Josh Peters1, Arne Brahms2, Vivian Janicaud1,3, Mariia Anikeeva1, Eva Peschke1, Frowin Ellermann1, Arianna Ferrari1, Konrad Aden4, Stefan Schreiber4, Rainer Herges2, Jan-Bernd Hövener1, and Andrey N. Pravdivtsev1
1SBMI, MOIN CC, UKSH, Kiel University, Kiel, Germany, 2Otto Diels Institute for Organic Chemistry, Kiel University, Kiel, Germany, 3Universität zu Lübeck, Lübeck, Germany, 4Institute of Clinical Molecular Biology, UKSH, Kiel University, Kiel, Germany
Synopsis
We synthesized 1-15N
nicotinamide (NA) and built a saddle-shaped mouse body linear coil for 15N
imaging. We found a way to polarize 1-15N-NA by dissolution dynamic
nuclear polarization to 9.1±4.4% and measured the effect of the radical load. The
lifetime of hyperpolarized NA was field-dependent: 100 s at 1 T, 60 s
at 7 T, and 30 s at 9.4 T. An effect of the pH on liquid-state polarization
was found. N FLASH MRI of hyperpolarized NA was acquired in less
than 1 s.
Introduction
The hyperpolarization of nuclear spins boosts the MR
signal of selected molecules and has enabled real-time metabolic imaging in
vivo.[1,2] 1-13C-pyruvate has the most striking
applications in cancer diagnosis by measuring the activity of lactate dehydrogenase.[2]
Most often, 13C was hyperpolarized, with
some examples for 1H, 15N, and other nuclei.[3,4] 15N is an attractive alternative for molecules
that do not have long-lived 13C or where the label or
polarization does not survive a chemical transformation.
Hyperpolarized nicotinamide (NA) is a promising
imaging target as it is a critical molecule in the glycolysis and metabolism of
fatty acids. NA can be used to spy on the activity of several enzymes: nicotinamide
N-methyltransferase[5], nicotinamide
phosphoribosyltransferase[6],
nicotinamide amidohydrolase,
and cytochrome P450.
We present the hyperpolarization of 1-15N-NA
for the first time, using dynamic nuclear polarization (dDNP), and demonstrate in
vitro 15N-MRI.Methods
NMR and MRI: 15N signals
were acquired using a 1T 15N benchtop NMR
(Spinsolve), 7T MRI (BioSpec 70/30), and 9.4T WB NMR (Avance NEO), the manufacturer’s software, and MestReNova.
dDNP. All dDNP experiments were performed a cryogen-free dDNP system
(SpinAligner)[10] at ~1.4K and 6.7T with a microwave
frequency of 187.185GHz at 35.2W and ~50mg samples. The built-up
of the 15N polarization in the solid-state was monitored every two
minutes with a ~3.5° pulse. After dissolution with 5mL superheated dissolution
media (Neutral-DM: pH 5.13, Basic-DM: pH 12.6), the sample was transferred to
the detection site in ~30s.
15N-contrast
agents (CA). 1-15N-NA
was synthesized from NA via spontaneous exchange with 15NH4
in methanol,[9] resulting in a white powder with a yellow tint at 40%
yield and 91±2 % 15N enrichment. We mixed 1,3-15N-urea (Sigma-Aldrich,
CAS: 2067-80-3), 1,3-15N, 2-13C-urea (Sigma-Aldrich,
CAS: 58069-83-3) or 1-15N-NA with trityl radical (AH111501,
POLARIZE) in deionized water and glycerol (Figure 1).
