Clinical guidelines for the diagnosis and follow-up of multiple sclerosis recommends brain MR imaging with gadolinium based contrast agents. Our aim was to address concerns about the use of gadolinium, the risk of accumulation in the brain and propose changes to clinical guidelines published in 2016. Group consensus is that GBCA remain essential in the diagnostic evaluation of a patient suspected of having MS to demonstrate active inflammatory lesions. GBCA should be used judiciously, minimizing gadolinium exposure and dose when possible.
BACKGROUND
Clinical guidelines for the diagnosis and follow-up of multiple sclerosis1 recommends brain MR imaging with gadolinium based contrast agents (GBCA). Soon after publication of these recommendations, because of the issue of tissue deposition of gadolinium, a subsequent follow-up Letter to the Editor2 recommended the “judicious use of GBCA in certain circumstances, including early diagnosis of MS,” which was an important change compared to the earlier recommendation.
OBJECTIVES
To address concerns about the use of gadolinium, the risk of accumulation in the brain and propose changes to clinical guidelines1 published in 2016.
METHODS
The authors convened a consensus conference in early 2017. The meeting, sponsored by the Consortium of MS Centers, brought together an international group of neurologists, radiologists, and imaging scientists with an expertise in MS to revise and update the guidelines and indications for standardized brain and spinal cord MRI for MS including attention to the use of gadolinium, based on new data, survey results and expert opinion.
RESULTS
Recent literature3 has shown that all GBCA lead to retention of very small amounts of gadolinium (Gad) in the brain (predominantly dentate nuclei and globus pallidus), bone, skin, and elsewhere in the body, although different GBCA seem to retain Gad at different rates/amounts, with macrocyclic agents, in general, retaining less than linear agents. Consideration of GBCA and dose should depend on agent sensitivity to detect new inflammatory lesions as GBCA may vary in the degree to which they induce lesion enhancement per dose versus likelihood of tissue deposition. There have been no documented cases of clinical neuro-toxicity associated with Gad retention in the brain. Because health effects of Gad deposition in the brain are unknown, the need for contrast-enhanced studies must be clearly indicated.
CONCLUSION
Group consensus is that GBCA remain essential in the diagnostic evaluation of a patient suspected of having MS to demonstrate active inflammatory lesions. GBCA should be used judiciously, minimizing gadolinium exposure and dose when possible. Routine monitoring with GBCA may be useful in the following circumstances: suspicion of clinical disease activity for which confirmation may modify therapy, confirmation of lack of disease activity, and facilitating selection of disease modifying therapy.
1. Traboulsee A, Simon JH, Stone L, et al. Revised Recommendations of the Consortium of MS Centers Task Force for a Standardized MRI Protocol and Clinical Guidelines for the Diagnosis and Follow-Up of Multiple Sclerosis. AJNR Am J Neuroradiol. 2016;37(3):394-401. doi:10.3174/ajnr.A4539.
2. Traboulsee A, Li D. Addressing Concerns Regarding the Use of Gadolinium in a Standardized MRI Protocol for the Diagnosis and Follow-Up of Multiple Sclerosis. AJNR Am J Neuroradiol. 2016;37(12):E82-E83. doi:10.3174/ajnr.A4943.
3. Gulani V, Calamante F, Shellock FG, Kanal E, Reeder SB, International Society for Magnetic Resonance in Medicine. Gadolinium deposition in the brain: summary of evidence and recommendations. Lancet Neurol. 2017;16(7):564-570.