William M Spees1,2, Tsen-Hsuan Lin1, Peng Sun1, Sam E Gary1, and Sheng-Kwei Song1,2
1Mallinckrodt Institute of Radiology, Washington University School of Medicine, St. Louis, MO, United States, 2Hope Center for Neurological Disorders, Washington University School of Medicine, St. Louis, MO, United States
Synopsis
Because of its
exceptionally robust nature, the ex vivo frog sciatic nerve has been the subject
of numerous electrophysiology and MRI studies over the years. Here we report on
diffusion MRI signal changes resulting from 50 and 100 Hz in-magnet electrical
stimulation of perfused bullfrog sciatic nerves. The inexpensive perfusion
system we have implemented allows for good long-term in-magnet stability and
simultaneous MRI/electrophysiology studies. Decreases in water diffusivity and compound
action potential conduction velocities accompany prolonged periods of
repetitive electrical stimulation. Both of these changes are consistent with
hypothesized microstructural alterations of the PNS myelin.
Introduction
Previously we
demonstrated the utility of diffusion fMRI to assess function in the mouse
optic nerve in vivo.1,2 In the current study, we developed and
implemented a perfusion system that allows for electrical stimulation and
compound action potential recording from ex vivo perfused frog sciatic nerves.
This setup allows for well-defined stimulus intensity (in terms of the
frequency of a supramaximal voltage stimulus) and recording of compound action
potentials (CAP) and diffusion fMRI imaging data. Perfused nerves were fixed
in-magnet in attempts to “lock in” microstructural alterations, which we hope
will be detectable with electron microscopy (currently ongoing). Methods
Freshly-harvested sciatic nerves from jumbo bullfrogs were used. Nerves
were held in a perfusion flow cell fitted with retractable suction electrodes
for in-magnet stimulation and recording (Figure 1). Electrical stimulation was
delivered as 100 μs voltage pulses
from an Agilent 33210A Pulse/Function Generator into an A-M System model
2200 Analog Stimulus Isolator. The recorded compound action potential was
amplified (gain = 1000×) with a home-built amplifier and averaged with a
Tektronix TBS1052B oscilloscope. Recorded CAP traces were transferred to a
laptop computer for offline analysis (Figure 2). Oxygen-saturated frog Ringer’s
solution flowing at a rate of 3 mL/hr is estimated to provide an ~ 20-fold
excess of O2 above that required even during stimulation at 100Hz.3,4
Diffusion fMRI studies were carried out in a 4.7 T Agilent small-animal imaging
system using home-built RF coils. Diffusion MRI data (25-direction, with
maximum b value of 3.1 ms/μm2, δ = 5 ms, Δ = 18 ms) was acquired
with a multi-echo spin-echo diffusion-weighted imaging sequence.5 To increase temporal
resolution with the spin-echo-based imaging, data was acquired with a 32×32 matrix (7×7 mm2 fov).
With TR = 1.5 s and 25 diffusion directions, this acquisition scheme results in
20-minute temporal resolution. A Hamming window was applied to the raw image
data prior to zero-filling to 128×128 and denoising.6
The smaller matrix size resulted in an approximately 10% baseline offset
(increase) in the ADC when compared to data
acquired on a 64×64 matrix from the same
nerves.
Results
The CAP conduction velocity decreases across a period of repetitive
electrical stimulation, as shown in the example (Figure 2). Accompanying the conduction velocity changes was a decreased ADCperp (the apparent diffusivity perpendicular to the axonal fibers). A 24-minute period of 50-Hz stimulus produced a 4% decrease in ADCperp (Figure 3). It is interesting that over the 3-hour period of observation post-stimulation, there is little tendency for the ADC to return to baseline levels. A subsequent 24-minute period of 100 Hz stimulation results in a further 5% decrease in ADCperp, after which there is some tendency to increase back to baseline levels (Figure 3). Stimulation at 100 Hz×40 min also results in a strong tendency to return to baseline ADC levels (data not shown), as if the PNS tissue exhibits an elastic response once tissue microstructure is altered significantly enough. A series of runs (Figure 3, inset) was also performed with immediate, in-magnet fixation. At the time of this writing, the tissue samples are being prepared for electron microscopy. In Figure 4, using sutures to tie off a perfused nerve, we demonstrate how the diffusion fMRI technique might be used to pinpoint a lesion site that results in CAP conduction blockage, even were the lesion not visible by other methods. In regions conducting repetitive electrical signals (upper portion) ADCperp decreases, while just distal to the conduction blockage this is not the case.
Discussion
Taken alone, the diffusion
MRI changes observed with repetitive electrical stimulation in the perfused
frog sciatic nerve suggest an altered tissue microstructure. Indeed, three
decades ago the production of (reversible) intramyelinic paranodal vacuoles
with repetitive electrical stimulation was demonstrated in the in situ fixed frog sciatic nerve.7 This may
be a general phenomenon, as exposure of potassium voltage gated channels
(typically sequestered underneath the paranodal myelin) has been reported in
repetitively-stimulated rat sciatic nerves.8 Our own in-magnet
electrophysiology data demonstrates a decreasing propagation
velocity for the compound action potential with repetitive electrical
stimulation (cf. Figure 2). A swelling of the paranodal myelin, should it
increase the exposed axonal membrane surface area at the nodes of Ranvier,
would increase the axonal membrane capacitance (Cn) at the nodes.
This, in turn, would be expected to increase the RC time-constant, τ, required to charge the nodal surface to
the action-potential-firing threshold (τ = Ri·Cn; here Ri is the
resistance to current flow through the intra-axonal cytoplasm).9
This provides a biophysical rationale for the observed decrease in CAP
conduction velocity.Acknowledgements
Supported in part by NIH R01-NS047592, P01-NS059560, U01-EY025500 and NMSS RG 5258-A-5.References
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