Maria Aristova1, Alireza Vali1, Sameer A Ansari1,2,3, Ali Shaibani1,2, Tord D Alden2,4, Michael C Hurley1,2, Babak S Jahromi1,2, Matthew B Potts1,2, Michael Markl1,5, and Susanne Schnell1
1Radiology, Northwestern University, Chicago, IL, United States, 2Department of Neurosurgery, Northwestern University, Chicago, IL, United States, 3Department of Neurology, Northwestern University, Chicago, IL, United States, 4Ann & Robert H. Lurie Children’s Hospital of Chicago, Chicago, IL, United States, 5McCormick School of Engineering, Biomedical Engineering, Northwestern University, Chicago, IL, United States
Synopsis
Dual-venc 4D flow MRI with PEAK-GRAPPA
acceleration provides time-resolved 3D cerebral hemodynamics and could be
applied to cerebral arteriovenous malformations (AVM) with an appropriate
standardized protocol. We optimize dual-venc 4D flow imaging for AVM in vitro
and in vivo, and apply a Flow Distribution Network Graph paradigm for storing
and analyzing complex neurovascular 4D flow data. In vitro and in vivo, 4
voxels across a typical vessel (achievable in vivo with 0.8mm isotropic
resolution) will yield flow conservation < 15% and high reproducibility. Venous-arterial ratios of peak velocity and
pulsatility index are proposed as potential network-based biomarkers characterizing
AVM hemodynamics.
Introduction
Cerebral arteriovenous malformations (AVMs) are pathological connections
between arteries and veins. Dual-venc 4D flow MRI, an extended 4D flow MRI
method with improved velocity dynamic range, provides time-resolved 3D cerebral
hemodynamics [2]; however, a standardized protocol appropriate for AVM
quantification is lacking [3]. The goal of this work is to optimize dual-venc
4D flow parameters for AVM in vitro and in vivo, and to introduce and apply the
Flow Distribution Network Graph (FDNG) paradigm for storing and analyzing
complex neurovascular 4D flow data.Methods
The relationship between spatial
resolution, acceleration, and flow quantification accuracy was
systematically assessed in a specialized MR-compatible phantom containing three
flow channels. The phantom was integrated into a
feedback-controlled steady flow circuit (Figure 1A). In vitro
experiments were done with a single channel open (to assess flow quantification
accuracy), all channels open in parallel (to assess flow conservation) and a reproducibility
study of the 6mm channel. Acceleration factor and spatial resolution were
systematically varied for each setup. Image quality and hemodynamics were
assessed in a retrospective cohort study of 26 healthy subjects (age 41±17
years) and five AVM patients (age 27-68 years). All images were acquired at 3T
(Figure 1B-C) and postprocessed as in Figure 2A-C [4].
Quantification accuracy was assessed in vitro by direct
comparison to measured flow (Figure 2D).
In vivo, hemodynamics were
assessed according to parameters derived from the FDNG (Figure 2E). For
AVM patient data nidal (feeding or draining the AVM nidus) hemodynamics are
distinguished from extranidal (vessels not connected to the nidus). For flow, peak
velocity (PV) and pulsatility index (PI) [4], a venous-arterial ratio
characterized differences between venous and arterial flow, caused by blood
passing through either the small vessels of the brain or through an AVM.
In vitro measurements were compared to ground
truth with Student t-test. In vivo groups were compared with Wilcoxon rank-sum
test and Kruskal-Wallis test.Results
In vitro, flow was overestimated by an average 7.1±1.4% for
all in vitro conditions (Figure 3A). Error in flow measurement was
significantly correlated with number of voxels across the channel (P=3.11×10-28)
but not acceleration factor. Flow conservation error decreased
2.5% with unit increase in voxels across channel (P=0.0002) and increased
(relative to GRAPPA R=2) with PEAK-GRAPPA R=2 (P=0.02), R=3 (P= 0.07),
and R=5 (P=4.24×10-5).
At R=5, flow conservation error was within 15% for 4 or 5 voxels across vessel.
For both flow and PV, there was no significant association of reproducibility
coefficient with acceleration factor or number of voxels across the channel.
In vivo, individual FDNGs were
constructed for all controls (Figure 4A) and AVM patients (Figure 4C-D). In
controls, average flow conservation error was 20±14%, but 12% in subjects with
a median 4 or more voxels across the vessel vs 25% in those with less than 4
(P=0.027). Three healthy variants predominated in the control cohort: those
with the full Circle of Willis, left PCOM only, and no PCOM. Based on group
FDNGs, increased left transverse sinus (LTS) flow (P=0.045) and significantly
decreased right transverse sinus (RTS) flow (P=0.027) were observed in the
left-PCOM group compared to both-PCOM, with right sinus predominance unlike
both-PCOM and no-PCOM groups (Figure 4B).
As expected, no
significant difference was observed between controls and intranidal or
extranidal AVM circulation in venous-arterial flow ratio, another flow
conservation measure. For the venous-arterial PV and PI ratios (Figure 5B-C), a
significant difference was found between AVM nidal and extranidal circulation
(P=0.008 and 0.05 respectively), and between AVM nidal and healthy control
circulation (P=0.005 and 0.003 respectively), but not between controls and the
extranidal circulation of AVM patients (P=0.32 and 0.65 respectively). For venous-arterial PV and PI ratios, a significant
difference was found between AVM nidal and extranidal circulation (P=0.008 and 0.05 respectively), and between
AVM nidal and healthy control circulation (P=0.005
and 0.003 respectively).Conclusions
Based on in vitro results, at least 5 voxels across a vessel should be
used for flow quantification within 10-15%. However, 4 voxels across a typical
vessel (achievable in vivo with 0.8mm isotropic resolution) will yield flow
conservation < 15% and high reproducibility of flow and PV. Dual-venc 4D flow MRI and
standardized FDNG analysis may be feasible for efficient and standardized evaluation of the brain vessels in
clinical applications. Venous-arterial PI ratio, which is the inverse of the damping factor
proposed by Gosling [5], intuitively characterizes a lack of damping by the AVM
nidus relative to normal vasculature. Venous-arterial ratios of PV and PI are proposed for further
investigation as network-based biomarkers characterizing AVM nidal hemodynamics.Acknowledgements
The authors gratefully acknowledge support from the
following grants:
NIH F30 HL140910 (Aristova)
AHA 16SDG30420005 (Schnell)
NIH
R01 HL117888 (Markl)
AHA 18POST33990451 (Vali)
NIH T32 GM815229
(Northwestern)
NIH R21NS106696
(Schnell)
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