Zhehao Hu1,2, Fei Han3, Andre J.W. Van der Kouwe4,5, Xiaoming Bi3, Bin Sun6, Jiayu Xiao1, Junzhou Chen1,2, Shlee S. Song7, Marcel M. Maya8, Debiao Li1,2,9, and Zhaoyang Fan1,2,9
1Biomedical Imaging Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, United States, 2Bioengineering Department, University of California, Los Angeles, Los Angeles, CA, United States, 3Siemens Healthineers, Los Angeles, CA, United States, 4A. A. Martinos Center for Biomedical Imaging, Massachusetts General Hospital, Charlestown, MA, United States, 5Department of Radiology, Harvard Medical School, Boston, MA, United States, 6Department of Radiology, Fujian Medical University Union Hospital, Fuzhou, China, 7Department of Neurology, Cedars-Sinai Medical Center, Los Angeles, CA, United States, 8Department of Imaging, Cedars-Sinai Medical Center, Los Angeles, CA, United States, 9Department of Medicine, University of California, Los Angeles, Los Angeles, CA, United States
Synopsis
While underexplored to date, motion
susceptibility may critically undermine clinical translation of 3D intracranial
MR vessel wall imaging (VWI). Motion artifacts observed in intracranial VWI are
either caused by head bulk motion or internally localized movement. By combing
volumetric navigators (vNav) and self-gating (SG) strategies, we propose a
novel motion compensation approach that can simultaneously address these two
motion issues. Our preliminary studies demonstrated the potential of using this
technique to improve robustness of 3D intracranial MR VWI.
Introduction
Intracranial MR vessel wall imaging
(VWI) is the only non-invasive modality that can directly visualize the vessel
wall and characterize wall pathologies, and has drawn great clinical interests
in the past few years1. 3D variable-flip-angle turbo spin-echo (aka.
SPACE, VISTA, or CUBE) is currently the method of choice for intracranial VWI.
However, the 3D acquisition fashion and relatively long scan times render this
method inherently susceptible to motion, which can result in the entire scan
being corrupted to the extent that it is unusable2. Motion artifacts observed in intracranial VWI are
either caused by head bulk motion (i.e. changing head position) or localized
movement of internal anatomic structures (i.e. cough). The volumetric
navigators (vNav) technique and self-gating (SG) strategy have demonstrated
effectiveness in reducing motion artifacts caused respectively by these two
motion types2,3. In this work, we develop a motion-robust
intracranial MR VWI technique by incorporating a combined vNav-SG strategy into
the SPACE sequence. Methods
Sequence Design
A commercially available T1-weighted
SPACE sequence was modified to include vNav and SG acquisition modules and
corresponding real-time decision-making and communication modules (Figure 1). A
center k-space line (SG line) is acquired from the first echo of the SPACE
readout to derive the projection of the entire imaging volume. The projections
acquired in subsequent TRs are cross-correlated (CC) to the reference
projection collected at the beginning. Based on their CC values, which indicate
the severity of motion contamination, all acquired TRs are prioritized and the
most affected TRs are reacquired at the end of the scan. vNav was implemented
as a 3D EPI module with 8-mm resolution and 256 mm FOV in all three directions
3. We insert one such 3D navigator at the end of
each TR to ensure that motion estimation is as close as possible to the SPACE
readout train. Each subsequent navigator is registered back to the first
navigator to realign the imaging coordinates of the SPACE readout and
navigator.
In vivo Study
7 healthy volunteers and 3 ischemic stroke
patients were recruited in the study. All scans were performed on a 3T system
(MAGNETOM Skyra; Siemens Healthineers, Germany) with a 20-channel head-neck
coil. For all healthy subjects, we performed directed-motion experiments to
evaluate the improvement produced by our technique when imaging subjects who
conduct both head bulk motion, i.e. changing head position, and localized
movement, i.e. cough. All subjects underwent 6 scans using the developed
vNav-SG SPACE sequence with different imaging conditions:
- Subject was asked to remain still; imaging without
vNav or SG (denoted as “W/O MO, W/O vNav-SG”);
- Subject was asked to remain still; imaging with
vNav and SG (“W/O MO, W/ vNav-SG”);
- Subject was asked to conduct motion; imaging without
vNav or SG (“W/ MO, W/O vNav-SG”);
-
Subject was asked to conduct motion; imaging with
vNav only (“W/ MO, W/vNav”);
-
Subject was asked to conduct motion; imaging with
SG only (“W/ MO, W/SG”);
-
Subject was asked to conduct motion; imaging with
vNav and SG (“W/ MO, W/ vNav-SG”).
Motion instructions containing two
types of motion were given over the intercom system at five preset stages. The
imaging protocol included: TR/TE=900/10 ms, 0.6mm isotropic resolution, 6 min
acquisition time without motion or vNav-SG. The number of SG reacquisition was
set to 30. Three strokes patients underwent the SPACE scan twice, one with
vNav-SG function off and the other on.
Data Analysis
All the vNav-SG SPACE image sets were
randomized and scored by a radiologist with 6 years of experience in
neurovascular imaging to demonstrate the effect of motion on image quality and
effectiveness of vNav-SG motion compensation technique in preventing quality
deterioration. A five-point scale was used: 0-poor, 1-fair, 2-average, 3-good,
4-excellent. For quantitative analysis, vessel wall sharpness was measured at
the inner and outer boundaries of 2 major vessel segments, i.e. middle cerebral
arteries (MCA) and basilar arteries (BA). Reformatted 2D cross-sectional images
were sharpness measured using an in-lab MATLAB sharpness measurement program
2.
The paired two-tailed Student’s t-test was used for the comparison between
scans.
Results
Figure 2 shows the images of the 6
imaging conditions in one healthy subject. Comparison of Figure 2A and 2B
demonstrates that navigators and reacquisition did not affect image intensity
or contrast. Motion artifacts led to severe degradation of overall image
quality (Figure 2C) but were reduced by either vNav (Figure 2D) or SG (Figure
2E) and well suppressed by a combined vNav-SG strategy (Figure 2F). Motion
significantly reduced overall image quality score and vessel wall sharpness at
either outer or inner boundary, whereas the adoption of our vNav-SG technique
significantly mitigated the effect (Figure 3 and Table 1). Figure 5 displays
the post-contrast results in a representative stroke patient. Plaques (Figure
5B) and vessels (Figure 5D and 5F) can be clearly depicted with vNav-SG
function on compared to their no motion-compensated counterparts (Figure 5A, 5C
and 5E). Conclusion
Head bulk motion and localized movement
can induce substantial artifactual effect in intracranial VWI, and a vNav-SG
based motion compensation strategy is feasible in mitigating the motion
artifacts and may help dramatically improve the robustness of the imaging
modality in clinical settings. Acknowledgements
Granted by NIH/NHLBI 1R01 HL147355. References
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