Alan C Seifert1,2,3 and S Johanna Vannesjo4,5
1Biomedical Engineering and Imaging Institute, Icahn School of Medicine at Mount Sinai, New York, NY, United States, 2Department of Radiology, Icahn School of Medicine at Mount Sinai, New York, NY, United States, 3Graduate School of Biomedical Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, United States, 4Spinal Cord Injury Center, University Hospital Balgrist, University of Zurich, Zurich, Switzerland, 5Wellcome Centre for Integrative Neuroimaging, FMRIB, University of Oxford, Oxford, United Kingdom
Synopsis
BOLD signal in
gradient-echo images is a combination of macrovascular and microvascular
contributions, where the macrovascular component, arising from larger veins
draining the activated tissue, is less specific to the site of activation. In this work, we image activation produced in
the cervical spinal cord by a noxious thermal stimulus at 7T. We consistently observed activation in the
dorsal white matter medial to the dorsal horn, rather than in the gray matter
itself. However, due to the relatively straightforward venous architecture of
the spinal cord, this observed displaced activation does remain closely related
to the true site of neuronal activation.
Introduction
The spinal cord
contains many neural circuits of scientific and clinical interest1, but fMRI of the spinal cord is complicated by
its small size and unfavorable location2. Higher field strengths yield greater SNR and
finer spatial resolution, but the spatial specificity of the BOLD signal to the
actual site of neuronal activation is complicated. BOLD signal in gradient-echo (GRE) images is
a combination of macrovascular and microvascular contributions, where the
macrovascular component, arising from larger veins draining the activated
tissue, is less specific to the site of activation. Increasing field strength increases the
microvascular BOLD signal to a greater extent than the macrovascular BOLD
signal, leading to greater spatial specificity in gradient-echo EPI (GRE-EPI)3,4. In this
work, we image activation produced in the cervical spinal cord by a noxious
thermal stimulus using two 7T BOLD fMRI protocols: single-shot GRE-EPI, and
4-shot GRE-EPI. The multi-shot protocol provides reduced spatial distortion and
signal drop-out.Methods
The cervical spinal cords (C4-C7 vertebral levels) of eight healthy
volunteers were scanned using a 7T whole-body MRI system (Magnetom, Siemens)
and a 22-channel RF coil5. Detailed parameters are
tabulated in Figure 1. Pulse-oximeter
and respiratory traces were simultaneously acquired. Multi-shot reconstruction
included a navigator-based per-shot frequency offset demodulation to mitigate
respiratory field variations.
In each 10min experiment, noxious thermal stimulation was
applied to the lateral surface of the base of the right thumb at a calibrated
intensity of 3/10 using an fMRI-compatible thermal stimulator (TSA-II, Medoc). Stimuli
were 10s in duration, baseline temperature was 32°C, and inter-stimulus
intervals were randomized between 25-45s.
This stimulus should produce sensory activation in the ipsilateral
dorsal horn at the neurological C6 (vertebral C5) level.
Four-dimensional timeseries images were
motion-corrected slicewise (x- and y-translation) using FSL FLIRT6, straightened using Spinal Cord Toolbox7, and spatially smoothed using an anisotropic Gaussian
kernel (2mm in-plane, 6mm through-slice). GLM analysis was performed within a
mask of the spinal cord in FSL FEAT8, incorporating a 37-term physiological noise
model (8 cardiac, 8 respiratory, 16 interaction, heart rate, respiratory volume
per time, CSF signal, and 2 motion correction terms)9.Results
In four subjects, activation occurred in the dorsal column
white matter adjacent to the dorsal nerve root (Figure 2). In one subject, activation occurred within
the dorsal horn gray matter (Figure 2, row 2), and in three subjects,
activation was not detected due to through-slice dephasing and respiratory
signal contamination (not illustrated).
Four-shot reconstructions were available in two
subjects. Activation in 4-shot images
also occurred in the dorsal column white matter, in the same location in the
transverse plane as in the single-shot images (Figure 3). In one subject, activation occurs one
vertebral level lower than expected in the single-shot image, due to subject
motion shifting the thermal stimulator to the palmar surface of the hand before
the single-shot scan.Discussion
Although the microvascular BOLD signal increases by a
greater factor than the macrovascular BOLD signal as field strength increases, larger
veins still produce the greatest BOLD signal in GRE-EPI at 7T, as they do at
lower fields10. However, the organization of the venous
drainage in the spinal cord (Figure 4) leads to a more straightforward
relationship between the location of macrovascular BOLD signal and the site of
neuronal activation than in the cortex11–13. Radial intramedullary veins drain into a circumferential
plexus on the surface of the cord, which in turn drains into six larger
longitudinal veins, then into radicular veins that exit the spinal canal along
nerve roots. The intramedullary veins
are small and roughly orthogonal to B0, so they produce strong extravascular
BOLD signal changes related to neuronal activation within their specific
watersheds. The larger longitudinal
veins produce less BOLD signal, due to their orientation largely parallel to B0
and the dilution of more oxygenated venous blood from activated tissue with
less oxygenated blood from unactivated tissue elsewhere in their watershed.
The venous architecture in the spinal cord explains our
observed BOLD activation in white matter.
Activation in the dorsal horn gray matter (Figure 4, yellow) causes
changes in blood oxygenation in the intramedullary veins and venous plexus
draining the activated tissue (Figure 4, purple), producing BOLD signal change
in the white matter tissue (Figure 4, orange) surrounding these veins. Because the venous plexus drains toward the
midline, activation is observed in the dorsal, rather than the lateral, white
matter. Although this activation is
displaced in space from the site of neuronal activation, the radial
intramedullary venous architecture means that this displaced activation has a
clear and direct relation to the true site of activation.
If greater specificity is desired, phase-based
post-acquisition suppression of macrovascular BOLD contribution14,15 and spin-echo-based fMRI16,17 have been demonstrated at lower fields, but may
prove difficult at 7T where SAR limits are more significant and background
phase distortions due to respiration are more severe.Conclusion
We have consistently
observed noxious thermal stimulus activation in the spinal cord dorsal white
matter, rather than in the gray matter itself. Based on the spinal cord vascular
architecture, this observed displaced activation remains closely related to the
true site of neuronal activation.Acknowledgements
This study was
supported by National Institutes of Health (NINDS) award number K01NS105160
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