There is a need to establish robust quantification pipelines to analyze 129Xe ventilation MRI for multi-center studies. Moreover, there is increasing interest in quantifying not only ventilation defect percent, but also regions of low and high ventilation. To this end, we sought to determine inter-method agreement between two different semi-automated quantitative mapping approaches — linear binning and adaptive K-means. The results suggest that once bias field corrections are applied consistently, both ventilation analysis methods agree well when classifying ventilation into 4 bins. Thus, with key steps outlined here, either method can be readily deployed in multi-center studies.
Ten controls (25.7±3.4 years) and 19 asthma subjects (45.1±20.4 years) were enrolled in HIPAA-compliant studies with IRB approval. Imaging was performed on a 1.5T EXCITE 15M4 MR system (GE Healthcare) using HP 129Xe with a 2D multislice GRE sequence: 40cm FOV, 12.5mm slice, matrix=128x128, 7-10° flip, TR/TE=8.1/1.9ms.
Components of the quantification pipeline shared between both algorithms are summarized as follows: (1) registration of the proton to the 129Xe image, (2) joint segmentation of the proton and 129Xe images to obtain a binary mask that included both the thoracic cavity and trachea, (3) sorting of the 129Xe images by acquisition order as well as intensity correction using N4BiasCorrection3 using the binary mask, and (4) segmentation of the pulmonary vasculature using a vesselness filter4.
Although both pipelines used the same intensity corrected 129Xe and the same lung mask with vasculature removed, the gas image was either classified by linear binning1 or adaptive K-means2. Each independently determined VDP as well as 3 signal intensity levels above VDP defined to reflect internally referenced ventilation levels: low-, medium-, and high- ventilation percent (LVP, MVP and HVP) respectively. These additional 3 classes are being explored for their possible utility in further enhancing the clinical interpretation of 129Xe MRI. The signals from trachea were specifically retained and used for histogram scaling in linear binning, whereas they were excluded in adaptive K-means due to the possibility of extreme intensities influencing cluster assignment (Fig.1).
Bland-Altman plots were used to compare whole lung (global) agreement of the metrics, while Dice coefficients were used to measure their spatial agreement. The Wilcoxon rank-sum test was used to compare between control and asthma for global measures from each method independently.Typical examples of ventilation maps for linear binning and adaptive K-means are shown for a control subject with low SNR (Fig.2) and an asthmatic subject with high SNR (Fig.3). In both cases spatial agreement for each cluster was excellent. The primary difference was slightly expanded LVP and HVP derived from adaptive K-means vs. linear binning.
Quantitative comparison of asthma vs. control subjects was similar in both methods (Table 1). Both showed significantly greater VDP and lower MVP in asthma (p<0.04). However, linear binning found a significantly higher LVP (the ventilated cluster immediately above VDP) in asthma compared to an insignificant increase by adaptive K-means (Table 1). The overall Dice coefficients were 0.4, 0.7, 0.9 and 0.8 for VDP, LVP, MVP, and HVP respectively. Bland-Altman analysis found negligible bias between the linear binning vs. adaptive K-means for VDP (-1.5%), but moderate bias for LVP (5.7%), MVP (-8.1%), and HVP (4.0%) (Fig.4).
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