Michelle Moerel1,2, Federico De Martino2,3, Kamil Ugurbil3, Elia Formisano1,2, and Essa Yacoub3
1Maastricht Centre for Systems Biology, Maastricht University, Maastricht, Netherlands, 2Faculty of Psychology and Neuroscience, Maastricht University, Maastricht, Netherlands, 3Center for Magnetic Resonance Research, University of Minnesota, Minneapolis, MN, United States
Synopsis
Using ultra-high field fMRI, we explored the
cortical depth dependent stability of acoustic feature preference in human
auditory cortex. In accordance with results from invasive recordings in cat
auditory cortex, we observed a relatively stable (i.e., columnar) tuning to
frequency and temporal modulations, while spectral modulation tuning was less
stable throughout the cortical depth. This could represent simpler spectral
tuning in middle auditory cortical layers, compared to more complex spectral
tuning superficially. Furthermore, results suggest a coding strategy in which
tuning to some features is kept stable orthogonal to the cortex, while tuning
to other features systematically varies.
Introduction
In the auditory cortex,
neural population preference to acoustic features varies orderly throughout the
cortical sheet, however, invasive recordings in cat auditory cortex showed relatively
stable (i.e., columnar) tuning to acoustic features orthogonal to the cortical
sheet1. Using the sensitivity and specificity of ultra-high field fMRI,
columnar processing in visual cortex has been successfully explored, showing a
columnar organization of ocular dominance2 and orientation tuning in
V13, color- and disparity-selectivity in V2/V34, and
direction of motion selectivity in V5/hMT5. In a subset of primary
auditory cortex (PAC), we recently observed columnar tuning to frequency6.
Here, we build on this previous work by exploring the cortical depth dependent
stability of acoustic features beyond frequency. Methods
Measurements were
performed at 7T (Siemens) using a custom whole head 32 channel loop transceiver
and a high performance head gradient insert. In six subjects, we acquired
high-resolution anatomical data (T1, T2*,
and proton density [PD] weighted data; 0.6 mm isotropic) used for segmentation7,
cortical layer sampling8, and myelin-based definition of PAC9.
Additionally, 0.8 mm isotropic 3D GRASE10 (TE = 27.9 ms; slices =
16; TR = 2000 ms; TA = 330 ms) images were acquired while the subjects listened
to 144 natural sounds. Maps of preferred frequency, temporal modulation rate,
and spectral modulation scale were computed with an encoding approach11-12.
Following previous work6, the stability of feature preference (i.e.,
columnarity) was assessed as the gradient of preference along vs. orthogonal to
the cortical sheet. We assessed significance of this columnarity index by
permuting (N = 1,000) the feature maps while preserving the 3D smoothness, and
setting the threshold at those values that occurred less than 5% of the time in
the permuted maps (p = 0.05, multiple comparison correction with a
permutation-based cluster threshold). We compared the columnarity across
feature maps by computing the average columnarity in PAC and non-PAC per map. Differences
across features and regions were tested for significance using a two-way
repeated measures ANOVA with factors ‘Feature’ [frequency, rate, scale] x
‘Region’ [PAC, non-PAC], followed by paired t-tests. Map smoothness along the
cortical sheet was assessed on the deepest cortical depth. For each gridpoint,
we binned the Euclidean Distance to all other gridpoints at 0.3 mm intervals,
and recorded the average feature preference of each bin. Across gridpoints, but
per bin, we computed the correlation between the feature preference of the
original gridpoints and those of the surrounding neighborhood.Results
Large-scale feature
maps were in accordance with previous results12 (Figure 1). Throughout
the cortical depth, regions with relatively stable and variable preference
could be observed (Figure 2). For each feature and hemisphere, we observed significantly
columnar regions and regions where preference varied throughout the cortical
depth (Figure 3A). Significantly columnar regions did not overlap perfectly
across features (Figure 3B). Only 0.6% of gridpoints were significantly
columnar for all three features. A combined columnarity for two out of three
features was slightly more common (8.6% of gridpoints), while the majority of
gridpoints were either significantly columnar for one or none of the features
(32.1% and 58.6% of the gridpoints, respectively). A repeated measures two-way
ANOVA with factors ‘Feature’ [frequency, rate, scale] x ‘Region’ [PAC, non-PAC]
showed that the columnarity was different across features (main effect of
‘Feature’; F(2,16) = 4.18, p = 0.023; no significant interaction; Figure 3C)
but not regions. Subsequent paired t-tests showed that the temporal rate maps
were significantly more columnar than the spectral scale maps (t[8] = 3.34, p
(corrected) = 0.031). The frequency maps were more columnar than the spectral
scale maps as well, but this difference did not reach statistical significance
(t[8] = 2.60; p (corrected) = 0.095). Map smoothness was comparable across
feature maps (Figure 4).Discussion
We observed columnar
regions in all feature maps, with stronger columnarity for maps of temporal
modulation rate than spectral modulation scale. This difference in columnarity
could not be explained by a difference in map smoothness (variance along the
cortical sheet), as smoothness was relatively similar across feature maps. Our
results are in accordance with invasive recordings from cat auditory cortex where
a transformation in spectral modulation tuning throughout the cortical depth
was observed1, and could represent simpler spectral tuning in middle
auditory cortical layers compared to more complex spectral tuning
superficially. Finally, the lack of overlap of columnar maps across acoustic
features suggests a coding strategy in which tuning to some features is kept
stable orthogonal to the cortex, while tuning to other features systematically
varies.Acknowledgements
This work was supported by the Netherlands Organization
for Scientific Research (NWO; Rubicon grant 446-12-010; VENI grant 451-15-012),
the National Institutes of Health (NIH grants P41 EB015894, P30 NS076408, and
S10 RR026783), and the WM KECK Foundation. This research has been made possible
with the support of the Dutch Province of Limburg.References
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