15N
MRI probes. 1H-15N, fixed dual-tune linear surface coil (1H/15N-SURF,
30 mm, O-XL-HL-070, Rapid Biomedical, Figure
3a) was compared to an in-house-built 3D-printed, linear 15N saddle-shaped
coil (15N-SLIN, L=52 mm, ID=46 mm, Figure 3b) in cross-coil mode with a 1H quadrature
resonator (112/086 QSN, Bruker). 15ml aqueous solution containing 800mM 15NH4
with 3 vol% Gd-contrast ([Gd], 1mmol/ml, Gadovist, Bayer) was used
as a model solution.Results
(13C)-15N-Urea polarization. 15N2-urea and 13C-15N2-urea
were reliably polarized to P15N
= 3.8 - 6.4 % (N=8, measured 30 s after dissolution), with
higher polarization (4.5±0.7 % vs 5.6±0.8%) and longer dDNP build-up constants
Tb-up = (2140±390s vs 3358±430s) for 12C
isotopomer (Figure 1). The relaxation times at 1T and 9.4T
were similar and about 30s; with this lifetime, the back-calculated
polarization at the time of dissolution was close to 20%.
15N-NA
polarization. We carried
out three to six dDNP experiments using four different 15N-NA
compositions with basic-DM (Figure 1);
no liquid-state polarization was observed when neutral-DM was used. The pH for
neutral-DM and basic-DM after the dissolution was 7.1 and 12.4, respectively.
Reducing the trityl radical concentration from 33mM to
13.8mM increased the 15N polarizations Pdiss from
2.6% to 8.4%, build-up constants Tb-up from 0.4h to 3.7h. T1 was strongly field-dependent:
100s at 1T, 60s at 7T, and 30s at 9.4T (Figure 2a). We could not observe a change in T1 and
polarization levels when deuterated dissolution media or a different radical
amount was used.
To investigate the influence of pH, we measured T1
of a 200mM 1-15N-NA sample (in H2O:D2O = 9:1)
in thermal equilibrium at 9.4T as a function of pH (Figure 2b); no significant T1 variation was found.
15N
MRI and probes. 1H/15N-SURF provided 2.8 times higher, but
much less homogeneous signals than 15N-SLIN (Figure 3, spectroscopy of 1 cm slice parallel to coil). Both
allowed 15N-spectroscopy (Figure
4a,b) and 15N-MRI (FLASH[11]) using the 15NH4 model
solution in thermal equilibrium (Figure
4c,d). Subsecond 15N-MRI of hyperpolarized 15N-NA provided
very high SNR (Figure 4e,f). Discussion
15N
probes. The in-house 15N-SLIN
coil can be built in one day and at the cost of only about 500 Euro (mainly
capacitors); it features about 3 times lower SNR and longer FA for the same
power as a commercial probe (Figure 3,4). Efforts are currently undertaken to improve SNR and reduce FA.
Hyperpolarization of 15N-NA.
15N-NA was successfully synthesized and polarized for
the first time. Already now, the polarization of ~10% is approaching the levels
needed for in vivo MRI; high SNR MRI was achieved in vitro in less than a
second. We assume that the reduced built-up time and polarization of the
dual-label urea were caused by an increased
solid-state relaxation.
Variation of the sample composition is expected to increase the polarization
yield further (water/glycerol ratio, DMSO). Previously, 3.4% 15N-NA
polarization was achieved in methanol using parahydrogen-induced polarization.[9] The protocols presented here are much closer to an in
vivo application, although the pH neutralizing step was not yet tested.Acknowledgements
We acknowledge support by funding from the German
Federal Ministry of Education and Research (BMBF) within the framework of the
e:Med research and funding concept (01ZX1915C), the Emmy Noether Program "metabolic
and molecular MR" (HO 4604/2-2), the research training group "materials
for brain" (GRK 2154/1-2019), the DFG grant INST 257/616-1 (FUGG), the SFB
"bulk-reaction" (TRR 287), the clusters of excellence "precision
medicine in inflammation" (PMI 1267) and "inflammation at interfaces"
(EXC 306, FOR 5042). Kiel University and the Medical Faculty are acknowledged
for supporting the Molecular Imaging North Competence Center (MOIN CC) as a
core facility for imaging in vivo. MOIN CC was founded by a grant from the
European Regional Development Fund (ERDF) and the Zukunftsprogramm Wirtschaft
of Schleswig-Holstein (Project no. 122-09-053). References
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