Synopsis
The rodent auditory
system has been a popular research subject for electrophysiological studies for
its complexity, fine tuning and adaptability. More recently, some studies on
auditory Functional Magnetic Resonance Imaging (fMRI) in rats have surfaced, aiming
to unravel this system’s intricacies by capturing whole brain activity
noninvasively. Auditory mapping in the mouse could be highly valuable given its
importance vis-à-vis transgenic models and optogenetics. This study provides
the first tonotopic mapping in the in vivo mouse via high resolution fMRI. We
demonstrate robust activation in the auditory pathway, and specific tonotopy in
several prominent regions along the pathway.
Purpose
Whereas
human tonotopy has been extensively studied via fMRI [1-4], rodent tonotopy is
still in its infancy. Rat experiments have been recently presented [5-12], but
–to our knowledge– auditory mapping in mice has never been performed. We here
studied the mouse auditory pathway in-vivo, and in particular, BOLD-derived
tonotopic maps.
Methods
Sound delivery system: A dedicated system for delivering accurate
auditory stimuli was custom designed. Briefly, the system consisted of: (1) a
Yamaha AG-03 soundboard allowing playback of sounds up to 96 kHz (sample rate
of 192 kHz) with a dynamic range of 24 bits and 2.451 V RMS output before
clipping; (2) an in-house designed voltage amplifier capable of a 3x
amplification up to 24 Vpp; (3) L010 Kemo piezoelectric speaker capable of
producing ultrasonic sounds at high output levels (above 100 dB) with a
relatively flat frequency response up to 75 kHz. The emitted sounds were then
delivered via custom tubing involving nylon and tygon tubes, interfaced with
custom 3D-printed PET parts to a single ear. A Brüel and Kjær 4939-A-011,
free-field microphone with a Type 2670 preamplifier was connected directly into
the Yamaha Soundboard and placed 1 mm away from the auditory setup to calibrate
and test the system’s performance and to record scanner-induced sounds.
MRI setup: All
experiments were pre-approved by the Institute’s animal ethics committee.
Experiments were performed on a Bruker BioSpec 9.4T scanner with a 20 cm bore
and gradients capable of producing up to 66 G/cm in all directions. For
transmittance, an 86 mm volume resonator was used, whereas a 4-channel array
cryocoil was used for signal detection. Male mice (c57bl/6, N=6) weighing
approximately 25 gr were briefly anesthetized with 4% isoflurane and then
quickly placed under a medetomidine sedation regime (bolus 0.4 mg/kg then 0.8 mg/kg/h, continuous
infusion) as described in [13], whereupon the isoflurane was discontinued.
Animal physiology including transcutaneous CO2, rectal temperature, and
breathing rate were constantly monitored. The animals breathed air enriched
with Oxygen (27%) measured by a oxygen Sensor (Viamed, United Kingdom)
throughout the experiments. One ear was carefully sealed with wax and the Tygon
tube was placed within the other ear, allowing the delivery of auditory stimuli
preferentially to that ear.
fMRI experiments and analysis: Following
localization scans, fMRI experiments were conducted using an ultrafast FISP
sequence with the following parameters: TR/TE = 2.8/1.4 ms, slice thickness =
650 μm, FOV = 15x15 mm2, matrix size 100x100 leading to an in-plane resolution
of 150x150 (μm)2, and a 1.307 seconds scan time per
volume. The fMRI paradigm consisted of 45 seconds of rest, followed by five
blocks of {15 seconds of auditory stimulation, 45 seconds of rest}, for a total
time of 7.5 min per scan. The auditory stimulus consisted of a pure tone of 5,
12 or 20kHz; animals were allowed to rest between paradigms for ~3 minutes. The
data were analyzed using SPM® (called through fMRat [14]) for mapping
activation in the mouse brain).
Results and Discussion
Figure 1
shows the spectral analysis of the FISP sounds as well as the frequencies
delivered to the ear of the mouse. The 5kHz stimulation show the most
(unwanted) harmonics. The FISP noise, while significant, was still at least
20dB lower compared to the stimuli given, and hence was considered not to
significantly affect the experiment. Figure 2 shows raw data from the
experiment, including a volume from the FISP experiments (upper panel) and
signal time course from a representative experiment (one single run in a single
rat) for three selected ROIs within the auditory pathway (lower panel).
Excellent SNR was documented, and the BOLD response can be observed with the
naked eye. Figure 3 shows t-maps for each frequency; notably, nearly the entire
auditory pathway is highlighted, including its major relays: Auditory nerve,
Superior Olivary Complex (SOC), Lateral Lemniscus (LL), Inferior Colliculus
(IC), and Medial Geniculate Nucleus (MGB), with t-statistics as high as t=19.3.
Auditory Cortex (AC) was not demonstrated in these maps, although ROI analysis
(not shown) suggests it may be active, but with different BOLD dynamics,
thereby reducing its significance levels in Figure 3. Figure 4 shows tonotopic
maps in the more posterior slices, where activation was most prominent.
Tonotopy was observed in several regions, most notably in IC, where three
layers show a clear tonotopic pattern going from dorsolateral regions to
medioventral ones with increasing frequency, confirming the
electrophysiological findings in mice [15] and fMRI in rats [7]. This study
thus bodes well for auditory pathway mapping in the in-vivo mouse in models of
plasticity / aberrations. Acknowledgements
The authors acknowledge funding from the European
Research Council (ERC) under the European Union’s Horizon 2020 research and
innovation programme (Starting Grant, agreement No. 679058). We thank Dr.
Daniel Nunes for technical assistance. References
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