Pragalv Karki1, Matthew C Murphy1, Petrice M Cogswell1, Matthew L Senjem1, Jonathan Graff-Radford2, Clifford R Jack Jr1, Richard L Ehman1, and John Huston III1
1Department of Radiology, Mayo Clinic, Rochester, MN, United States, 2Department of Neurology, Mayo Clinic, Rochester, MN, United States
Synopsis
Keywords: Dementia, Brain, Normal pressure hydrocephalus
Normal
pressure hydrocephalus (NPH) is a brain disorder that is often misdiagnosed as
Alzheimer’s or Parkinson’s disease due to overlapping symptoms. However, unlike
such neurodegenerative diseases, NPH can be surgically treated by shunt
placement with a success rate of around 80%, depending on phenotype. Therefore,
correct diagnosis and categorization of NPH is important. As a step towards
that goal, we demonstrate that analyzing the
different morphologic phenotypes of NPH using magnetic resonance elastography can help establish unique viscoelastic signatures associated with
each phenotype providing distinguishing biomarkers.
INTRODUCTION
Normal pressure hydrocephalus (NPH) is a brain pathology
with symptoms of cognitive decline, gait disturbance, and urinary incontinence with
an estimated prevalence of 8.9%1 in the elderly population of age 80
and above. It is often misdiagnosed as Alzheimer’s2 or Parkinson’s3 due to overlapping symptoms. But unlike such degenerative diseases, NPH is
treatable with ventriculoperitoneal shunt placement with sustained improvement
in 75 - 91%4,5 of cases. Due to the invasive nature of the surgery, accurate
screening is important. Enlargement of ventricles in NPH can reflect mechanical
processes that affect the viscoelastic properties of adjacent brain tissue.
Therefore, magnetic resonance elastography (MRE), an MRI based non-invasive technique
that can measure viscoelastic properties of tissues, is a logical tool for
investigating NPH-related effects. A prior study demonstrated that MRE can
distinguish disease specific patterns of NPH compared to control and
Alzheimer’s disease6. However,
patients with NPH may have different morphologic phenotypes that can be
assessed by MRI. With the potential
implication to improve the diagnosis and prediction of shunt effectiveness in
NPH, the hypothesis of this study is that each NPH
phenotype is associated with a unique mechanical property signature.METHODS
An expert neuroradiologist categorized
the patients with suspected NPH into four groups based on their morphologic
features: high convexity tight sulci (HCTS7), ventriculomegaly only
(Ventric, Evan’s Index, EI>0.3), neither ventriculomegaly (EI<0.3) or
HCTS, and congenital hydrocephalus (ventriculomegaly, diffusely narrowed
cerebral sulci and aqueductal stenosis or web8). There were 158
participants in total, with 44 in the Control group, 83 in HCTS, 18 in Ventric,
8 in Congenital, and 5 in Neither. MRE scans were performed on 3T MR scanners
using a spin-echo EPI pulse sequence. MRE displacement data were inverted using
a neural network inversion9,10,11 to obtain the viscoelastic
property maps, stiffness and damping ratio. Groups were compared by fitting a
linear model at each voxel with model predictors including age, sex, scanner,
and a categorical variable for group assignment. A voxel-wise false discovery
rate corrected Q-value less than 0.05 was considered significant. HCTS pattern
scores were computed using leave-one-out cross validation as previously
described6.RESULTS
Group-wise mean stiffness maps show distinct patterns as shown in right
panel of Figure 1. On the left of Figure 1, a boxplot is shown with a jitter
plot of averaged stiffness values for each individual. Welch’s t-test and
Wilcoxon rank sum test for stiffness between the groups show significant
difference between the HCTS and the Control group (p<0.05). Damping ratio maps of the groups with the accompanying boxplots and
jitter plots are shown in Figure 2. For the Welch’s t-test and the Wilcoxon
rank sum test, the Ventric and the HCTS groups have significantly lower damping
ratio values compared to the Control group with p<0.001. HCTS and Congenital groups have lower damping ratio compared to the
Neither and the Control group respectively, with p<0.05. Figures 3
and 4 show difference and thresholded t-statistic maps (q<0.05) between the HCTS and each of the other groups.
The number of significant voxels is reported in the respective figures. Leave-one-out
cross validated correlation scores were calculated for all the cases with HCTS
group as the reference. Correlation scores were calculated separately for the
stiffness and the damping ratio. The pattern analysis plot with damping ratio
scores in y-axis and stiffness scores in x-axis is shown in Figure 5. DISCUSSION
Damping ratio showed more distinction between groups than stiffness for
both voxel-wise patterns and global values. The only exception was the HCTS versus
congenital comparison, where stiffness had 493 voxels while the damping ratio
had none. Both viscoelastic properties show more significant voxels at the
vertex. The pattern-analysis plot in Figure 5 shows distinct separation of the
HCTS and control cases. Congenital cases are clustered towards the HCTS group as
expected from the similarity in their viscoelastic property maps. Cases from Neither
and Ventric groups tend to lie between HCTS and control groups with a few
Ventric cases closer to the HCTS cluster. CONCLUSION
Disproportionately enlarged subarachnoid-space hydrocephalus (HCTS with
enlarged sylvian fissures) is a key feature for diagnosing NPH under the widely established Japanese
criteria12. In a multi-regression analysis study, HCTS was shown to
have highest correlation with shunt surgery success13. But studies
have highlighted that relying solely on tight high convexity for predicting shunt
outcome14,15 would exclude cases from other phenotypes of NPH that
could also benefit from surgery. Therefore, it is important to establish
distinct biomarker-based signatures for different morphologic phenotypes of
NPH. In this study, we have demonstrated that MRE can provide such a biomarker.
Furthermore, MRE-based
pattern analysis separates some ventriculomegaly
cases into HCTS spectrum
or cluster, signaling that viscoelastic signatures could predict development of
HCTS in those cases. The result provides motivation for further MRE-based studies
to better understand the underlying viscoelastic biomarker findings in such cases.Acknowledgements
No acknowledgement found.References
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