Spencer L. Waddle1, Maria E. Garza1, and Manus J. Donahue1,2,3
1Department of Radiology, Vanderbilt University Medical Center, Nashville, TN, United States, 2Department of Neurology, Vanderbilt University Medical Center, Nashville, TN, United States, 3Department of Psychiatry, Vanderbilt University Medical Center, Nashville, TN, United States
Synopsis
The asymmetric spin echo
technique (ASE) can be used to collect maps of R2’ and calculate cerebral
oxygen extraction fraction (OEF). However, these models require extravascular
signal only, which is often not fully achieved in standard ASE approaches. Here,
a vascular-space-occupancy (VASO) prepulse is used to null intravascular signal
in a novel VASO-ASE method, and repeatability measurements of parenchymal
versus extravascular R2’ are presented. VASO-ASE was found to provide
physiological OEF (34.1±5.3%)
and higher reproducibility in controls compared to standard ASE.
Introduction
Cerebral oxygen extraction
fraction (OEF) describes the ratio of oxygen consumed to oxygen delivered, and
is a biomarker of cerebral metabolic dysfunction related to infarct risk in
patients with multiple cerebrovascular diseases(1-3). OEF has historically been measured with 15-O
PET, however this method is not available outside of specialized facilities.
Quantitative R2' maps
obtained from asymmetric spin echo (ASE) sequences(4-6) can also be converted to OEF and venous
cerebral blood volume (vCBV) using a model of extravascular static signal
dephasing from magnetically inhomogeneous tissues(7). Importantly, this approach assumes that
diffusion from large vessels is negligible on the order of typical echo times
(TE) of 20-80 ms, and also that intravascular signal has been sufficiently suppressed.
When these criteria are not met however, vCBV can be overestimated and OEF
underestimated. Inaccuracy varies with voxel composition, which has limited ASE
generalizability.
At 3T and intermediate TE, it is well-known that signal is only
approximately 70% extravascular(8, 9). A common approach to suppress intravascular
signal is to apply bipolar gradients to dephase flowing blood-water(10). However this method increases TE, is
incompletely effective depending on b-value choice, and also can be dependent
on vessel orientation and gradient scheme.
A logical improvement is to exploit differences between 3T T1 of
blood (~1.65s) and tissue (~0.8-1.2s) water in an inversion recovery sequence
to null the longitudinal component of blood magnetization using a
vascular-space-occupancy (VASO) pre-pulse(11). Additionally, since the VASO pre-pulse is
played before excitation, this allows for shorter TE in this so-called VASO-ASE
acquisition compared to the gradient suppressed method. However, it remains
unclear whether signal-to-noise ratio (SNR) reduction in such a sequence may
confound the ability to obtain reproducible and physiological measures, and also
how intravascular vs. extravascular diffusion effects at short and long echo
shifting times influence quantitative accuracy. Here, we evaluate a novel
VASO-ASE protocol for regional OEF and vCBV determination, and quantify R2’
separately in brain parenchyma and extravascular tissue for different echo
shifting protocols. Methods
Participants.
All participants provided informed, written consent, and were scanned at 3.0T
(Philips Healthcare, Best, The Netherlands).
Sequence. A single-shot echo planar imaging (EPI) spin echo sequence was
modified for (i) an adiabatic pre-pulse to be applied and (ii) the spin echo to
increment for ASE contrast. To reduce inflow effects, a long TR=5000 ms and
blood water nulling time of 1.054s was used, corresponding to a blood water T1
of 1.65s. This allows approximately 20-30% of equilibrium longitudinal tissue
magnetization to remain at the blood water nulling time. Data were collected
with (VASO-ASE) and without (ASE) the intravascular suppression pre-pulse at
spatial resolution=3x3x3mm and TE=100ms; each scan was repeated once for
reproducibility. The readouts investigated included three τ=0 ms images, and 21 images sampled for τ=10-20 ms with a 0.5ms step. In some cases, ASE
was also sampled from τ=10-41 ms in 1 ms increments (Figure 1).
Analysis. OEF was calculated using a fitting procedure outlined in An, Lin
2003(10). Large vessel hematocrit (Hct) was assumed to
be 42%, and small vessel hematocrit incorporated here was 0.85·Hct=0.357. The susceptibility difference between fully oxygenated
and fully deoxygenated blood was 0.18 ppm per unit hematocrit(12). OEF and vCBV were calculated in bi-frontal
and bi-parietal regions. Signal-to-noise ratio (SNR), intraclass correlation
coefficient (ICC), and OEF and vCBV values (mean±standard deviation) were recorded. Results
Representative images and
decay curves for both sequences are shown in Figures 2 and 3, respectively. Across
multiple healthy subjects, ASE provided OEF=38.7±5.6% and vCBV=4.1±0.9 ml blood/ml brain (n=6), and
VASO-ASE yielded OEF=34.1±5.3 and vCBV=5.5±2.1 ml blood/ml brain (n=5). In reproducibility
considerations, the (ICC) was fair at 0.46 for ASE but moderate-to-good at 0.80
for VASO-ASE. SNR maps in Figure 4 show whole-brain SNR as 62.5±21.4 in ASE,
and 39.8±20.3 in VASO-ASE, consistent with reduced longitudinal tissue
magnetization in VASO-ASE. At
long τ values (τ>20ms) non-linear signal decay was present
in both sequences (Figure 5). Discussion
We investigated a novel
contrast mechanism to isolate extravascular R2’, which in principle enables
more appropriate application of extravascular static dephasing models for OEF
and vCBV determination. In VASO-ASE (extravascular R2’), mean OEF decreased and
vCBV increased compared to ASE (total parenchyma R2’). As seen in Figure 3, ASE
also presents faster decay (greater R2’) compared to VASO-ASE, especially for
long echo shifting times (Figure 5). Reduced R2’ is consistent with VASO-ASE
measuring extravascular water only, as intravascular venous signal will decay
more rapidly for this TE and field strength given longer tissue T2 relative to
venous blood T2. There was only mild variation in SNR across the range
investigated (Figure 4) and ICC values indicate higher reproducibility in VASO-ASE relative to ASE acquisitions. Comparison of the green line (linear fit τ=10-20ms) to the red line (linear fit τ=10-41ms) in Figure 5 (a) and (b) illustrates
how non-linearity at long τ values
results in over-estimated R2’ and vCBV values. Non-linearity is not consistent
with the static-dephasing model assumptions, and may indicate that the
contribution of total versus extravascular water diffusion differs with
asymmetric shifting time, a novel finding which could improve OEF quantitation
or perhaps serve as a foundation to interrogate extravascular diffusion with
higher SNR compared to standard diffusion-weighted-imaging approaches.Acknowledgements
No acknowledgement found.References
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