Einar Heiberg1,2, Henrik Engblom1, Marcus Carlsson1, David Erlinge3, Dan Atar4, Anthony H Aletras1,5, and Hakan Arheden1
1Department of Clinical Sciences Lund, Clinical Physiology, Skåne University Hospital, Lund University, Lund, Sweden, 2Lund University, Wallenberg Center for Molecular Medicine, Lund, Sweden, 3Lund University, Department of Clinical Sciences Lund, Cardiology, Skåne University Hospital, Lund, Sweden, Lund, Sweden, 4Department of Cardiology, Oslo University Hospital Ullevål, and Instititute of Clinical Sciences, University of Oslo, Oslo, Norway, 5School of Medicine, Aristotele University of Thessaloniki, Laboratory of Computing, Medical Informatics and Biomedical – Imaging Technologies, Thessaloniki, Greece
Synopsis
The purpose of
this study is to systematically evaluate sources to variability in the n-SD from
remote method for infarct quantification. Remote ROI position, size, and number
of standard deviations all to a large extent affected infarct size. The main
driver of infarct variability in the n-SD method are the differences in myocardial
SD level, that varies between subjects, site and vendors. Based on the source
of variability in infarct size we conclude the n-SD method lack accuracy for infarct
quantification, especially in multi-center, multi-vendor setting.
INTRODUCTION
Cardiac MR late
gadolinium enhancement (LGE) is considered the gold standard for in vivo
myocardial infarct quantification. There are a multitude of methods for infarct
quantification such as manual planimetry, standard deviations from remote
(n-SD)1-3,
Full Width Half Maximum (FWHM)4,5
Otsu6,
expectation maximization (EWA)7,
level set methods6, Gaussian mixture model classification8. The latest consensus document from the Society of Cardiovascular Magnetic
Resonance (2013) refrains from making a statement regarding the optimal method
for infarct quantification as evidence is still being accumulated9.
Thus, the purpose of this study was to build on the existing research body of infarct
quantification by a systematic evaluation of the sources to variability in n-SD
method.METHODS
Subjects from two
multi-center, multi-vendor, prospective,
cardioprotective trials; CHILL-MI
10,
MITOCARE
11 were included with a total of 214 subjects from 17 sites and 6 countries. Scanners
from three different vendors were used for data acquisition (47 % Siemens,
37 % Philips, and 16 % GE). Infarct quantification was performed by a
corelab (Imacor AB, Lund, Sweden). Delineations were used to defined eligible
remote myocardium. Four sets of experiments were performed, all designed to only
evaluate variability as no gold standard infarct size is available in patients.
- Impact
on placement of remote ROI
A fixed size ROI consisting of 45 degrees
of the circumference was placed in the remote myocardium. The position was
shifted in steps of at least 10 degrees so that, if possible, 7 ROI’s could be placed
per slice.
- Impact
of size of remote ROI
A set of 7 different sized ROIs were placed
in the remote myocardium.
- Impact
of number of SD from remote
A fixed size ROI consisting of 45 degrees
of the circumference was placed centrally in the remote myocardium. n-SD from
remote was varied from 2 to 5 in steps of 0.5 (i.e. total 7 steps).
- Comparison
of remote myocardial SD between subjects, slices and vendors
Relative myocardial SD was computed as the median of the SD over different ROI positions
in each slice divided by the median signal intensity.
Thus, each slice
generated up to 7 measurements of infarct size and from this relative infarct
size, variability was computed by subtracting the median infarct size for the
slice and dividing it with the median infarct size. All slices that contained
≥10% and had at least a 50% circumference that contained no infarct were
included. The ROIs excluded the most endocardial and epicardial pixels (Figure
1).
RESULTS
In total,
1268 slices were included. The variability due to positioning, size, and
standard deviations is shown in Figure 2, and impact on infarct size
variability is shown in Table 1. The correlation between myocardial SD and infarct
size variability was r2=0.59, i.e 59% of the infarct variation was explained
by different levels of SD in the myocardium. The relative myocardial SD was
25%±8%, 41%±29%, and 39%±19% for GE, Philips, and Siemens scanners,
respectively. The differences between GE and the two other vendors were
statistically significant (p<0.01).DISCUSSION
The results show
that the ROI position, size, and number of standard deviations all to a large
extent affect infarct size. The main driver of the resulting variability in
infarct size from using n-SD method is the differences in noise level.
Therefore, it can be concluded that the n-SD method will inevitably have large
variability. The magnitude in variability of infarct size caused by different positions
of remote ROI is similar to the variability of the n of standard deviations
used (2-5). Whereas this has been extensively studied, little is discussed how
to place the remote ROI. Previously, different SD from remote have been
proposed ranging from 2-5 without consensus. This may be explained by the
finding that the myocardial SD levels were statistically different between
vendors. These differences should likely not be attributed to hardware
differences, but rather different noise removal algorithms.
Theoretically, the
size of remote ROI and the number of standard deviations could be standardized,
but that would not solve shortcomings in dependency of SD in myocardium as it varies
between subjects, sites and vendors. The relative myocardial SD varied with
276% between the lower and the upper quartile, which corresponds to a change from 2 to 5.5 SD from remote. In
trained hands the n-SD method may work if great care is applied in manual
placement of ROI’s. However, such careful application will closely correspond to
manual delineations. There is no validation of n-SD in a multi-center,
multi-vendor setting. Contrary, it has been shown that n-SD methods perform
worse than non-SD methods7.
Thus, previous multicenter studies using n-SD should be interpreted with
caution.
This study investigates
infarct quantification, however, the same arguments apply to other usages of
standard deviations from remote such as quantification of Myocardium at Risk
(MaR), or border zone.CONCLUSIONS
Based on the
source of variability in infarct size and the variability in myocardial SD levels
we conclude the n-SD method lack accuracy for infarct quantification, especially
in multi-center, multi-vendor setting.Acknowledgements
Einar Heiberg would like to acknowledge fruitful discussions with Jane Tufvesson on issues with n-SD method over the years.
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