Jasmien Orije1, Geert De Groof1, Elisabeth Jonckers1, Sander Raymaekers2, Marleen Verhoye1, Veerle Darras2, and Annemie Van der Linden1
1Bio-Imaging Lab, University of Antwerp, Wilrijk, Belgium, 2Laboratory of Comparative Endocrinology, KU Leuven, Leuven, Belgium
Synopsis
The effect of thyroid
hormones on seasonal neuroplasticity in adult songbirds was never investigated
before. To study this we treated photorefractory starlings with methimazole,
which resulted in a depletion of thyroid hormones plasma levels. Using repeated
measures of in vivo DTI and song recordings, neuroplasticity and song behaviour
were monitored over different photoperiods. We found indications that thyroid
hormones can modulate the seasonal neuroplasticity both directly at the level
of the song control system, as suggested by direct changes in song behaviour
after methimazole-treatment, and indirectly via the
hypothalamus-pituitary-gonad axis, as methimazole-treatment inhibited the
testosterone increase upon photostimulation.
Purpose
One of the most dramatic examples of adult neuroplasticity is
the seasonal plasticity that occurs in the song control system (SCS) of
songbirds. The primary factor inducing this neuroplasticity is the
photoperiodic induced increase in testosterone. However, it has been shown that
steroid-independent photostimulation can also induce SCS plasticity. One of the
proposed alternatives is the mediating effect of thyroid hormones (THs),
as THs are associated with neurogenesis and play an important role in the
regulation of seasonal reproduction. Prior studies1-3 showed that
photostimulation induces an upregulation of thyroid stimulating hormone
and type 2 deiodinase, resulting in increased active T3 levels which modulate gonadotropin-releasing hormone neurons leading to an activation of the hypothalamus-pituitary-gonadal
(HPG) axis. We investigated whether THs are
required to induce SCS plasticity and whether THs could affect the seasonal
neuroplasticity (1) directly at the level of the SCS by binding to TH
receptors or (2) indirectly via the HPG axis.
Methods
Thirty photorefractory male starlings were divided into a hypothyroid (n=16) and a control group (n=14). Hypothyroidism was induced by adding 0.05% methimazole (MMI) to the drinking water upon switching from long days (16L:8D) to short days (8L:16D). Starlings followed a photoperiodic regime where they became subsequently photorefractory, photosensitive and photostimulated. Structural neuroplasticity was monitored over these different photoperiods by acquiring MRI images on a 7T Bruker MR system equipped with a 400 mT/m gradient insert. Each imaging session consisted of a T2-weighted 3D anatomical RARE scan (TR: 2000 ms; TE: 11 ms; RARE factor: 8; zerofilled to a matrix of (256x92x64) with voxel resolution (0.089x0.25x0.25)mm³) followed by a DTI scan (TR: 7000 ms; TE: 23 ms; d 4ms, D 12ms; b-value 670 s/mm²; 60 diffusion gradient directions; spatial resolution: (0.179x0.179x0.35)mm³; 28 horizontal slices). Throughout the entire imaging procedure, respiration rate and temperature were kept within narrow physiological ranges (41.0 ± 0.2)°C. All datasets were spatially normalized to a population-based template after which a whole-brain voxel-wise repeated measures two-way ANOVA was performed on the smoothed Fractional Anisotropy (FA) and Mean Diffusion (MD) maps (SPM12). Based on the anatomical contrast of the FA maps AreaX, RA and the HVC-RA tract were manually delineated using Amira 5.4. At each time point blood samples were taken to determine plasma levels of T3, T4 and testosterone. At each photoperiod songs were recorded and the song bout length and singing rate were quantified as a measure of the singing behavior.Results & Discussion
MMI treatment successfully depleted TH plasma levels, while in the control group TH levels decreased during the photosensitive phase and increased again upon photostimulation (Fig. 1A). Interestingly, MMI treatment also inhibited the increase in testosterone levels upon photostimulation, indicating that hypothyroidism affects the HPG axis activation (Fig. 1B). A voxel based analysis of the MD and FA maps showed a widespread increase over time located in the nidopallium and HVC respectively, in both groups (Fig. 2). Similarly, AreaX and RA volumes increased gradually over time in both groups. These neuroplasticity changes started already during the photosensitive phase. The FA value of the HVC-RA tract increased significantly upon photostimulation in the control group, but not in MMI-treated starlings. This could be due to the decreased levels of both THs and testosterone, as testosterone is known to play a role in the increased connection between HVC and RA4. Analysis of the song recordings showed that the average singing rate increased gradually over time in both groups (Fig. 3B). The song bout length increased upon photostimulation in the control group, while in MMI treated birds song bout length already increased during the photosensitive phase (Fig. 3A). This indicates that hypothyroidism can directly affect the singing behavior, irrespective of increased testosterone concentrations and photostimulation.Conclusion
Based on these results we have found indications that THs can modulate the SCS neuroplasticity both directly at the level of the SCS, as suggested by the direct changes in the song behavior of MMI-treated starlings, and indirectly via the HPG axis, as MMI treatment inhibited the testosterone increase upon photostimulation. This inhibition of testosterone increase could explain the absence of the increased connection between HVC and RA. However, additional experiments are required to discriminate fully between the effects of T and THs, as the current treatment affected both testosterone and TH levels. Acknowledgements
This
research is supported by a Belspo IAP
grant (PLASTOSCINE: P7/17) from the Belgian Science Policy Office and by grants
from the Research Foundation – Flanders (FWO, project Nr
1115217N, 1274714N, 12R1917N and
G030213N).References
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