Sonia Waiczies1, Jens T. Rosenberg2, Christian Prinz1, Ludger Starke1, Jason M. Millward1, Paula Ramos Delgado1, Andreas Pohlmann1, Andre Kuehne3, Helmar Waiczies3, and Thoralf Niendorf1,4
1Berlin Ultrahigh Field Facility (B.U.F.F.), Max Delbrueck Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany, 2The National High Magnetic Field Laboratory, Florida State University, Tallahassee, FL, United States, 3MRI TOOLs GmbH, Berlin, Germany, 4Experimental and Clinical Research Center (ECRC), a joint cooperation between the Charité Medical Faculty and the Max Delbrueck Center, Berlin, Germany
Synopsis
Detection of brain inflammation by fluorine-19 (19F) MRI was studied for the first time at 21.1 T (NHMFL, Tallahassee, FL). Studies on phantoms showed an SNR gain of 2.1 at 21.1 T, when compared to 9.4 T. A dependency between 19F-MR relaxation and magnetic field strength (B0) was demonstrated. A T1 reduction and higher SNR per unit time counterbalanced the T2 shortening-induced loss in 19F MR signal when performing high spatially-resolved MRI of post mortem experimental autoimmune encephalomyelitis (EAE) mouse samples at 21.1 T. 19F-signals, not seen at 9.4 T, were revealed in both mouse brain and draining lymph nodes.
Introduction
Neuroinflammation can be monitored with 19F MRI using 19F-nanoparticles
(NPs) that label immune cells in vivo.
The migration of these cells into the brain can then be studied in EAE,1,2 a model of multiple sclerosis.3 Although the absence of background
signal is an advantage, the low abundance of 19F nuclei in vivo poses a major challenge for MR detection
in neuroinflammation. An increase in B0 from 9.4 to 21.1 T
would result in a theoretical
5-fold increase in the received MR signal. This is the main driving force for using higher B0. However, the theoretical SNR gain is reduced with increasing frequencies due to increases in noise
from sample and coil losses (e.g. SNR$$$\propto$$$B01.75 for solenoidal coils).4,5 Recognizing
these opportunities and challenges, we investigated the influence of 21.1 T on 19F
relaxation times and SNR gain, compared to 9.4 T.
Methods
Experiments were carried out on the 21.1 T vertical
bore MR system (1H: f=900 MHz, 19F: f=844.9 MHz) of the
National High Magnetic Field Laboratory (NHMFL) using a 19F/1H
birdcage coil (linear, LP, 8 rungs, lengthcoil=54.5mm, Øinner=33mm,
lengthshield=64mm, Øshield=53mm).
On the 9.4 T small animal MR system (1H: f=400 MHz, 19F: f=376
MHz, Bruker BioSpin, Ettlingen, Germany) a similar coil was used (linear, HP,
12 rungs, lengthcoil=50mm, Øinner=35mm,
lengthshield = 80mm, Øshield=
57mm). Electromagnetic
field simulations at the same absorbed power were used to determine B1
fields for both coils.6 Relaxation
time and SNR measurements: Four NMR tubes were filled with 19F-NPs
prepared from perfluoro-15-crown-5-ether (60mM, 120mM, 600mM, 1200mM
PFCE (Fluorochem))7
and submerged in a 50ml tube containing 4.5g/L NaCl. Parametric mapping was
performed on axial slices (FOV=[30x30]mm²,
matrix=96x96,
slice=10mm). RARE sequence for T1-mapping:
15xTRs=25-8000ms, TE=9.24ms, NEX=36 (9.4 T: TA=5h42m46s; 21.1 T: TA=4h7m19s).
MSME protocol for T2-mapping:
TR=4000ms, 150xTEs=7-1050ms (9.4 T: NEX=64, TA=6h49m36s; 21.1 T: NEX=36,
TA=3h50m24s). SNR was calculated8 on
axial
2D-RARE images (TR=4000ms, TE=9.1ms, ETL=4, FOV=[30x30]mm²,
matrix=128x126,
slices=1-10mm, NEX=1, TA=17m4s). Animal
experiments were carried out in accordance with local animal welfare protocols:
EAE was induced in SJL/J mice,2 PFCE
NPs were administered daily for five days2 after
which mouse tissue was prepared for ex
vivo MRI.2 3D-RARE brain 19F MRI: TR=800ms,
TE=4.9ms, FOV=[30x20x20]mm³,
NEX=256 (high-resolution: matrix=195x130x130,
ETL=33, TA= 7h30m33s;
medium-resolution: matrix=135x90x90,
ETL=23, TA=5h14m1s; low-resolution:
matrix=90x60x60,
ETL=15, TA=3h24m46s). FLASH brain 1H MRI: TR=150ms, TE=7.5ms,
FOV=[30x20x20]mm³,
matrix=600x400x400,
NEX=2, TA=3h20m. Results
Both T1 and T2 values for PFCE NPs were influenced by B0. Similar to other nuclei, the transverse spin-spin
relaxation was decreased at 21.1 T (Figure
1A). Contrary to 1H, where T1
values increase with B0,9 the T1
of the PFCE NPs decreased by nearly 50% at 21.1 T (Figure 1B). The PFCE concentration did not influence T1 and T2 values (Table
1). For SNR measurements, slice thickness was varied for all PFCE
concentrations. SNR was obtained as a function of the number of 19F
atoms per voxel (Figure 2). An SNR
gain of 2.1 was calculated at 21.1 T versus 9.4 T. The EM simulations revealed a 1.8-fold decrease in
the B1 field at 21.1 T, when compared to 9.4 T: the maximum SNR gain
is expected to be 2.8 (5/1.8), assuming total sample noise dominance and a
lossless receive chain. High resolved MRI of EAE mice at 21.1 T revealed a greater
level of detail of the immune cell migration in the inflamed brain and draining
lymph nodes (Figure 3).Discussion
We demonstrate the feasibility of 19F MRI at 21.1 T for
detecting inflammation in the brain and adjacent lymphatic system with high spatial
definition. The decline in T1 with
increasing B0 is consistent with previous studies.10,11 This suggests that at higher B0, data
acquisition can be better accelerated. The decline in T2 at higher B0 hampers the expected increase in 19F MR
signal at 21.1 T. The difference in the experimental SNR gain (2.1) and the maximum
expected SNR gain (2.8)
could be explained by coil and receive chain losses as well as preamplifier
noise variations between both setups. Conclusion
For a swift translation into application, high spatially-resolved 19F
MRI was performed on EAE post mortem
tissue to determine the feasibility of improved spatial definition of
inflammatory cell location. This will allow us to study
neuroinflammation with greater detail in future in vivo studies. Acknowledgements
Part of this work was
performed at the US National High Magnetic Field Laboratory, which is supported
by the State of Florida and the National Science Foundation Cooperative
Agreement No. DMR-1157490. The authors wish to thank Dr. William Brey and his team at the National High Magnetic Field Laboratory for valuable input and discussions on coil selection and design. The NHMFL User Collaborations Grant Program also
provided travel funding to SW and HW.
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