Guodong Weng1,2, Piotr Radojewski1,2, and Johannes Slotboom1,2
1Institute for Diagnostic and Interventional Neuroradiology, Support Center for Advanced Neuroimaging (SCAN), University of Bern, Bern, Switzerland, 2Translational Imaging Center, sitem-insel AG, Bern, Switzerland
Synopsis
Purpose: whole
brain in vivo detection of GABA/GABA+ using SLOW-editing at 7T.
Methods:
An EPSI-based B0/B1+ robust SLOW-editing
was applied in four healthy subjects. Pure GABA was measured by two different macromolecular-nulling
inversion recovery pulses, namely a broadband- and a narrowband-inversion pulse.
Results:
the editing GABA/GABA+ signal at 3.01 ppm are presented. The signal intensity
of GABA/GABA+ ratio is 40 – 60%.
Conclusions:
whole-brain in vivo GABA/GABA+-editing can be performed using SLOW-EPSI in
around 10 minutes measurement time and is therefore clinically
applicable.
INTRODUCTION
Recently, SLOW-editing has been
proposed as an alternative MEGA-editing at UHF1,2. The advantage of SLOW-editing
compared to MEGA-editing is that it requires very low SAR, and, if combined
with an EPSI readout 3, enables whole-brain coverage of GABA+ (i.e.,
GABA plus the coedited macromolecules (MM)) within a measurement of 10 minutes.
Like MEGA-based GABA-editing 4, SLOW-editing as presented in 1, has the
property that the MM at ~3 ppm is also refocussed. In this abstract we present
two different methods to eliminate the MM-component, to measure GABA instead of
GABA+.
We compared (i.) the
original SLOW-editing technique 1 (denoted by
IR-off) for detecting GABA+, (ii.) with a broadband MM
suppression adiabatic inversion recovery pulse 5 (denoted by IR-on), and (iii.) a
narrowband MM suppression adiabatic inversion recovery (denoted by sIR-on). The
three methods were tested and compared in vivo.METHODS
The MRI and MRSI was performed on a
Siemens 7T MR scanner in clinical mode (MAGNETOM Terra, Germany) using the Nova
1Tx 32Rx head coil.
The SLOW-EPSI 1–3 (Figure
1A-B) was applied on a spherical GABA-phantom prepared in-house (~10 mmol/L of
GABA, creatine, and glycine), and 4 healthy volunteers at 7T (TE = 68 ms, TR = 1500/1700
ms, matrix = 65 x 23 x 9, FOV = 280 x 180 x 70 mm, averages = 1, and TA = 9/10
min). The refocusing/editing chemical-selective adiabatic pulse (CSAP) for
SLOW-editing has a 24 ms duration. The bandwidth (BW) of editing-full and
editing-partial ranges from 1.6 – 4.2 ppm and 2.7 – 4.2 ppm, respectively
(Figure 2A). The editing result was obtained by the subtraction of editing-full
(acquisition #1) and editing-partial signal (acquisition #2). In SLOW-editing,
the 2π-CSAP acts as a refocusing- and as well as an editing-pulse.
For GABA+ detection, no suppression
pulse was applied to suppress the ~3.0 ppm MM-component (Figure 4A). For GABA
detection, two types of suppressed pulses were used: a broadband (1.2-4.9 ppm) adiabatic
inversion recovery (IR) pulse 5 (Figure
4B) and a narrowband (2.75 - 3.15 ppm) asymmetric adiabatic inversion recovery
(sIR) pulse (Figure 4C). Note that the same sIR pulse was used to suppress lipid
(1.45 – 1.85 ppm) instead of MM for GABA+ detection. The IR and sIR pulse have both
complex secant hyperbolic B1(t) = Ω0·sech(βt)1+μt modulation6.
Simulations of spin system were
performed using in-house MATLAB (R2019b) code, by solving relaxation-free
Liouville-von Neumann Equation alike in reference7.RESULTS
Figure 2A shows the pulse profiles
of editing-full (1.6 – 4.2 ppm) and editing-partial (2.7 – 4.2 ppm). The
corresponding in vitro measurement for GABA was shown in Figure 2B. Figure 2C-D
shows the simulation of Glx (glutamate/glutamine = 2/1) at 3.76 ppm and GABA at
3.01 ppm using SLOW-editing.
Figure 3A shows the in vivo measurement
(spectral editing difference) of three healthy volunteers with baseline
correction by splines. The GABA+ signal at 3.01 ppm is clearly present in both
gray- and white-matter, as well as the co-edited Glx at 3.76 ppm. The GABA+
signal is higher in gray matter than in white matter, as expected. Figure 3B
shows the results of SLOW/editing-full and -partial of subject 1. The black
lines indicate the passband of the 2π-CSAP pulses. Due to the implicit water
and lipid suppression of the 2π-CSAP pulses, no baseline correction was
applied, and no water/lipid removal needed in this case.
Figure 4 shows the pulse sequences
for GABA+ and GABA detection. The TIs used, are 234, 300 5 and 180 ms, respectively. The TI of
sIR-on (Figure 4C) was experimentally determined such that the reduction of the
signal at 3 ppm coincides with IR-on to obtain MM-nulling spectrum (Figure 4B)5.
Figure 5 shows the in vivo
measurement to detect GABA+ and GABA in a healthy subject. The pulse sequences
used to detect MM suppressed IR-off, IR-on and sIR-on spectra were shown in
Figure 4A-C. Figure 5C-D show the SLOW/editing-full and -partial results in
location 1 (indicated in Figure 4B), and the IR-on and sIR-on have the similar
reduction (65 - 75%) of signal at 3 ppm. The spectral editing differences for
GABA+ and GABA are illustrated in Figure 5A, E-F for three different locations (as
displayed in Figure 5B). The peak intensity ratio of GABA/GABA+ signal at 3.01
ppm ranges from 40 - 60%, which is consistent with the reference5.DISCUSSION
Both IR-on and sIR-on can
eliminate the MM signal from the GABA+ signal, whereas broadband IR-on MM nulling can result in a flatter baseline due to better suppression of lipids and other MMs. However, sIR-on has the clear advantage of lower SAR requirement and high signal intensity
of other metabolites. Since the sIR- and the IR-pulse are of adiabatic complex
sech-type, their performance is robust to B1+-inhomogeneities
as present in the 1Tx 32Rx head coil.CONCLUSION
SLOW-editing has successfully been
combined with adiabatic IR-on and sIR-on MM-nulling RF-pulses enabling the
unbiased measurement 3D spatial resolved editing of GABA at UHF within 10
minutes of measurement time. The GABA/GABA+ ratio at 3.01 ppm is consistent
with values given in literature. Acknowledgements
The
research is supported by the Swiss National Science Foundation (SNSF-182569).References
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