Francesca Reineri1, Eleonora Cavallari1, Carla Carrera1, and Silvio Aime1
1Molecular Biotechnology and Health Sciences, University of Torino, Torino, Italy
Synopsis
Hyperpolarized [1-13C]pyruvate has been widely
exploited for the in vivo
investigation of metabolic processes under normal and diseased conditions. The possibility
to obtain it using the cost effective and fast PHIP (ParaHydrogen Induced
Polarization) method would allow a widespread application of this powerful
diagnostic tool to pre-clinical research and would pave the way to future
clinical translation. Here we show the first in vivo studies carried out on
genetically modified mice using [1-13C]pyruvate obtained by means of
the PHIP-SAH (PHIP-Side Arm Hydrogenation) method. The results obtained from PHIP-SAH
hyperpolarized pyruvate are consistent with the pathologic state of the heart
tissue.
Introduction
[1-13C]-pyruvate is at a central crossroad of cellular metabolism
as it leads to energy production as well as to the formation of lactate and
alanine . It is
the hyperpolarized (HP) metabolite that has been most widely exploited for the in vivo investigation of metabolic
processes under normal and diseased conditions [1] and is currently
obtained by means of dissolution Dynamic Nuclear Polarization (d-DNP), a method
that requires quite expensive and technically demanding polarizer, and is intrinsically
slow.[2]
ParaHydrogen
Induced Polarization (PHIP) is a method for the generation of HP molecules that
requires much more affordable equipment than d-DNP and it owns the great
advantage that the polarization cycle takes only few minutes.
The recently introduced PHIP-SAH strategy (PHIP
by means of Side Arm Hydrogenation)[3] allowed to hyperpolarize pyruvate, and other metabolites,
that, previously, could be obtained only by d-DNP.
Aim of this work is to show the
first application of PHIP hyperpolarized pyruvate to in vivo MRS-MRI metabolic
studies.Methods
[1-13C] pyruvate
has been hyperpolarized by means of PHIP-SAH. [3]
Genetically
modified Lmna mice and the corresponding WT mice were used for in vivo 13C-MRS-MRI
studies, that were carried out on a 1T-MR system.
13C dynamic studies were performed and
space-selective 13C-MR spectra were acquired on slices centered
either on the heart or on the kidneys. 13C-CSI images were also obtained.Results
The 13C
signal enhancement observed on the
carboxylate moiety of [1-13C]pyruvate resulted to be 50600±5600 fold, with respect to thermal polarization at 1T, corresponding to
4.5±0.5%. 13C hyperpolarization has been
back-calculated at time zero, i.e. at the end of MFC, and reached the value of
10.6±1.5 %. In order to obtain a bio-compatible aqueous solution of the HP
metabolite, a diluted base (NaOH 0.1M)
has been used for hydrolysis, instead of the initially reported 1M base, and methanol,
used in the proof-of-concept study as hydrogenation co-solvent, has been eliminated.
13C-CSI (13C
chemical shift imaging) acquisitions have been obtained within 15-20 seconds
after the i.v. injection of the dose of HP pyruvate in heathy mice (wild type
mice). Metabolite maps have been generated at the resonance frequencies of
pyruvate and lactate (figure 1). Series of 13C-MR spectra acquired on a slab centered on heart
or kidneys showed the metabolic build-up of lactate (figure 2), in both organs.
Kinetic analysis of the metabolic exchange of the 13C label between [1-13C]pyruvate
and [1- 13C]lactate has been carried out by monitoring the time
courses of the signals of hyperpolarized lactate and pyruvate (figure 2b). The metabolic
response of a WT mouse was compared to the one obtained in an analogous
experiment with a Lmna mouse. The 13C HP label exchange rate between pyruvate and lactate is
markedly lower in the mutant mouse than in the WT one. On the contrary, when
the slice-selective 13C MR spectra are acquired on the kidneys, no
difference can be observed between the transgenic Lmna and the WT mice.Discussion
Lmna
mice show several diseases, called laminopaties, the most important one is
cardiomyopathy,[4] i.e. a decrease of the cardiac contractile function, that is
usually assessed by echocardiographic investigations. The observed reduction in
the pyruvate/lactate exchange rate may be accounted for in terms of this
disease. The reduced pyruvate/lactate exchange rate is a reporter of the
general metabolic activity of the cells and might be due either to lower
activity of the transporters (MCT) or to an altered cytosolic redox state in
Lmna mice.
The 13C-MRS
carried out on the kidneys allow to
confirm that the altered metabolism observed in the heart of mutant mice is due
to the Lmna mutations, that are limited to striatus tissues, muscles and heart,
while kidneys remain unaffected.
Conclusions
This
study has shown that in vivo metabolic imaging investigations
based on the administration of a dose of [1-13C]pyruvate obtained
from the PHIP-SAH procedure is possible. The in vivo results obtained from PHIP-SAH
hyperpolarized pyruvate are consistent with the pathologic state of the heart
tissue. Consequently,
one may expect that the easy access to HP pyruvate by PHIP-SAH methodology will prompt new comers to
the use of this powerful metabolic imaging tool that was previously available
only to the d-DNP-equipped labs.Acknowledgements
This
work was carried out thanks to the support of Airc (Italian Association for Cancer Research, TRIDEO
call 2015) and Compagnia
di San Paolo (Athenaeum Research 2016, n. CSTO164550). Scientific support from Aspect Imaging is gratefully acknowledged.
References
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