Daniele Mascali1, Emily Kittelson2, Keith Jamison2, Kâmil Uğurbil2, Essa Yacoub2, Shalom Michaeli2, Lynn Eberly3, Melissa Terpstra2, Federico Giove1, and Silvia Mangia2
1Museo Storico della Fisica e Centro Studi e Ricerche “Enrico Fermi”, Rome, Italy, 2Center for Magnetic Resonance Research, Dept. of Radiology, University of Minnesota, Minneapolis, MN, United States, 3Division of Biostatistics, University of Minnesota, Minneapolis, MN, United States
Synopsis
Age-courses of multiple MRI outcomes were here characterized
with a specific focus to default mode network (DMN) regions. Data were
collected with unprecedented sensitivity and spatial resolution using the Human
Connectome Project Lifespan Pilot protocol from 65 subjects divided in 4
age-groups (teen, young, middle-age and older adults). Age-related decreases of
grey matter volumes, mean diffusivity, amplitude of resting-state oscillations
and regional homogeneity were observed in both anterior and posterior DMN, and
were more pronounced in anterior than in posterior DMN. Connectivity between posterior and anterior
DMN regions remained relatively stable during the lifespan.
Purpose
The lifespan Human Connectome Project (HCP)1 aims
at characterizing the development and aging of the human brain by utilizing multiple
MRI outcomes with unprecedented sensitivity and spatial resolution. The goal of
the present work was to use data from the HCP Lifespan Pilot study for describing
the age-course of structural and functional parameters obtained with structural
MRI, diffusion weighted imaging for tractography (dMRI) and resting-state
functional MRI (rsfMRI). We specifically focused on two major hubs of the default
mode network (DMN), namely the anterior DMN (aDMN) and posterior DMN (pDMN).
The DMN is a critical network due to its link to cognitive and self-awareness
functions.2Methods
MRI outcomes were obtained from a group of 65 subjects (27M/38F)
who underwent the HCP Lifespan Pilot protocol at 3 T. Subjects belonged to 4
age-groups: teen (n=14, 8M/6F, 15.1±0.8
y.o.); young (n=18, 8M/10F, 29.8±3.2
y.o.); middle-age (n=18, 8M/10F, 48.9±3.3
y.o.); older adults (n=15, 3M/12F, 71.1±2.5
y.o.). The imaging protocol included MPRAGE (TE/TR=2.22/2400 ms, TI=1000 ms) and
T2-SPACE (TE/TR=563/3200 ms) at 0.8 mm isotropic resolution; multiband dMRI (MB4,
TR/TE=3222/89.2 ms, 1.5 mm isotropic voxels, 92 slices, 184 diffusion weighting
directions, b=1500 and 3000 s/mm2) for estimates of fractional
anisotropy (FA) and mean diffusivity (MD); multiband gradient-recalled echo EPI
(MB8, TR/TE =720/37 ms, 2.0 mm isotropic voxels, 72 oblique-axial slices) for
estimates of resting-state activity and connectivity.
Data analysis followed pipelines established by the HCP and available via
the GitHub repository (http://humanconnectome.org/documentation/HCP-pipelines/), including the FIX fMRI denoising and Diffusion Pipelines.3-5
Structural parameters included grey matter (GM) volume, white matter (WM)
volume, and brain parenchyma fraction (BPF). Microstructural parameters
included T1w/T2w ratio, FA and MD. Resting-state functional parameters included
region-to-region connectivity, power spectrum and fractional amplitude of low
frequency fluctuations (fALFF), and connectivity of each voxel with its
neighbors, also known as regional homogeneity (ReHo). Age-group
comparisons were performed with two-tailed t-tests. Framewise displacement was
used as nuisance covariate in resting-state comparisons to take into account
variability in head movements.Results
Signs of
structural differences with age were observed in the whole brain (Figure 1),
including reductions of cortical GM volumes and BPF, whereas the cortical WM
volume was somewhat more preserved during the lifespan. Microstructural changes
occurred especially in the grey matter (Figure 2). In particular, consistently lower
MD and higher T1w/T2w ratio were observed across the entire investigated
lifespan, whereas higher grey matter FA was observed only starting from the
young age group.
The rsfMRI seed-analysis
with the seed in the posterior cingulate cortex (PCC) revealed the typical DMN
(Figure 3). No significant changes of connectivity between pDMN and aDMN were
detected during the lifespan. On the other hand, age-related differences in
structural, microstructural and the other functional resting-state parameters
were observed in both pDMN and aDMN during the entire lifespan (Figure 4). The lower
grey matter MD, power spectrum, fALFF and ReHo, and the higher FA were more
pronounced in aDMN as compared to pDMN. Discussion
Progressively
lower grey matter volumes and MD across the lifespan were in agreement with
previous reports.6 Interestingly,
MD in the GM was strongly correlated with GM volume (r=0.68, p<10-10),
perhaps indicating that the observed microstructural changes highlight
substrates of macrostructural changes. The T1w/T2w ratios are commonly
considered as a myelin marker.7 However the origin of the age-related
T1w/T2w increase in the GM is unclear, and is likely due to a factor which
shortens T2 (e.g., iron accumulation) rather than myelin change.
The reduced age-associated resting-state activity in
both aDMN and pDMN occurred in the presence of age-related structural and
microstructural changes, but did not result in reduced connectivity between the
two hubs. Importantly, all resting-state parameters were strictly calculated only
in GM voxels as identified by the segmentation of each subject, and therefore were
not biased by the GM atrophy that also occurs with age. Yet, there was a strong
correlation between GM volumes and functional parameters (GMV:fALFF r=0.66
p<10-9, GMV:REHO r=0.58 p<10-7), although causality
cannot be established based on these findings only. Further studies with larger
cohort of subjects will allow identifying the age-courses of structural and
functional brain parameters with more refined time-granularity.Conclusion
The lifespan HCP collects
a variety of structural and functional brain properties with unprecedented
sensitivity, thus leading to a deeper understanding of how the human brain
develops and ages. Based on the present data obtained with the lifespan HCP
piloting protocol, aging may impact the aDMN faster and more profoundly than
the pDMN, however the connectivity between these two brain regions remains
relatively unaffected in healthy aging. Acknowledgements
This work was supported by Supplement U54 MH091657,
U01AG052564, U91MH109589, P41 EB015894, P30 NS076408. This project has also received funding from the European Union's Horizon2020 research and innovation programme under the Marie Sklodowska-Curie grant agreement No 691110 (MICROBRADAM).References
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