Marios Georgiadis1,2,3, Els Fieremans2, Aileen Schroeter3, Manuel Guizar-Sicairos4, Zirui Gao4, Aleezah Balolia5, Piotr Walczak6,7, Lin Yang8, Gergely David9, Jiangyang Zhang2, Dmitry S. Novikov10, Markus Rudin3,11, and Michael Zeineh1
1Radiology, Stanford University, Stanford, CA, United States, 2NYU School of Medicine, New York, NY, United States, 3Institute for Biomedical Engineering, ETH Zurich, Zurich, Switzerland, 4Swiss Light Source, Paul Scherrer Institute, Villigen, Switzerland, 5Psychology, University of Colorado Denver, Denver, CO, United States, 6Radiology, Johns Hopkins Medicine, Baltimore, MD, United States, 7Diagnostic Radiology & Nuclear Medicine, University of Maryland School of Medicine, Baltimore, MD, United States, 8National Synchrotron Light Source II, Brookhaven National Laboratory, Upton, NY, United States, 9Balgrist University Hospital, University of Zurich, Zurich, Switzerland, 10Center for Biomedical Imaging, NYU School of Medicine, New York, NY, United States, 11Institute of Pharmacology and Toxicology, University of Zurich, Zurich, Switzerland
Synopsis
Axonal myelination is
an important indicator of brain development and is implicated in many
neurologic diseases. However, MRI methods to probe myelin are sensitive but not
specific. Small-angle X-ray scattering (SAXS) produces signal specific to myelin’s
nanostructural periodicity. Here we apply the recently developed SAXS tensor
tomography (SAXS-TT) to non-invasively retrieve myelin levels in mouse brains,
and compare them to myelin-sensitive MRI methods. We demonstrate SAXS-TT myelin
specificity i) using myelin histology, ii) on a dysmyelination model and iii) by
selectively probing central and peripheral nervous system myelin. We propose
SAXS-TT as quantitative tomographic method for validating MRI myelin-sensitive
sequences.
Introduction
Axonal
myelination is an important indicator of brain development and aging, and is associated
with multiple neurological/neurodegenerative diseases. Yet, spatially resolved
assessment of myelin levels remains a challenge. MRI methods sensitive to
myelin include (quantitative) magnetization transfer (MT), T1- and T2-relaxation,
and diffusion MRI (dMRI) metrics, among others. Myelin histology/immunohistochemistry
remains the gold standard for assessing myelin levels, despite artifacts
introduced during sample preparation, sectioning and staining. The recently
developed small-angle X-ray scattering
tensor tomography (SAXS-TT)1–3 provides a direct, non-destructive
approach for myelin imaging, given its specificity to myelin’s nanostructure,
i.e. its ~18nm layer periodicity. Here, we apply SAXS-TT on a mouse brain,
retrieve myelin levels and compare these to myelin histology and
myelin-sensitive MRI contrasts such as (quantitative) MT, T1-/T2-relaxation
and dMRI parameters. We also demonstrate SAXS-TT myelin specificity, by
studying a dysmyelination model (shiverer) against a control. Finally, we highlight
SAXS-TT sensitivity to different myelin nanostructural arrangements, by selectively
imaging central and peripheral nervous system myelin, exploiting their distinct
periodicities.Methods
Experiments:
a)
Multimodal
experiments on mouse brain:
MRI: A 5-month-old C57BL/6
mouse brain was MRI-scanned ex vivo on a 9.4T Bruker MRI scanner: i)
dMRI: voxel-size=75μm iso, 200 q-space
points, b-values=[1,2,3,4]ms/μm², ii) MT: voxel-size=150μm iso, offset=1500Hz, B1-amplitude=40μT, iii)
quantitative MT: multi-parameter mapping4,5 using proton-density-weighted (PDw),
magnetization-transfer-weighted (MTw) and T1-weigthed (T1w)
scans at TEs=1.5-19.5ms every 1.5ms, TR=25ms, flip angle: MTw/PDw=6o,
T1w=15o, iv) T1 mapping: voxel-size=75μm iso, TE=7.5ms, TR=[0.1,0.2,0.4,0.8,1,1.2,1.6,2,3]s, v) T2
mapping: voxel-size=75μm iso, TR=3s, 25 TEs=8.3-207.5ms every 8.3ms.
X-ray scattering: SAXS-TT
was performed in the cSAXS beamline of the Swiss Light Source synchrotron, with
voxel-size=150μm, Ephoton=16.3KeV, 267 projections, Fig. 1.
