Dynamic 13C data were acquired following injection of hyperpolarized [1-13C]pyruvate from 3 patients previously diagnosed with glioblastoma. Pyruvate, lactate and bicarbonate signal with high SNR were detected in human brain. Lactate/Pyruvate appeared to be relatively high in the contra-lateral, normal appearing brain. In contrast, tumor regions produced higher Lactate/Bicarbonate than contra-lateral brain. The contrast-enhancing lesion of one patient, who underwent surgical resection shortly after 13C imaging due to suspected recurrence, produced a relatively low level of Lactate/Pyruvate compared to contra-lateral brain and Lactate/Bicarbonate similar to the value in contra-lateral brain. Subsequent biopsy of the contrast-enhancing lesion indicated treatment effect.
HP 13C MRSI were able to detect metabolic processes in human brain: Figure 1 shows an example of HP 13C spectra from a patient with a maximum SNR of 320 for pyruvate and 180 for lactate. The original 13C spectra (Figure1B) were intensity-corrected using the coil sensitivity map obtained by summing all metabolites over all time points (Figure1C). The flip angle- and intensity-corrected dynamic pyruvate and lactate signal demonstrated the spatial and temporal evolution of 13C-labeled pyruvate and lactate resonances in normal appearing brain (Figure2). The pyruvate appeared in multiple regions, with the maximum pyruvate in the cavernous sinus (Figure2A) or superior sagittal sinus (Figure2B) appearing approximately 9s from the start of data acquisition. Maximum lactate signal was observed approximately 15-18s from the start of data acquisition. The Kpl shows similar values in the normal-appearing brain from 3 patients (right panels in Figure2A,2B,2C).
HP 13C MRSI was able to detect bicarbonate with high SNR: Figure 3 shows an example of bicarbonate signals, with peaks being observed in the raw spectra (Figure3B), as well as the intensity-corrected spectra (Figure3C). The ability to detect bicarbonate (Figure3D,3E) suggests that HP [1-13C]bicarbonate may be useful for studying the metabolism of not only glioma, but of a number of other neurological diseases.
HP Lac/Bicarb and HP Lac/Pyr show different aspects of tumor metabolism: Figure 4 shows comparisons between Lac/Bicarb and Lac/Pyr for 3 patients. The contra-lateral normal-appearing brain produced higher or similar levels of Lac/Pyr to those in the anatomic lesion (Figure4B,4D,4F). In contrast, regions of suspected tumor demonstrated higher levels of Lac/Bicarb than the contra-lateral normal appearing brain in 2 of the patients (patient#2,#3 in Figure4).
The lesion that had lower levels of HP Lac/Bicarb and Lac/Pyr was associated with treatment effect: Patient #1 presented with findings from a prior MRI that were suspected of recurrent tumor (see Figure4). This patient exhibited high Lac/Pyr throughout normal-appearing brain, but relatively low Lac/Pyr in the enhancing lesion (Figure4B). The level of Lac/Bicarb in the enhancing lesion was similar to that in normal-appearing brain (Figure4A). Analysis of tissue obtained from the enhancing lesion at a subsequent resection confirmed the diagnosis of treatment effect with predominant microglia//macrophages (Figure5).
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