Vadim Malis1, Usha Sinha2, Robert Csapo3, and Shantanu Sinha3
1Physics, UC San Diego, San Diego, CA, United States, 2Physics, San Diego State University, San Diego, CA, United States, 3Radiology, UC San Diego, San Diego, CA, United States
Synopsis
The disproportionate loss of muscle force in comparison to
loss of muscle mass with age remains unexplained. Recent studies indicate that the remodeling
of the extracellular matrix (ECM) may disrupt lateral force transmission
pathways mediated by the ECM. Shear
strain is the mechanism that supports lateral transmission of force. We quantified shear strain in muscle from the
strain rate tensor derived from velocity encoded phase contrast dynamic images
of the in-vivo human calf muscle
under isometric contractions. The
maximal shear strain was significantly lower in the older cohort compared to
the younger cohort which potentially identifies that lateral force transmission
decreases with age.
Purpose
Aging results in muscle force loss disproportionate to the
loss of muscle mass1. Recent
studies indicate that compromised lateral transmission of force pathways could
potentially contribute to the force loss with age2. Shear strain in the endomysium has been
postulated as the most likely mechanism of lateral force transmission3
and can be derived from strain rate tensors computed from velocity encoded
phase contrast images. This study explores
the age related shear strain in the medial gastrocnemius (MG) from a series of
velocity encoded phase contrast (VE-PC) images acquired under isometric
contraction. Methods
Force measurements and Magnetic Resonance Imaging
studies were performed on nine young (27.5 ± 4.8 years,
height: 159.6 ± 6.5 cm,
mass: 54.6±7.3 kg) and
eight senior (77.6 ± 7.3 years, height: 154.3 ± 2.9 cm,
mass: 57.9 ± 3.7 kg) female subjects recruited after IRB approval.
Dynamic images of the lower leg were acquired during isometric contraction using
a gated VE-PC sequence on a 1.5-T GE scanner with a specially designed 8-Ch
phased array coil, and the leg in a plaster cast4. Images were velocity
encoded in all three directions, and collected in 22 phases, 7 oblique-sagittal
contiguous slices, 5mm thick and 1.7x1.7 mm in-plane resolution. Subjects were
provided visual feedback within the scanner to maintain consistent contractions
at ~40% MVC. Muscle fascicles were manually delineated on water-suppressed
images to obtain fiber orientation during the dynamic cycle and tracked using
VE-PC. The symmetric part, of the
strain rate tensor SR tensor was calculated from the spatial gradient tensor, and diagonalized; eigenvectors corresponding to the positive (PEV) and negative
(NEV) eigenvalues were analyzed separately. The SR tensor in the principal axis basis (no
off-diagonal terms) was rotated to that of the muscle fiber (fascicle
direction) basis (Fig. 1 shows the relationship between the two basis as well
as the source of shear strain). Maximum
values of shear strain were then obtained by rotating the tensor from the
muscle fiber basis by 45o. Results
Fig. 2 shows the maps of the strain along the
muscle fiber (SRfiber or SRff), in the fiber
cross-section (SRin-plane or SRcc) and shear strain in
the fiber basis (SRfc) from one young and one old subject at the
peak of the contraction cycle. The maps of
the shear strain clearly show decreased values in the older subject (Fig. 2). Table 1 lists the shear strain in the muscle
fiber basis as well as the maximum shear basis for the ROI placed at proximal,
middle, and distal regions.
While a decrease in shear strain was seen in the
muscle fiber basis, significant differences [F(1,34) = 10.302, P=0.003] were
only seen in the maximum shear strain basis.Discussion
Force generated in the muscle fibers is
transmitted longitudinally through the myotendinous junction and laterally
through myofascial pathways. While the
contractile and neural determinants of force loss with age have been studied
extensively, very little is known about lateral transmission of force. The
lateral transmission of force was shown to decrease in a rodent model of aging
and accounts for as much as 50% of the force loss with aging. However, the challenge is that there are no
non-invasive approaches to determine lateral transmission of force. This report on shear strain is the first
non-invasive marker of lateral force transmission and is a reflection of
extracellular matrix (ECM) remodeling that occurs with age. ECM remodeling
includes increase in the width of the endomysium and perimysium as well as in
the stiffness of the ECM (potentially from abnormal deposition of collagen).
The decrease of shear strain with age indicates that some of the force loss in
the aging cohort could be attributed to a reduction in lateral force transmission. Conclusions
The potential to
determine lateral transmission of force non-invasively offers a powerful tool
to monitor the functional consequences of structural remodeling of the ECM and is
one that will enable the design of optimum rehabilitative strategies.Acknowledgements
This work was supported by National Institute of Arthritis and Musculo- skeletal and Skin Diseases Grant 5RO1-AR-053343-08.References
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