Sebastian Werner1, Bernhard Krauss2, and Marius Horger1
1Radiology, University Hospital Tuebingen, Germany, Tuebingen, Germany, 2Siemens Healthineers, Forchheim, Germany
Synopsis
Retrospective
quantitative evaluation of 103 focal osteolytic lesions of the axial skeleton
in virtual-non-calcium bone marrow images of 32 multiple myeloma patients in correlation to hematologic disease
status, T1w signal intensity and ADC. Virtual-non-calcium bone marrow imaging allows differentiation between
overall active and inactive disease with higher attenuation signifying an increasing
likelihood of active disease. This is supported by a significant positive correlation between the
attenuation and the ADC, as well as a corresponding inverse correlation to T1w
signal intensity.
Abstract body
Introduction: Reduced dose unenhanced whole-body computed tomography is recommended
as one of the standard imaging techniques in the initial diagnosis of multiple myeloma
(MM) bone disease according to the diagnostic criteria of the International
Myeloma Working Group (IMWG) 1. However in standard CT-imaging, even well-delineated lytic lesions often
do not significantly change their appearance during therapy 2,3. Recent publications
suggest that dual-energy CT (DECT) based BM imaging might be a viable
alternative to MRI in the assessment of BM involvement and thus in the
evaluation of the disease status in the course of therapy 4-6. In this study we aim at
defining the benefit of using VNCa images in assessing MM disease activity via
quantitative and qualitative analysis of the characteristics of focal bone
lesions and their relation to the hematologic laboratory as the ground truth,
as well as to the signal intensity in T1-weighted (T1w) MRI images and the
apparent diffusion coefficient (ADC) as currently used in routine BM imaging.
The working hypothesis is that the content of osteolytic lesions shows high
attenuation values in active myeloma and that inactive disease should lead to a
convergence in attenuation between focal lytic bone lesions and non-lytic
skeletal areas due to conversion of the lesions’ content to so-called yellow
marrow.
Methods: We retrospectively
evaluated 103 focal osteolytic lesions of the axial skeleton in VNCa
bone marrow images of 32 patients. Region of interest-based attenuation
measurements of lesion content and background bone marrow were correlated with
T1w signal intensity and ADC. Results were compared between patients in active
and inactive disease. Receiver operating characteristic (ROC) analysis was
performed to determine a cut-off value of VNCa attenuation for differentiation between the two
groups. Standard of reference was the overall disease status according to
International Myeloma Working Group response criteria.
Results: Mean absolute lesion
attenuation (HUabs) was 12.4 HU (SD, 24.6) in active disease and
-25.3 HU (SD, 32.0) in inactive disease (p<0.001). Mean attenuation
difference between lesions and background bone marrow (HUdiff) was
35.1 HU (SD, 28.5) in active disease and 16.2 HU (SD, 30.1) in inactive disease
(p=0.002). VNCa attenuation measurement allowed for differentiation between active
and inactive disease with a sensitivity of 92% and a specificity of 58% at a
cut-off value of -21 HU. In active disease, 42 lesions (70.0%) were either hypo- (21.7%) or isointense
(48.3%) compared to the intervertebral disc in the T1w images. In inactive
disease 34 lesions (79.1%) were hyperintense, 7 (16.3%) were isointense and 2
(4.7%) were hypointense. There was a statistically significant negative
correlation between the T1w signal intensity of the focal lesions and HUabs
(Spearman’s ρ = -0.617,
p<0.001). The mean ADC of lesions in the active disease category was 694.0 x 10-6
mm2/s (SD, 273.5) compared to
607.6 x 10-6 mm2/s (SD, 534.3) in the inactive disease
category (p < 0.001). We found a statistically significant positive
correlation between the ADC and HUabs (Spearman’s ρ = 0.521, p<0.001). VNCa
attenuation was negatively correlated to T1w signal intensity (Spearman’s ρ
-0.617, p<0.001) and positively correlated to ADC (Spearman’s ρ 0.521, p<0.001).
Discussion: This study shows a clear correlation between the attenuation of the BM
inside osteolytic lesions and the overall disease status, with higher
attenuation values increasing the likelihood of active disease. Furthermore,
the results show that in inactive disease the attenuation of the lesions and
the attenuation of the non-osteolytic areas tend to converge. Conversely, we
found significant inverse correlation between attenuation values measured
inside focal lytic lesions and their signal intensity on unenhanced T1w-images.
The latter is considered one of the most sensitive MR-sequences for imaging BM,
both in the initial diagnosis as well as during treatment. The use of DECT
based BM imaging in MM has been addressed in previous studies 4-7. Yet to our knowledge this is the first study that focuses on the
assessment of the disease activity in larger osteolytic lesions in MM using the
overall disease status as a standard of reference, providing new information on
the lesions’ characteristics in different clinical scenarios and showing
correlations to MRI signal intensities.
Conclusion: Quantitative assessment of attenuation of focal osteolytic lesions in VNCa
bone marrow images allows
differentiation between overall active and inactive disease with higher
attenuation signifying an increasing likelihood of active disease.
This is supported by a significant positive correlation between the attenuation
and the ADC, as well as a corresponding inverse correlation to T1w signal
intensity.Acknowledgements
This
research did not receive any specific grant from funding agencies in the
public, commercial, or not-for-profit sectors.
Berhard
Krauss is an employee of Siemens Healthineers.
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