Myelin histology: half
of the MRI- and SAXS-TT-scanned brain was dehydrated in increasing concentration
ethanol baths, paraffin-embedded, microtome-sectioned in 310 consecutive
10-μm-thick sections, luxol-fast-blue stained, and brightfield-imaged at a
resolution of 0.9μm/pixel.
b)
SAXS-TT
on control and dysmyelinated (shiverer) mouse brains
The extracted brains from one myelinated control (Rag2-/- ) and one
shiverer (Rag2-/-sh-/-) 50-day-old mice were SAXS-TT-scanned in the LiX
beamline of the NSLS-II synchrotron (voxel-size=120μm, Ephoton=16.3KeV, 180
projections).
Analysis:
dMRI-based parameters
(fractional anisotropy-FA, radial diffusivity-RD, axonal water fraction-AWF,
extra-axonal perpendicular diffusivity-De⊥)
were derived using the DESIGNER pipeline,6 that includes noise and artifact removal,7,8 and calculation of diffusion and kurtosis
tensors, and white matter tract integrity9 (WMTI) parameters.
MTR was calculated as
(MTnopulse–MTpulse)/MTnopulse. MTsat
was calculated according to 4,5 using the PDw, T1w and
MTw scans.
T1 and T2
maps were derived from mono-exponential fitting of the respective signals.
For SAXS-TT, the
myelin-specific signal was isolated (Fig. 6b), and tensor-tomographic
reconstruction3 provided distribution of brain myelin levels.
Histology sections
were rigidly registered into a stack using Fiji.10
SAXS-TT and MRI datasets
were non-linearly registered11 to the myelin histology dataset, down-sampled
to 75μm. Pearson (r) and Spearman (ρ) correlations were performed over the entire hemisphere (362,303
voxels).Results
SAXS-TT whole mouse
brain experiments resulted in a quantitative myelin map, Fig. 1d. A coronal
section from the MRI-sensitive parameter maps is shown in Fig. 2. Correlations
between histology and SAXS-TT/MRI myelin metrics are shown in Fig. 3a. All
correlations are significant (p<0.0001) due to the high voxel number. The
highest linear (Pearson) correlation is with SAXS-TT (rSAXS-TT=0.86),
followed by MT parameters (rMTsat=0.68, rMTR=0.50),
FA and AWF (rFA=0.44, rAWF=0.41). Similarly,
SAXS-TT has the highest monotonic (Spearman) correlation (ρSAXS-TT=0.87) with histology, followed by MT parameters (ρMTsat=0.83, ρMTR=0.74), T2
and AWF (ρT2=-0.50, ρAWF=0.46). Since we are suggesting the use of SAXS-TT as a gold standard
for assessing myelin levels, we also correlated all MRI metrics with SAXS-TT,
Fig. 3a, third row. We assessed the equivalence of using SAXS-TT instead of
histology as reference for MRI metrics, Fig. 3b-c, where we plot the Spearman
correlations with SAXS-TT vs. the correlations with histology. We found a
Pearson coefficient of r=1 (Fig. 3b) and a Bland-Altman reproducibility
coefficient12 of 89% (Fig. 3c).
Figure 4 shows the
myelin maps for the control versus dysmyelinated mice, with the dysmyelinated brain
displaying very low myelination levels. Figure 5 shows the separate SAXS-TT
analysis of central and peripheral myelin, due to their distinct periodicities,
with Fig. 5c,d illustrating the (peripheral) trigeminal nerve (green) synapsing
with the brain (magenta) at the trigeminal nucleus, in a ball-socket fit.Discussion
We show that
SAXS-TT-derived myelin levels correlate highly with myelin histology, and
produce similar correlation coefficients as histology against MRI metrics.
Given SAXS-TT’s capability for tomographic and quantitative myelin analyses,
this suggests its potential use as a gold standard in myelin imaging, and as
reference for MR metrics, instead of histology, overcoming the need for sample
preparation, sectioning and staining. We further demonstrate SAXS-TT
specificity to myelin by studying a myelinated control versus a dysmyelination
model, and show that SAXS-TT can also capture this very limited myelination stemming
from the few myelin layers.13 Finally, we show that SAXS-TT is very
sensitive to myelin nanostructure differences by clearly distinguishing between
central and peripheral myelin, due to their distinct sheath periodicity (~16 vs.
~20nm). Overall, we present SAXS-TT as a non-invasive, myelin-specific imaging
method, that can be used for validation of MRI myelin-sensitive metrics and
quantitative (de-/re-)myelination investigations in the nervous system.Acknowledgements
Research was partly
supported by Swiss National Science Foundation (SNSF) grant numbers
P2EZP3_168920, P400PM_180773, 200021_178788, and by the National Institutes of
Health (NIH) award numbers R01 NS088040 and P41EB017183.References
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