Yi-Chen Tsai1, Hui-Ru Tsai1, Ming-Chung Chou2, Ping-Hong Lai3,4, Jie-Yuan Li5,6, and Cheng-Wen Ko1
1Dept. of Computer Science and Engineering, National Sun Yat-sen University, Kaohsiung, Taiwan, 2Dept. of Medical Imaging and Radiological Sciences, Kaohsiung Medical University, Kaohsiung, Taiwan, 3Dept. of Radiology, Veterans General Hospital-Kaohsiung, Kaohsiung, Taiwan, 4School of Medicine, National Yang-Ming University, Taipei, Taiwan, 5Dept. of Neurology, E-Da Hospital, Kaohsiung, Kaohsiung, Taiwan, 6School of Medicine, I-Shou University, Kaohsiung, Kaohsiung, Taiwan
Synopsis
In this study, we explored the metabolic changes in WM of patients with
and without DNS using 1H MRS and DKI longitudinally at 1 week and 1, 3, and 9
months after CO intoxication. Increased Cr and Cho were observed at early stage
in WM of patients with DNS. Decreased
tNAA showed higher correlation with the presence of WM lesion than of DNS. The chronic change of Ins and Glx in patients
with DNS implies themselves as potential indices to provide valuable
information for monitoring DNS development.
Introduction
Delayed neurologic sequelae (DNS) is commonly
developed in patients with CO intoxication, which exhibits recurrent
neuropsychiatric symptoms after an interval of apparent normality1,2.
A neural marker for DNS is thus desirable to facilitate
prediction and monitoring of neuropathology for appropriate diagnosis and
treatment2,3. In this study,
we investigated the metabolic change in WM of patients with DNS at 1 week and
1, 3, and 9 months after CO intoxication using absolute quantification of 1H magnetic resonance
spectroscopy (MRS). Voxel-based diffusion kurtosis analysis was included for
comparison4.Methods
A total of 24 patients who experienced CO intoxication were enrolled in
this study with IRB approval. Eleven patients who exhibited white matter lesion
(WML) within follow-up period were categorized as WML+ and others as WML-. In
WML+ group, 5 patients were diagnosed with the presence of DNS (DNS+), and
others as DNS-. All patients were
evaluated regularly at 1 week and 1, 3, 9 months after CO intoxication. 25
normal subjects were included as control group.
All MR studies were conducted on a 1.5T
system (General
Electric) using 8-ch head coil with single voxel spectroscopy protocol (PRESS,
TR/TE = 1600/35 ms, Ave =128, voxel size = 2x2x2 cm3). By referencing to
axial T2 images, MRS voxel was
placed on the left occipital WM region
where WM abnormalities of DNS frequently occurred (Fig.1a). Acquired
spectra were analyzed by LCModel to compare the absolute metabolic levels of
NAA+NAAG (tNAA), Creatine (Cr), Choline (Cho), Myo-Inositol (Ins) and
Glutamate+Glutamine (Glx). 3D axial T1 images were used for white matter (WM),
gray matter (GM) and cerebrospinal fluid (CSF) segmentation by SPM12. The fractions of WM, GM and CSF within voxel
of interest (VOI) were applied to LCModel analysis for partial volume
correction. Cramér-Rao lower bounds (SD%) of values larger than 20% were
excluded in the statistical analysis. Absolute concentrations were presented as
mean and standard deviation in mmol/mL (institutional unit). Whole-brain DKI data were acquired using a
twice-refocused spin-echo diffusion-weighted pulse sequence with
TR/TE=10000/91ms, b-values=1000 and 2000 s/mm2, number of b0 images=4, matrix
size=80x80, FOV=240x240 mm2. Parameters of
fractional anisotropy (FA), axial kurtosis (AK), radial kurtosis (RK) and mean
kurtosis (MK) were calculated in the corresponding VOI of MRS. Non-parametric Mann–Whitney U test was
applied to group analysis where the p-value < 0.05 was considered
statistically significant. Results and Discussion
Quantitative comparison of tNAA, Cr, Cho, Ins
and Glx between patients with/without DNS are demonstrated in Fig.1. The tNAA of WML+DNS+ patients are
dramatically decreasing from 10.58±3.51 mmpl/mL at 1 week to 6.36±1.03 mmpl/mL
at 1 month. Both of Cr and Cho in patients with DNS (WML+DNS+) show significant
increase at 1 month (p<0.05) whilst Ins increases significantly at late
stage from 3 months to 9 months (p<0.05).
DKI indices (FA, AK, MK and RK) derived from VOI
of MRS demonstrate that they performed well in differencing patients with DNS
from DNS- ones (Fig.2). Patients with
DNS had higher diffusional kurtosis values than those without DNS at early
stage (1 month) after CO intoxication (p<0.05). Notably, the diffusional
kurtosis indices (AK, MK, RK) dropped in 3 months after CO intoxication and
became lower than those without DNS.
The absolute concentrations of Cr and Cho in
DNS+ patients increased after CO intoxication and showed significant difference
from DNS- patients (Fig.3). Cho of DNS+
patients also showed high correlation with AK values (r = 0.6, p<0.05). Although tNAA decreased significantly in
DNS+ patients in 1 month after CO intoxication,
it appeared similar longitudinal changes with WML+DNS- patients. The
elevation of Ins level of DNS+ patients reached maximum at 3 month after CO
intoxication and showed significant difference from DNS- group (Fig.4). Meanwhile, the separation of Glx level from
DNS- to DNS+ in patients with WML at 3 month can be observed (Fig.4). However, metabolic mechanism of Glx related
to DNS remains further investigation due to the small sample size of Glx
concentrations in this study.
Several 1H MRS studies of CO intoxication with
long TE has reported decreased tNAA/Cr and increased Cho/Cr ratios for DNS+
patients after CO intoxication1.
However, the increased Cr appeared in DNS+ patients reveals its
correlation with DNS at early stage after CO intoxication. Decreased tNAA of
WML+ patients with either DNS+ or DNS- can be biased by using Cr as denominator
as proposed in the previous studies.
Additionally, the use of short TE in our study enables the investigation
of WML and DNS development associated with Ins and Glx changes. Ins has been
proposed as a glial marker and increased Ins has been found in patients of
Parkinson’s disease5. Change in Glx has been implicated in studies
of neuropsychiatric and
neurological disorders6. Since DNS may
develop parkinsonism with a median latency of 4 weeks and mild cognitive impairment2, Glx
and Ins may provide valuable information for monitoring DNS development.Acknowledgements
No acknowledgement found.References
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