Kiyohisa Kamimura1, Masanori Nakajo1, Bohara Manisha1, Yoshihiko Fukukura1, Hiroyuki Uchida2, Takashi Iwanaga3, Thorsten Feiweier4, Hiroshi Imai5, and Takashi Yoshiura1
1Radiology, Kagoshima University, Kagoshima, Japan, 2Neurosurgery, Kagoshima University, Kagoshima, Japan, 3Radiological Technology, Kagoshima University Hospital, Kagoshima, Japan, 4Siemens Healthcare GmbH, Erlangen, Germany, 5Siemens Healthcare K.K., Tokyo, Japan
Synopsis
To determine whether time-dependent DWI parameters
and microscopic diffusion anisotropy (μFA) are useful for differentiating between
glioblastoma and primary central nervous system lymphoma (PCNSL), 51 patients
with glioblastoma and 13 with PCNSL were examined. In addition to ADC at two
different diffusion times, ADC difference (ΔADC)
and ADC change ratio (rcADC) were significantly different between the two tumor
types, while no difference was shown for μFA. rcADC showed the best diagnostic
performance followed by ΔADC.
INTRODUCTION
Preoperative differentiation between
glioblastoma and primary central nervous system lymphoma (PCNSL)
is problematic as they often show similar findings on conventional MR images. It
has been well documented that lower ADC on diffusion-weighted imaging (DWI) helps diagnose PCNSL which is
characterized by high cell density. Recently, more advanced DWI techniques have
become available. Oscillating gradient spin-echo (OGSE) sequences1 allow for DWI with a short diffusion time,
enabling time-dependent diffusion analysis which provides specific information
regarding restricted diffusion. Double-diffusion-encoding (DDE) pulse sequences encode diffusion twice, along two directions, and enable quantification of
microscopic fractional anisotropy (μFA) independent of orientation dispersion.2 Previous studies have suggested that μFA
provides information regarding tumor cell morphology.3
Our purpose for this study was to determine whether these advanced DWI techniques are useful for
differentiating between glioblastoma and PCNSL.METHODS
A retrospective study was performed on
data acquired for 51 patients with glioblastoma (mean age, 68.3 ± 12.8 years)
and 13 patients with PCNSL (mean age, 70.4 ± 10.7 years). All patients
underwent preoperative MR imaging using a 3T system (MAGNETOM Prisma; Siemens
Healthcare, Erlangen, Germany) with a 20-channel head/neck coil. Time-dependent
DW images were acquired using a prototype DWI sequences with OGSE1 and pulsed gradient
preparation (PGSE) (effective diffusion time [Δeff]: 8.5 ms and 44.5
ms, respectively) at b-values of 0 and 1500 s/mm2. ADC difference maps were generated to estimate the difference in ADC values between OGSE and PGSE sequences: ΔADC = ADC8.5ms - ADC44.5ms. ADC relative change maps were generated to estimate the ratio of
change in ADC values between OGSE and PGSE sequences: rcADC = (ADC8.5ms
- ADC44.5ms)/ADC44.5ms × 100 (%). μFA images were extracted from data acquired using a prototype single-shot
echo planar DWI sequence with DDE. For the motion probing gradient directions, the double pulsed
field diffusion gradient (d-PFG) 5-design (Fig 1)
was used.4 The number of directions
for b = 1000 s/mm2 was 72, including 12 parallel and 60 orthogonal
combinations. The μFA was calculated according to the equation of Yang G et al.5 Instead of obtaining an additional standard diffusion
tensor imaging dataset, we determined the ADC and fractional anisotropy (FA)
using b = 0 s/mm2 and a part of b = 1000 s/mm2 of the
d-PFG dataset corresponding to the parallel wave vectors. The mean values of ADC44.5ms,
ADC8.5ms, ΔADC,
rcADC, FA, and μFA in the enhancing tumor were compared between glioblastomas
and PCNSLs using the Mann-Whitney U test. In addition, the diagnostic
performances of the parameters were evaluated using receiver operating characteristic (ROC) curve analysis. The
area under ROC curve (AUC) was compared using DeLong’s method. P < 0.05 was
considered as statistically significant.RESULTS
Representative cases and statistical results
are shown in Fig 2 and Figs
3-5, respectively. The ADC44.5ms and ADC8.5ms (x10-3
mm2/s) of PCNSLs were significantly lower than those of glioblastomas
(0.960 ± 0.197 vs. 1.144 ± 0.293; P = 0.0016, 1.081 ± 0.182 vs. 1.305 ± 0.285;
P = 0.0091). The ΔADC (x10-3 mm2/s) and rcADC (%) of PCNSLs
was significantly higher than those of glioblastomas (0.222 ± 0.036 vs. 0.163 ±
0.037; P < 0.0001, 32.6 ± 18.0 vs. 17.0 ± 8.6; P < 0.0001). There was no
significant difference for FA and μFA between PCNSLs and glioblastomas (0.203 ±
0.151 vs. 0.138 ± 0.048; P = 0.2409, 0.390 ± 0.141 vs. 0.372 ± 0.142; P = 0.6118).
The ROC curve analysis showed significance for ADC44.5ms, ADC8.5ms,
ΔADC and rcADC (AUC = 0.778, 0.773, 0.887, 0.888; P <0.0001, 0.0003, <0.0001,
<0.0001; respectively), but not for FA and μFA (AUC = 0.607, 0.547; P = 0.2855, 0.6123; respectively). The AUC for rcADC was significantly greater than that for ADC44.5ms,
ADC8.5ms, FA and μFA (P = 0.020, 0.009, 0.006, 0.004; respectively), but not than that for ΔADC (P = 0.961).
The AUC for ΔADC was not significantly different
from that for ADC44.5ms (P = 0.071).DISCUSSION
The lower ADC44.5ms and ADC8.5ms
in PCNSLs than those in glioblastomas are consistent with previous reports,6, 7 and
likely reflect higher cell density in PCNSLs. The higher ΔADC and rcADC in the PCNSLs
than those in glioblastomas indicates that restricted diffusion is more
dominant in PCNSLs than in glioblastomas presumably due to abundant cell
membrane of densely packed tumor cells in PCNSLs, which may result in stronger
diffusion time dependency in ADC. The diagnostic performance of rcADC in
differentiating between glioblastomas and PCNSLs was the highest with a
significantly higher AUC than those of ADC44.5ms and ADC8.5ms,
while the AUCs for rcADC and ΔADC were similar. These observations suggest that rcADC
and ΔADC are useful DWI markers for distinguishing glioblastomas and PCNSLs. Lack
of significant difference in μFA between PCNSLs and glioblastomas was somewhat
unexpected. Difference in cell morphology between the two tumor types (irregular
and pleomorphic cells in glioblastomas vs. homogeneously round cells in PCNSLs)
was not reflected in μFA. Possible explanation for this negative result is relative
preservation of background neuronal fibers in PCNSLs, which might have been
associated with increased μFA, while destruction of neuronal fibers is more
common in glioblastomas.CONCLUSION
Time-dependent DWI parameters,
specifically rcADC and ΔADC are useful for differentiating between glioblastoma
and PCNSL.Acknowledgements
No acknowledgement found.References
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