Ajin Joy1, Manoj K Sarma1, Jhelum Paul1, Andres Saucedo1, Paul M Macey2, Dieter J Meyerhoff3, Ebrahim Haroon4, Margaret A Keller5, and M Albert Thomas1
1Radiological Sciences, University of California-Los Angeles, Los Angeles, CA, United States, 2School of Nursing and Brain Research Institute, University of California-Los Angeles, Los Angeles, CA, United States, 3Radiology and Biomedical Imaging, University of California-San Francisco, San Francisco, CA, United States, 4Psychiatry and Behavioral Sciences, Emory University, Atlanta, GA, United States, 5Pediatrics, The Lundquist Institute, Harbor-UCLA Medical Center, Torrance, CA, United States
Synopsis
The human immunodeficiency virus (HIV) can affect the morphometry of the
developing brain of perinatally HIV (PHIV)-infected youths. Surface-based
morphometric analysis can reveal if local effects on gray and white matter volumes
is significant in the regions with alterations in cortical thickness (CT), gyrification
index (GI), or sulcal depth (SD). So, we compared these morphometric parameters
of 19 PHIV-youths on combination antiretroviral therapy with those in 26
uninfected healthy controls. PHIV-youths had altered gray and white matter regional
volume, CT, GI, and SD, partly in overlapping regions. The findings survived multiple
comparisons using both false discovery rate and Holm-Bonferroni corrections.
Introduction
The success of combined antiretroviral therapy (cART) has reduced the
risk of major neurological complications in perinatally HIV (PHIV)-infected
youth, thereby significantly improving life expectancy (1-2). However,
HIV-associated neurocognitive disorders (HAND) continue to be widely prevalent
among PHIV-youths (3-5). Previous publications have reported volumetric and
surface area changes in different brain regions of PHIV-youths compared with
healthy controls (HC) (6-11). However, the reported high variability in these structural
group differences indicate that the reported information is inconclusive and
further studies may lead to a better understanding of the morphometric alterations
in the brain of PHIV-youths (12). The variability in reports can be attributed
to different factors, including but not limited to differences in socioeconomic
status, health conditions, and medical comorbidities like psychiatric disorders
or drug use (12). The purpose of this study is to use cutting-edge structural
MRI data to measure regional grey matter (GM) and white matter (WM) volumes, as
well as to determine the variations in cortical thickness (CT), gyrification
index (GI), and sulcal depth (SD) in a group of PHIV-youths and HIV-negative
controls selected with an active psychiatric diagnosis based exclusion
criteria.Materials and Methods
We investigated nineteen PHIV-youths
(mean age:18.7±3.9yrs, range 14–29yrs, m/f 8/11) and twenty-six HC (mean age:19.6±3.7yrs,
range 13-28, m/f 14/12). From PHIV-youths, we obtained age at first treatment,
HIV viral load, highest viral load, current antiretroviral therapy (yes/no),
and presence/absence of HIV encephalopathy.
We excluded volunteers with current alcohol or other substance
use/abuse, current or past attention deficit disorder, active depression or
other psychiatric diagnoses, metabolic disturbances, metallic implants, claustrophobia,
pregnancy, and non-HIV-related brain diseases. All MRI studies were performed
on a Siemens 3T Prisma MRI scanner using a 16-channel phased-array head
'receive' coil. High-resolution T1-weighted images were acquired using an
MPRAGE sequence with TR/TE=2200/2.41ms, inversion time=900ms, flip angle=9°,
matrix size=320×320, FOV=230×256mm2, slice thickness=0.9 mm, 192 slices.
Voxel-based and
surface-based morphometric analyses were performed using the MATLAB
Computational Anatomy Toolbox (CAT12.7(1700), http://dbm.neuro.uni-jena.de/cat/), which is an
extension of SPM12 (https://www.fil.ion.ucl.ac.uk/spm/software/spm12/) (13-14). An
independent 2-sample t-test was used to compare PHIV-youths with controls on
imaging outcome measures. A p-value<0.001 was used for GM and WM volume
measures in voxel-based morphometry to limit type 1 errors. We used a p<0.05
threshold for region-of-interest analysis (surface measurements) with
Holm-Bonferroni and false discovery rate (FDR) corrections. Age and gender were included as covariates in
the analysis of CT, while total intracranial volume (TIV), age, and gender were
used to analyze GM and WM volumes, GI, and SD.Results
Significant differences were observed in PHIV-youths compared to HC
despite the demographic similarities and cART. Fig 1a shows the regions with larger
GM volume in PHIV-youths compared to HC. Whereas we identified no regions with smaller
GM in the PHIV-youths, we noted smaller WM volume in regions demonstrated in
Fig 1b. Fig 2a and b show regions identified with cortical thickening and
thinning, respectively, and Fig 2c and d show regions with greater and smaller
GI. Fig 3a and b shows regions with lower SD after FDR and Holm-Bonferroni
corrections, respectively. No regions with greater SD were identified. Labels for
the regions shown in Figs 1-4 are shown in Table 1. Finally, Fig 4 shows plots
of GM and WM volumes versus age. We detected significant negative correlations of
age with total GM volume in both HC (P = 0.004) and PHIV-youths (P = 0.017), which was also significant
in females but not males. This GM decline with age appeared accelerated in PHIV-youths
compared to HC (cf. Fig 4a).Discussion
Our results demonstrate larger GM and smaller WM volumes in PHIV-youths compared
to healthy controls, which is consistent with an earlier study on PHIV-youths (7).
According to (15), these larger GM volumes could be related to cortical thickening
and/or less cortical folding (lower GI). Earlier reports suggested that cortical
thickening in PHIV-youths may be due to abnormal developmental trajectories in
the presence of HIV and cART, while cortical thinning may be due to neuronal
and glial cell injury resulting from HIV (7,16-17). Our results suggest an
association between the GM volume alterations and cortical thickening in the
temporal gyrus of PHIV-youths (cf. Figs 1 and 3 and Table 1).
Furthermore, the cortical thickening and GI loss in the insula region suggests an
association between these abnormalities as well. (cf. Figs 3-4 and Table
1). After FDR corrections, we also found lower SD in the precuneus, isthmus
cingulate, and paracentral regions in PHIV-youths versus HC. Finally,
PHIV-youths tended to display greater age-related GM volume loss than HC.Conclusion
We confirmed key volumetric differences in GM and WM volumes between PHIV-youths
and HC, and we found more specific regional alterations in CT, GI, and depth
related to PHIV in regions overlapping with the observed volumetric alterations.
In addition, we found regions with smaller GM volumes and GI overlapping with
regions of greater cortical thickness. We also identified regions of lower SD in
PHIVY than HC after correction for multiple comparisons. Taken together, these
findings suggest that a combination of voxel-based and surface-based analyses
sheds more light on the specific morphometric alterations in the PHIV adolescent
brain that may be related to the widely prevalent HAND observed in PHIV.Acknowledgements
This research was funded by the NIH grants: 5R21NS090956-02 and
5R21MH125349-02.References
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