Christopher D Rowley1,2, Zhe Wu2,3, Ilana R. Leppert2, Jennifer S.W. Campbell 2, David A. Rudko2,4,5, G. Bruce Pike6, and Christine L. Tardif1,2,4
1Neurology and Neurosurgery, McGill University, Montreal, QC, Canada, 2McConnell Brain Imaging Center, McGill Unversity, Montreal, QC, Canada, 3Techna Institute, University Health Network, Toronto, ON, Canada, 4Department of Biomedical Engineering, McGill Unversity, Montreal, QC, Canada, 5Department of Neurology and Neurosurgery, McGill Unversity, Montreal, QC, Canada, 6Hotchkiss Brain Institute and Departments of Radiology and Clinical Neuroscience, University of Calgary, Calgary, AB, Canada
Synopsis
Inhomogeneous magnetization transfer (ihMT) contrast in
the brain has been reported to be a myelin-specific biomarker, but can be impacted
by B1 inhomogeneities, reducing its accuracy. ihMT equations
incorporating B1 correction assume a single excitation and readout,
either a k-space line or plane, per saturation module. Here we use an arbitrary
number of readout segments collected after an MT saturation preparation module.
T1 and B1 variations are included in the signal equations
to increase specificity of the contrast to the microstructure. The resulting
implementation yields a balance between acquisition efficiency and contrast
resolution for different brain imaging applications.
Introduction
Inhomogeneous magnetization transfer (ihMT) is a novel
contrast mechanism that has been reported to have enhanced myelin specificity compared
to conventional MT imaging1. The ihMT contrast
is primarily driven by the size of the semi-solid dipolar coupled pool, where
it is produced during single frequency offset MT saturation but not in dual
frequency MT saturation, following from Provotorov theory2. Thus, the
difference between dual and single frequency MT-weighted images can provide
insight into the dipolar coupled pool. Methylene groups, which are abundant in
lipid-rich membranes such as myelin, are a dominant contributor to the dipolar
coupled pool in the human brain3, which promotes
increased myelin-specificity with decreased contribution to the contrast from
proteins.
The saturation of a dipolar coupled pool is necessary
for the formation of ihMT contrast, which requires the use of strong B1
MT pulses. While multi-echo GRE readouts have been used previously4, there has not
been an effort to correct for the decrease in the formation of the dipolar
coupled pool, and ultimately the resulting ihMT contrast, due to B1
inhomogeneity. This work addresses the problem by deriving the Bloch equations
for a centric-out encoded MT-RAGE sequence (Figure 1), with an arbitrary number of readout lines. Centric-out
encoding is important to obtain high MT-weighted contrast in images with longer
echo trains. Following the framework of Deichmann et al.5, the
magnetization preparation was modified to be a percentage drop in the steady
state signal due to the MT pulses (termed here MTsat). Swanson et al.
have previously described the equations to calculate the ihMT-ratio (ihMTR)
provided the T2B and T1B
value for the semisolid pool are known, as well as the dipolar relaxation time
(T1D) of the tissue3. Since grey
matter (GM) and white matter (WM) have similar T1D values6, the impact of B1
on the formation of the dipolar pool can be approximated and used to restore the
ihMT contrast in images.Methods
One healthy volunteer was scanned on a 3T Siemens
Prisma MRI system with a 64-ch head receive coil. T1 mapping was
performed using the variable flip angle method with the following parameters: α1
= 5°, α2 = 20°, TR = 30ms. MT-weighted images were subsequently
collected using the following parameters: Δf=7kHz, B1=9.8μT, 12 MT
pulses in each preparation module (each RF with 0.96 ms duration, 1.76 ms
spacing), α = 5°, 11 readouts per 175ms TR, and 5.3ms echo-spacing for the GRE
kernel. The central frequency for the MT pulses was shifted by -100Hz to better
align with the center of the lipid pool1. All images were
collected with 1.5mm isotropic resolution and 250Hz/px bandwidth. B1 7
and B0 maps were also collected.
T1 and M0 values were calculated
based on variable flip angle8 analysis of the
images without MT contrast. The steady state signal was solved using the
equations in Figure 1 in each voxel over
a range of MTsat values. This required the following input values
for each voxel: T1, M0, B1 values to correct the
excitation flip angle α, and the B1 correction factor to address the
non-linear impact of B1 on MT saturation. The latter was calculated
using the equations from Swanson et al.3, assuming T2B
= 12µs 9, T1D =
6ms 6, T1B = 25ms 10, and a
super-Lorentzian lineshape. The theoretical MTsat was determined for
a range of B1 values, which were normalized by the MTsat
value at the relative B1 of 1.
ihMTsat was calculated as the difference
between the saturation in the dual vs single offset frequency cases. ihMTR was
calculated for comparison by using the 5° flip angle image with no MT preparation
from the VFA experiment to normalize the signal change.
The brain was manually skull-stripped using ITK-SNAP (http://www.itksnap.org),
and GM and WM tissues were segmented using the FANTASM algorithm11 on the masked R1
map. Mid-depth surfaces were generated using CBS tools (www.nitrc.org/projects/cbs-tools/),
and MR values were displayed on the cortical surface in MATLAB using Surfstat (www.math.mcgill.ca/keith/surfstat/).Results and Discussion
Figure
3 presents
ihMTR and ihMTsat values in an axial brain slice for visual
comparison of the two metrics, as well as the impact of B1 on each. Figure 4 displays the calculated
maps mapped onto the mid-cortical surface, derived from the equations in Figure 1 and smoothed with a 4.5mm
kernel. The ihMTsat map follows the myelination patterns across the
cortex as suggested by the R1 map. The ihMTR map varies
significantly from this pattern, which appears to be due to spatial non-uniformity
in B1. Cortical variations in ihMTsat and ihMTR are
plotted against B1 and R1 values in Figure 5. Cortical ihMTR is strongly correlated with B1
(r = 0.46), whereas ihMTsat displays no correlation with B1
(r = 0.07). Both ihMTR (r = 0.74) and ihMTsat (r = 0.75) show
correlations with the myelin-sensitive metric R1.Conclusion
Our results support that ihMTsat is a sensitive
marker of cortical myelination with minimal bias from B1
inhomogeneity. This formulation of ihMT may be more sensitive to the underlying
microstructure compared to ihMTR, in particular in high and ultra-high field
applications in humans where B1 inhomogeneity and SAR constraints
may impact the formation of a large dipolar coupled pool.Acknowledgements
No acknowledgement found.References
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