Liver Fat, Iron, & Fibrosis Abbreviated Protocols
Sudhakar K Venkatesh1
1Radiology, Mayo Clinic, Rochester, MN, United States
Synopsis
With the emergence of non-alcoholic fatty liver disease (NAFLD) as most common
chronic liver disease, detection and quantification of liver fat and fibrosis
becomes important. Iron overload frequently seen in association with fatty liver and liver fibrosis. Noninvasive quantification of liver fat, iron and fibrosis
with MRI is now considered the standard for diagnosis and monitoring. In this
presentation, typical clinical scenarios that requires MRI abbreviated protocol for liver fat, iron and fibrosis quantification, candidate sequences and multi-parametric MRI protocols will be discussed. MRI protocols will be illustrated with clinical examples and limitations will be highlighted.
Outcome/Objectives
At
the end of this lecture,the participant will be able to
-
Understand the clinical scenarios that
require abbreviated protocol for hepatic fat, iron and fibrosis
- Learn about multi-parametric MRI protocols
- Understand
the limitations of abbreviated protocol
Background
Non-alcoholic fatty liver disease is the leading cause of
chronic liver disease (CLD), hepatocellular carcinoma and liver transplantation
in the world. NAFLD has a spectrum of relatively benign non-progressive
non-alcoholic fatty liver (NAFL) to progressive nonalcoholic steatohepatitis
(NASH) which would finally lead to fibrosis or cirrhosis.
Physicians evaluating the patients at risk of NAFLD
(obesity, diabetes, and metabolic syndrome) would first confirm the diagnosis of
NAFLD, then determine NASH vs. NAFL and finally assess the severity of hepatic
fibrosis. Fibrosis is the most important prognostic factor in NAFLD
patients and advanced fibrosis (≥ stage 3) carry worse prognosis.
Increased iron deposition commonly occurs in many CLD
including NAFLD. Quantifying Liver iron content (LIC) is important for
confirmation of hereditary hemochromatosis (HH) in patients with genetic
predisposition and for management decisions in known HH patients. LIC is also
important in patients with iron overload due to transfusions or hematologic
disorders for decisions on chelation therapy.
Liver fibrosis is the end-stage healing response to
injury in CLDs and the outcome depends on fibrosis stage. Early detection is of
paramount importance as anti-fibrotic treatment would be most effective at
earlier stages of liver fibrosis. Monitoring progression and response to anti-fibrotic
treatment are invaluable in the follow up of CLD patients.Typical clinical scenarios for abbreviated liver quantification protocol
-
Living
liver transplant donor
- Detection
and quantification of fat, iron and fibrosis in patients with risk factors
- Assessment
of response to treatment or interventions
- Clinical
trials
- Patient
factors- mild claustrophobia, chronic disability, spine issues, etc
Ideal abbreviated Protocol
-
Reduced
scan time (time is money!)- A breath hold sequence would be great
- Simple
to perform- set up, running the sequence, minimum post processing
- Proven
clinical utility and diagnostic accuracy
- Low
failure rate/recalls
- No
need for intravenous contrast
Candidate sequences for abbreviated protocol
Fat
- Conventional In/Opp sequence; 2D or 3D dual echo
- Chemical shift based Proton density fat fraction calculation (PDFF)
Iron
- Signal intensity ratio
- R2* Relaxometry
Fibrosis
- MR Elastography
- Liver T1-mapping
- Diffusion weighted imaging and ADC
Multi-parametric protocol examples
- Localizer + 2D/3D dual echo + MRE: liver fat detection and rule out steatohepatitis +/- fibrosis
- Localizer + IDEAL + MRE: liver fat, iron and fibrosis quantification
- Localizer + T1 map+ T2*+ PDFF: liver fat, iron and LIF marker quantification
Limitations
-
Potentially miss focal liver lesions such as
HCC developing in background of liver fibrosis
-
Limited
evaluation of portal hypertension features particularly esophageal varices that
have potential to bleed with high mortality
Acknowledgements
No acknowledgement found.References
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