Hesamoddin Jahanian1, Thomas Christen2, Michael Moseley2, and Greg Zaharchuk2
1Radiology, University of Washington, Seattle, WA, United States, 2Radiology, Stanford University, Palo Alto, CA, United States
Synopsis
We studied
the default mode network in a group of Moyamoya patients using ICA method and
observed erroneous assessments of functional connectivity in regions with
prolonged arterial arrival time. We showed that these arterial delays could
lead to erroneous elimination of affected brain regions from a functional
connectivity network. We proposed a method called “temporal realignment” to
mitigate this problem.
Introduction
rs-fMRI has been increasingly used
to explore the brain’s functional organization and to probe its alterations in
neurological or psychiatric diseases1-3. Because functional connectivity
networks are identified utilizing the temporal correlation between signal
fluctuations of spatially remote areas of the brain, rs-fMRI analyses are
inherently sensitive to delays in signal time-series that may exist in some neurological diseases.
Moyamoya is an example of such diseases
in which
a chronic steno-occlusive vasculopathy causes considerably
increased regional arterial transit delays.
We previously demonstrated
that not accounting for arterial transit delays might lead to erroneous
identification of functional connectivity networks and proposed a method called
multi-delay analysis to correct for these errors4. The multi-delay
approach, however, can only be applied to seed-based analysis methods and is
not applicable to other popular rs-fMRI analysis methods such as Independent
component analysis (ICA)5. To remedy this problem, we propose a
novel prep-processing approach called temporal
realignment to correct the arterial transit delays before analyzing the
data. This method can be applied to any fMRI analysis method. In this study we
evaluated the proposed method using ICA technique.
Methods and Materials
Nineteen Moyamoya patients
(39±10 yrs; range 25-61 yrs; M/F 4/15) and ten healthy volunteers (30±6 yrs;
range 24-45 yrs, M/F 6/4) were included in this study. Subjects were scanned at
3T (GE MR750). For all subjects, rs-fMRI data was collected using a 2D gradient
echo EPI sequence (FOV=22 cm, matrix= 64×64, slice thickness=3.5 mm, number of
slices=35, TR/TE=2s/30ms, scan duration = 6 min). We analyzed the data using ICA,
with and without the proposed temporal realignment
technique. For temporal realignment, we first create a delay map from the
rs-fMRI data using a cross correlation analysis6, in which the lag
between each voxel’s time-course and a reference time-series (mean global
signal) is calculated (e.g. Figure 1.b). Then we shift the time-series of each
voxel according to the corresponding lag, estimated in the previous step, to
obtain a temporally realigned 4D rs-fMRI time-series (Figure 1). After the
temporal realignment followed by standard preprocessing steps, the default mode
network (DMN) was probed using ICA carried out by FSL’s MELODIC. For Moyamoya
patients we also collected gadolinium-based DSC images and calculated Tmax maps
using RAPID software7.
Results
Resting-state connectivity analyses
in 3 Moyamoya patients with increased arterial arrival delays are shown in Figure
2. In all of them, ICA did not detect the connectivity of one of the major DMN nodes.
These “missing” nodes were recovered after temporal realignment.
We compared the DMN
connectivity maps obtained before and after temporal realignment using two metrics:
1) mean Z-score and 2) Signal-noise-separation (SNS)8 defined as the
two-sample t-test comparing signal (voxels inside the DMN) and noise (voxels outside
the DMN). We calculated these metrics within the whole DMN for Moyamoya
(Figure 3.a,b) and healthy subjects (Figure 3.c,d). we observed a significant
improvement in both the mean Z-score and SNS after temporal realignment. Whereas
in healthy subjects the temporal realignment did not lead to a significant change
in either Z-score or SNS. For Moyamoya patients we also
calculated the mean Z-score only in the affected nodes (Figure 4). Affected
nodes were defined by finding the overlap between the thresholded DSC Tmax maps
(Tmax > 3 sec) and the predefined DMN mask in MNI space. In this analysis, the difference between the measured Z-scores
before and after the correction were even more significant (p<0.001).Conclusion
We studied the DMN in a group of
Moyamoya patients using ICA and observed significantly weaker connectivity in
regions with prolonged arterial arrival time leading to inaccurate and
incomplete characterization of functional connectivity in these patients. We
demonstrate that using the proposed temporal realignment technique it is
possible to mitigate the deleterious effects of arterial arrival delay on the
assessment of functional connectivity of the DMN. The
relationship between the delays observed in DSC Tmax maps and functional
connectivity in the affected area strongly suggests that the apparent
disruption in connectivity in these regions are due to spatial differences in
arterial arrival delay rather true disruptions in neuronal connectivity, in
which case it would have been unlikely to be able to recover them with simple
temporal shifting techniques.
Based on
this, weakened connectivity of a node in functional connectivity maps in
cerebrovascular disease patients obtained using the standard methods do not
necessarily correspond to a “functional disconnectivity.” When studying
patients with potential arterial arrival delays, it is crucial to account for
this to be able to distinguish between true and apparent lack of functional
connectivity.Acknowledgements
This work is supported by NIH grants 1R01NS066506, 2R01NS047607, R01 DK092241.References
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