The lateral subventricular zone has been explored in association with high-grade gliomas, both in-vivo and with MRI. The third ventricle subventricular zone (TVZ) has been explored in-vivo, using immunohistochemistry and microarray analysis, with regard to neurofibromatosis type-1-associated low-grade optic pathway gliomas. This remains unexplored with MRI. This study examined diffusion MRI features of the TVZ and its association with NF1-status and peri-tumour white matter integrity. TVZ features correlated with NF1-status, and peri-tumour white matter integrity. These results suggest that the state of the TVZ environment can potentially indicate whether a sporadic tumour might behave like its less disruptive NF1-associated counterpart.
20 paediatric patients with OPG were imaged on a Siemens 3.0 T Prisma scanner (Siemens, Erlangen), with multi-shell diffusion MRI (b=0,1000,2200 s/mm2, 60 directions per shell) in addition to clinical protocols. Diffusion pre-processing was performed using FSL (FMRIB, Oxford). Optic pathway tractography was performed using multi-shell, multi-tissue constrained spherical deconvolution6,7 with MRtrix3 (Brain Research Institute, Melbourne), utilising an existing protocol.4
The third ventricle mask was manually drawn in FSL and the TVZ was automatically segmented by dilating the third ventricle mask by 15 mm. A 10 mm restriction was applied inside the dilation to exclude regions potentially affected by partial voluming from cerebral spinal fluid, resulting in a 5 mm dilation. The TVZ ROIs were located within the thalamus and excluded unidentified bright objects and tumour (Figure 1). TVZ-ADCmedian values were recorded. Tumour masks, based on T2-weighted hyperintensity, were manually drawn by a neuro-radiologist using FSL. The tumour mask was dilated by 4 mm increments in all directions, four times, using Matlab, 2016b, (MathWorks Inc., Natick, MA). The tumour dilations that intersected with optic pathway WM were selected and their FAmedian values recorded (Figure 2). The FAmedian gradients, running from the first intersecting WM (4 mm) through to the fourth intersecting WM (total gradient along 16 mm outside the tumour), were calculated after applying a linear fit. TVZ-ADC values were compared according to NF1-status using Students’ t-tests. TVZ-ADC values were then correlated with FAmedian gradient values, while controlling for NF1-status, using partial correlation.
The higher ADC in the NF1-associated TVZ, and its negative correlation with FA gradients along peritumoural white matter, point to a possible association of TVZ not only with NF1-status, but also with tumour behaviour. NF1-associated OPGs tend to have better prognosis in terms of both visual outcome and tumour progression, whereas sporadic OPGs are more likely to symptomatically progress in comparison.8 The peritumoural biologic and imaging-defined mechanisms involved in tumour behaviour, in the directly adjacent WM, remain relatively unexplored. A shallow FA gradient beyond the T2-defined tumour boundary suggests less locally disrupted white matter. A sharper FA gradient suggests potentially disrupted, and less well-structured, WM adjacent to the tumour. The clinical relevance of these results is highlighted when one considers that, though imaging correlates of symptomatic progression have been previously identified,4 reliable predictive imaging markers of longitudinal symptom change do not yet exist. Furthermore, chemotherapy is generally given to preserve visual function, or in response to a growing tumour. However, response to chemotherapeutic treatment is unpredictable. Determining which child would benefit from, or which child would do just as well without chemotherapy, remains a challenge. These results suggest that NF1-associated OPG cases with lower TVZ-ADC may behave more similarly to sporadic tumours. Conversely, sporadic OPGs with higher TVZ-ADC might behave in a less locally disruptive manner. Incorporating TVZ features into longitudinal MRI analyses of OPG behaviour could add value for non-invasively identifying markers of more disruptive tumour behaviour to predict symptomatic progression.
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