Mario G. Báez-Yáñez1, Jeroen Siero1,2, and Natalia Petridou1
1Department of Radiology, Center for Image Sciences, UMC Utrecht, Utrecht, Netherlands, 2Spinoza Centre for Neuroimaging Amsterdam, Royal Netherlands Academy of Arts and Sciences, Amsterdam, Netherlands
Synopsis
In
order to quantify the hemodynamic contributions to the BOLD fMRI signal in
humans, it is necessary to adopt a computational model that resembles the cortical
vasculature and mimics hemodynamic changes triggered by neurovascular coupling.
Moreover,
simulation of the local magnetic disturbance induced by the geometry, hemodynamic
changes, and the biophysical properties of the tissues can provide accurate insights
on the physiological fingerprint of the BOLD fMRI signal.
In
this work, based on a realistic 3D computational approach of the human cortical
vasculature, we simulate the biophysical effects produced by hemodynamic
changes to compute a dynamic BOLD fMRI signal response.
INTRODUCTION
Blood
oxygenation level-dependent functional magnetic resonance imaging (BOLD fMRI)
is a powerful noninvasive tool to study in-vivo brain function. However, the
BOLD fMRI signal is only an indirect measurement of brain function that results
from the hemodynamic fluctuations triggered by neurovascular coupling and the biophysical
properties of the tissues1.
In
order to investigate the BOLD fMRI signal formation, computational modeling
gives a comprehensive perspective on the relation between the brain’s
vasculature and associated hemodynamics. Furthermore, computational approaches can
provide fundamental insights on the complexity of the biophysical interactions
at mesoscopic level2.
Numerical
simulations of the BOLD fMRI signal based on non-realistic representations of
the vasculature3, such as spheres or cylinders, and on realistic 3D
vascular models4,5, obtained from the parietal cortex of mice with
two-photon microscopy, have so far only described some static properties of the
BOLD signal dependent on specific parameters of blood and tissue in
active/resting states.
Here,
using a realistic 3D computational model of the human vascular network, we have
performed numerical simulations to investigate the effect of the biophysical interaction
produced by realistic hemodynamic changes and the properties of the tissues
and, therefore, the impact of these changes on the time-course of the BOLD fMRI
signal.METHODS
We
have developed an algorithm that produces a synthetic 3D vascular model (SVM) that
statistically conforms to the properties of the human cortical vasculature6,7,8
(Figure1,left). Properties accounted in the network generation are
vessel’s radius, vessel’s length, number of connections between vessels, and
orientation with respect to the cortical surface (detailed in abstract #478). The
SVM thus generates a vascular network where hemodynamic simulations can be
performed9, and gives the possibility to study different hemodynamic
conditions.
Here,
we assumed an arterial dilation that produces a differential change of blood
pressure, blood flow and oxygenation level across the SVM network. In order to
compute the local magnetic field distortion, each microvessel is mimicked as a
straight finite cylinder. The local magnetic variation depends on vessel radius,
orientation with respect to the main magnetic field and oxygen saturation
computed from the SVM model.
Then,
for each differential hemodynamic response, it is possible to compute a differential
local magnetic field variation. In addition, water molecules in thermal motion
are simulated by a Monte Carlo approach (1 µm2/ms), which sense the local
magnetic field variation.
The
BOLD fMRI signal is computed by a phase accumulation of a bulk of spins
depicting diffusion sensing the local magnetic field. All spin dephases are
integrated over the echo-time corresponding to the particular hemodynamic stage.
Simulations were computed for 1E7 spins for gradient-echo (TE=27ms) and
spin-echo (TE=48ms) at 7T with a time resolution of 500µs.RESULTS
In
Figure 1, a representative SVM model is schemed. The three magnetic field maps
represent the local magnetic field variation produced on the top, middle and bottom
planes of the SVM model for an exemplary time point. It is possible to observe
the magnetic distortions produced by the microvascular network in the middle
plane (green colorscale). The top plane highlights the magnetic field
distortion induced by the macrovascular compartment.
Figure
2 shows the hemodynamic changes (blood pressure, blood flow and oxygen
saturation level) that resulted from an arterial dilation on the SVM network. The
main parameter that alters the local magnetic field is the oxygen saturation
level of each vascular segment.
Figure
3 shows the BOLD fMRI time-course computed for a gradient-echo and spin-echo
pulse sequence. It is possible to observe qualitative differences on both the
amplitude (~two times fold) and the width of the BOLD fMRI signal related to
the global oxygen saturation change. DISCUSSION/CONCLUSION
We
have simulated the evolution of the BOLD fMRI time course using a synthetic 3D
model of the human cortical vasculature and a realistic hemodynamic response
for an arterial dilation. Further, this model allows us to convert the
oxygenation level changes into a spatial magnetic field variation. In addition,
simulation of water diffusion sensing the differential local magnetic field
gives insights to disentangle quantitative hemodynamic properties of the BOLD
fMRI signal. Simulated BOLD signals show qualitative differences in the
amplitude and width for a gradient-echo and spin-echo pulse sequence. This sort
of simulation helps to understand the physiological fingerprint of the BOLD
fMRI signal formation in humans. Acknowledgements
This work was supported by the National Institute Of Mental Health of theNational Institutes of Health under Award Number R01MH111417References
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