Graph theoretical network properties, while successful in exploring topological features of entire brain networks, have limited sensitivity to localized disease effects. This work explores the role of node strength as an objective way to characterize disease. Differences in the default mode network (DMN) between a cohort of relapsing-remitting multiple sclerosis (RRMS) patients and healthy controls (HC) have been explored using standard graph metrics (e.g. efficiency) and node strength. No differences in graph metrics were observed between the groups; however several key regions of the DMN had a significantly reduced strength in RRMS than HC (5% significance level).
Graphs representing brain networks are useful for visualizing connectivity and exploring the effects of different pathologies beyond localized damage. Graph theory provides composite measures that encapsulate topological properties of the whole network, and has successfully demonstrated disrupted connectivity in a range of diseases1,2,3. Graph theoretical properties are directly dependent on connection (edge) locations and weights; however, because information is typically condensed into a single measure, some local information (for instance which regions and connections are most affected in pathologies) is lost. As a result, the sensitivity of these properties to subtle, localized disease effects is reduced. It is possible to examine the integrity of each edge individually (e.g. number of streamlines of a connection), or a property of the node itself (e.g. cortical thickness) and investigate the correspondence with clinical outcomes.
Crucially, neither graph theoretical properties nor individual edges are able to capture the possible cascade effect that several damaged edges may have on a specific node. While centrality measures, for instance, are based on the presence or absence of a connection, it may be that connection integrity, and the identification of nodes that are impaired by one or more damaged connection, is more informative.
This work is an exploratory study to assess the validity of a property less explored, i.e. node strength, obtained by assigning to each node the composite weight of all its connected tracts. This approach has the advantage of retaining local information while reducing the need for excessive multiple comparisons unavoidable when testing individual edges. Moreover it could highlight nodes that, while not directly affected by the disease, may be connected to several affected edges; these nodes may then become pivotal in disease mechanisms. The various levels of analyses (graph topological properties, individual edge weights and node strength) are applied in a pilot study of relapsing-remitting multiple sclerosis (RRMS) patients and healthy controls (HCs).
Data from 23 HCs (14 female; mean age 37.9 ± 12.6 years) and 22 RRMS patients (18 female; mean age 42.8 ± 10.7 years; mean disease duration 14.0 ± 8.4 years; median EDSS score 2.0) were recruited. Diffusion-weighted (DW) and T1-weighted images were acquired using an Achieva 3T MR scanner (Philips Healthcare, Best, Netherlands) with a 32-channel head coil (Table 1). Tractography was performed between 48 regions generated from a parcellation4,5 in and around the default mode network (DMN) using TractoR6 (Table 2 gives correspondences between node number and parcellation); average group-wise brain graphs, weighted using fractional anisotropy (FA), were created (Figure 1).
A non-parametric permutation test was adopted7 to investigate network differences between groups. Differences in topological properties (global and local efficiency, mean clustering coefficient and characteristic path length), individual edge weights and node strengths were explored.
No differences in the topological properties of HC and RRMS networks were seen (at 5% significance level) (Figure 2); however approximately 23% of edge weights were found to be significantly different between groups (Figure 3a), 19% of which remained significant at 1% significance level (p<0.01) (Figure 3b).
When considering node strength, eight specific regions demonstrated a significant difference (strength lower in RRMS) between groups at 5% significance level (Figure 3).
Graph properties of RRMS patients were not altered compared to HCs in this cohort, possibly reflecting the minimal disability of the RRMS group; individual edge weights, though, demonstrated sensitivity to disease effects. Interestingly, several nodes with different strength characteristics between groups correspond to core DMN regions8, such as the posterior cingulate gyrus (node 35) and precuneus (nodes 36, 37). These are densely connected areas with a higher probability to be linked to damaged edges.
A key feature of this approach is to be objective in assessing damage. Future work will explore node strength using several weights and MS subtypes, correlating node strength and lesion topology.
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