Kristina Andelovic1, Patrick Winter2, Thomas Kampf2,3, Volker Herold2, Sebastian Schuerlein4, Jan Hansmann4, Peter Jakob2,5, and Wolfgang Bauer1
1Medizinische Klinik und Poliklinik I, Universitätsklinikum Würzburg, Würzburg, Germany, 2Experimentelle Physik V, Universität Würzburg, Würzburg, Germany, 3Institut für Diagnostische und Interventionelle Neuroradiologie, Universitätsklinikum Würzburg, Würzburg, Germany, 4Deptartment of Tissue Engineering and Regenerative Medicine, Universitätsklinikum Würzburg, Würzburg, Germany, 5Fraunhofer IIS, Fraunhofer EZRT, Magnetresonanz- und Röntgenbildgebung (MRB), Würzburg, Germany
Synopsis
Biological
artery models,
cultured in a bioreactor-platform with adjustable pulsatile flow conditions,
represent a potential in vitro test
system for atherosclerosis research and provide
a suitable tool for the development of new flow quantification techniques as well as
studies of arterial elasticity and flow dynamics ex vivo and in vitro. A major requirement for these studies is viable motion synchronization
in order to achieve time-resolved flow measurements. We present a new platform
that uses self-navigation instead of external trigger signals for measurements of
2D- and 4D flow dynamics, vessel wall morphology and quantifications of arterial
pulse-wave-velocity and wall shear stress.
Purpose
The knowledge
of the correlation between arterial pulse-wave-velocity (PWV) and endothelial
wall shear stress (WSS) is of great relevance to understand the interaction of
arterial stiffness with pathologic flow patterns. Non-invasive measurements of
these parameters using flow MRI in vivo
are challenging due to long measurement times and limited spatiotemporal
resolution. Flow phantoms are a major requirement for the development of fast
and robust flow quantification techniques. However, artificial vessel phantoms
often consider only the flow patterns but neglect the elastic properties of the
blood vessels. At this point, tissue engineering poses a potential alternative to
conventional flow phantoms since tissue-engineered arteries have similar
physical properties than native arteries1 and enable the study of
pathologies in controlled environments. In this abstract we present a setup for
studying arterial hemodynamics ex vivo
under realistic conditions. A modular bioreactor platform with a pulsatile
perfusion pump2 was used to mimic the in
vivo blood circulation. Radial acquisitions and self-navigation were
applied for retrospective cine reconstructions. Materials and Methods
Experimental setup
Carotid
arteries were obtained from German landrace pigs and were inserted into a
tissue chamber (Fig. 1). A pulsatile flow with a pressure of 120/80 mmHg,
adjustable flow rates and pump frequencies was realized with an air-driven
perfusion pump2 (Fig. 1 a&b). As fluid VascuLife® Medium (CellSystems
Biotechnologie Vertrieb GmbH) was used.
MR measurements
Measurements
were conducted on a 17.6T MRI scanner with a 1 T/m gradient system and a 24 mm
birdcage coil.
For
flow measurements radial 2D- and 4D-PC MRI sequences were used. The scan
parameters were: VENC=150 cm/s (2D)/167 cm/s (4D). TR/TE=3.0/1.1 ms,
FOV = 25x25x10 mm3 (4D). Morphology was imaged with a flow compensated radial
3D-FLASH sequence (TR/TE=4/1.5 ms, FOV = 25x25x10 mm3). The total measurement
time was 2.4 minutes (2D) and 32 minutes (4D) for flow measurements and 4
minutes for morphology measurement.
Self-navigation
signals extracted from the radial MR signal were used for retrospective
reconstructions, as described previously3. Flow cines were reconstructed
with a frame rate of 120 frames (2D) and 30 frames (4D) per pump cycle. The
spatial resolution was 100 µm (isotropic) for the flow measurements and 98 µm
(isotropic) for the morphology measurement. All reconstructions were conducted
with MATLAB (The MathWorks, Inc., Natick, USA).
PWV and WSS calculation
The
local PWV was determined from the 2D flow measurements using the Q-A method
described previously4. Using the data points of the early upstroke of the
flow pulse, the PWV value can be assessed as the slope of a linear fit of the
Q(A) curve, where Q(t) represents the volume flow and A(t) is the
cross-sectional area of the artery. WSS was calculated from the 3D-velocity derivations
at the vessel wall, which were determined with the 4D flow measurement using
EnSight (CEI Systems, USA) for data processing5.
Results
Self-navigation
signals could successfully be extracted from the ex vivo measurements of carotid arteries. Fig. 2a shows exemplary
signals acquired during 3D flow measurements at 2 different pump frequencies
(0.5 Hz/1 Hz). The strong modulations due to the pulsatile flow are clearly
visible. The pulsatile behavior is also reflected by the results of the 2D- (Fig.
2b&c) and 4D flow measurements (Fig. 2d). Figure 3 displays a volume
rendering of the 3D morphology measurement. The local PWV was calculated at 4
different locations of the artery (Fig. 4). The mean PWV value over all slices
was 5.7±0.5 m/s, which corresponds well with literature6. Figure 5 displays
the calculated WSS for six phases of the pump cycle. The values are in good
accordance with the results reported previously for porcine carotid arteries in vivo7. Discussion and Conclusion
This abstract demonstrates a
suitable setup to study the hemodynamics of arteries ex vivo under physiological as well as pathological pulsatile flow
conditions, e.g. high pressure values and exceeding shear rates. Since the
proposed platform does not require additional trigger signals for
synchronization with the MR measurements, it can be implemented easily on every
MR scanner. This setup can also be used as a flow phantom for validation and
optimization of new sequences and sequence parameters. In future applications,
this platform is considered for measurements in tissue engineered arteries,
which provide a suitable in vitro
test system for atherosclerosis research. It offers a great possibility to
examine the effects of specific biological modifications on the physical,
flow dynamic properties as well as changes in vessel wall morphology in a fully
controllable environment.Acknowledgements
This work was supported by grants from the Deutsche
Forschungsgemeinschaft (SFB 688 B5, Z2), the Bundesministerium für Bildung
und Forschung (BMBF01 EO1004) and the Comprehensive Heart Failure Center (CHFC).References
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