Dvir Radunsky1, Tamar Blumenfeld-Katzir1, and Noam Ben-Eliezer1,2,3
1Bio-medical Engineering, Tel Aviv University, Tel Aviv, Israel, 2Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel, 3Center for Advanced Imaging Innovation and Research (CAI2R), New-York University Langone Medical Center, New York, NY, United States
Synopsis
Mapping
of T2 values is highly valuable for a wide range of applications.
Still, accurate mapping is challenging due to the inherent bias of rapid Multi-Echo
Spin-Echo (MESE) protocols by stimulated echoes, and also by magnetization
transfer (MT). In this work, we investigate the different effects of macromolecular-driven
MT (MTSAT) and direct water saturation (MTDIR)
mechanisms
on MESE signals, and their respective influence on T2 values. The investigation
includes quantitative MT measurements between protocols with different scan
settings and protocol schemes, aiming to isolate the individual roles of MTSAT
and MTDIR.
Introduction
To
date, Multi-Echo Spin-Echo (MESE) protocols constitute the most effective tool for quantifying
T2 relaxation times in vivo. Nevertheless, the extensive use of
radiofrequency (RF) pulses in MESE schemes leads to magnetization
transfer (MT) related bias of the measured signal, and potential
distortion of the measured T2 values1,2.
Traditionally,
MT ratio (MTR) measures the total relative signal loss between an MT-free
protocol, e.g. Single-echo SE (SSE), and an RF intensive protocol, e.g.
MESE, instigating MT effects. The measured MTR includes two distinct mechanisms:
macromolecular-driven MT (MTSAT) and direct water saturation
(MTDIR)3 and is calculated according to: $$$MTR=\frac{S_{MT-free}-S_{saturated}}{S_{MT-free}}$$$.
MTSAT
refers to the transfer of magnetization between the saturated macromolecular
pool (MMP) and the free water pool and can occur via cross-relaxation
or chemical exchange phenomena4. MTDIR, on the other hand, refers to signal
attenuation that originates from partial recovery of water protons between
consecutive TRs, or from incidental irradiation of neighboring
slices due to imperfect slice profile (i.e. slice crosstalk)5.
Previously
we investigated the influence of MT on quantitative mapping of T2 values
and showed that MT effect on MESE decay curve is constant and does not depend on scan
settings. Still, MTSAT occurring within the imaged slice does distort
the measured T2 values in comparison to SSE6. In this work we further explore the individual roles of MTSAT
and MTDIR in MESE acquisitions, and quantify the
effect of slice ordering on the measured signal and the ensuing T2 values.
Since SSE and MESE signals are essentially different7,8, we
analyze the evolution of MT along the respective echo trains, rather than
echo-to-echo comparison.Methods
Phantom preparation: six tubes were doped with 0.25 [mM] of MnCl2
and a unique urea concentrations of
0, 0.5, 1, 2, 3, and 4 Molars (Fig. 1).
The urea molecule [CO(NH)2] is favorable for studying MT due to the
high saturation efficiency of the amide groups9, and the
spectral characteristics, which allows to design targeted experiments for probing
MTDIR and MTSAT10.
MRI Scans were performed on a
whole-body 3T Siemens Prisma scanner and included MRS analysis (STRESS),
quantitative T1 (IR) and T2 (SSE and MESE) measurements,
and estimation of MTR.
Scan parameters
for the T2-encoding protocols: TR/TE=3000/10 ms; Nechoes
=1 (SSE), 10-30 (MESE), resolution=0.8x0.8 mm ; slice=4 mm; BW=200 Hz/Px
; Nslices =1-9; inter-slice gap= 0-200%; acceleration =2xGRAPPA;
Post-processing: Quantitative
T2 maps were generated using mono-exponential fit to the SSE data,
and using the Echo-Modulation-Curve (EMC) algorithm for the MESE data11–13.
MTSAT
evaluation
was based on the single-slice T2 dispersion between SSE and MESE
protocols.
MTDIR
was
also estimated for MESE vs. SSE, and for different variants of
MESE using varying ETL, TR, number of slices, inter-slice gap, and slice
ordering schemes.Results and Discussion
Fig. 2 shows the different effects
of diffusion and MTSAT on SSE and MESE signals and T2 values.
An expected decrease in baseline T2 is observed due to interactions
with the urea molecules14. While SSE-T2
is lower at zero concentration due to diffusion effects, MTSAT
dominates at higher concentrations, causing MESE-T2 values to be
underestimated. Diffusion and MTSAT effects, which correspondly cause a positive and negative
signal dispersion, shortens the measured T2 values and therefore
depend on the pulse scheme and the MMP content (i.e. causing the MESE signal to
be higher or lower).
The
individual contribution of MTDIR in this case is presented in Fig. 3, where the 1st echo MTR was measured between single-slice SSE and MESE scans with
different ETLs and TRs. Full transverse relaxation occurred between
consecutive sequence repetitions (T2 << T1), thus the increased MTR
baseline involves only MTDIR, which, in turn, depends on the T1/T2
ratio and the absolute T1 value of each tube15. Due to the shorter recovery time in
short-TR experiments, the MTDIR increases with longer ETLs (*P<0.001).
Consequently, MTDIR was not present for longer TRs. For any TR, the
T2 values remained stable for increasing ETLs (not shown),
indicating that MTSAT attenuation build up at a constant rate along
the echo train.
Fig. 4 shows the effect of slice ordering in MESE scans.
Sequential ordering yields higher MTDIR, compared to the interleaved
mode, due to increased slice crosstalk effect (i.e. allowing minimal recovery time before scanning adjacent slices)5,16,17. The MTR increased with the number of
slices in both ordering schemes, while increasing inter-slice gap exhibits an
inverse correlation with MTR. This result indicates MTDIR changes,
as the urea protons are excited in the boundaries of the central slice solely
(i.e. confined spectral width), thereby off-resonance MTSAT is
neglected. The T2 values (result not
shown) are independent of the slice ordering, number of acquires slices or the
gap between them, as MTDIR causes to a uniform signal attenuation,
thereby not affecting the relative decay pattern6.Conclusions
Herein
we investigate the individual contribution of MTSAT and MTDIR to the signal and T2
bias in MESE protocols. This study is also relevant for Turbo-SE schemes, and
can be generalized to any sequence where multiple RF pulses are applied in a
series. More ex vivo and in vivo experiments will
shed light on the effect and extent of MT mechanisms in quantitative MRI
measurements, and will expand the understanding on the macromolecular regime.Acknowledgements
ISF Grant 2009/17References
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