SbO2 is
measured in the CS compared to the other heart chambers. Quantitative
measurements of , OEF, MBF and from all
subjects were compared to the invasive ground truth in Fig. 5. For the CMR estimation
shown in panel (A), the proposed approach shows comparable means to the ground
truth at LV, RV and CS. (LV est. = 93.7±1.9%, LV meas. = 96.3±2.5%, RV est. =
75.4±3.8%, RV meas. =78.5±4.8 %, CS est. = 48.6±2.9%, CS meas. = 50.1±12.6%).
The difference of means between estimations in LV, RV and CS are also
significant. (all p<0.05)The CMR OEF (B), MBF(C) and MvO2(D) also
showed comparable values to the invasive ground truth. (OEF est. = 10.3±1.0%,
OEF meas. = 10.6±3.4%, OEF: p = 0.89, MBF est. = 228.5±84.3ml/min, MBF meas. =
183.3±57.7ml/min, MBF: p = 0.11, est. = 23.6±8.5ml/min,
meas. =
18.7±4.9ml/min, : p = 0.29)Conclusion and Future Work
In this study, we developed a
non-invasive, free breathing technique to quantify and assess the cardiac metabolism of the whole
heart. Based on the current results, the
proposed method has the potential to accurately quantify myocardial oxygen consumption.
Further improvement can be made by involving T1 in HCT calibration5 instead of using invasively measurement to avoid bias from the equipment. The
next step is to test the technique’s feasibility on subjects with cardiac diseases
and human subjects.Acknowledgements
This work is supported by 1R01HL136578; 1R01HL165211; 1R01HL148788; 1R01HL156818.References
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