White Matter Imaging: Emerging Techniques
Susie Yi Huang1

1Department of Radiology, Massachusetts General Hospital, Boston, MA, United States

Synopsis

In the past decade, rapid advancements in MR technology have resulted in more sophisticated and higher strength magnets and gradient systems, introduction of new imaging sequences, and refinement of existing protocols that have furthered our capability to probe cerebral white matter microstructure. These newer methods have the potential to expand our understanding of white matter on a microscopic level. I will discuss emerging techniques for imaging white matter based on the main structural components of white matter from the inside out, focusing on methods for studying axonal microstructure and the quantification of myelin.

Introduction

Cerebral white matter develops rapidly during the perinatal period and continues to mature in an orderly pattern that parallels evolving neural functionality. Understanding the changes that occur in white matter throughout the lifespan is therefore essential to understanding brain function in states of health and disease. In the past decade, rapid advancements in MR technology have resulted in more sophisticated and higher strength magnets and gradient systems, introduction of new imaging sequences, and refinement of existing protocols that have furthered the capability to probe cerebral white matter microstructure. These newer methods have the potential to expand our understanding of white matter on a microscopic level. I will discuss emerging techniques for imaging white matter based on the main structural components of white matter from the inside out, focusing on methods for probing axonal microstructure and the quantification of myelin.

Characterizing axonal microstructure by diffusion MRI

Axons are the structural and physiological conduit for signal transmission in the brain and are one of the fundamental elements of brain function. The diameter of both myelinated and unmyelinated axons is related to the speed at which action potentials are conducted along the length of the axon (1, 2). Variations in axon diameter are thought to be closely tied to function, with networks that demand fast response times (such as motor networks) demonstrating larger axon diameters. Therefore, non-invasive methods for mapping axon diameters in vivo would provide new insight into brain function and connectivity.

Recognizing the potential impact of an MRI technique to map axon diameters, several groups have started to exploit the sensitivity of diffusion-weighted MRI (DW-MRI) to tissue microstructure for the purpose of estimating axon diameter distributions and fiber density in white matter bundles (3-15). DW-MRI is well-established clinically and plays a key role in the diagnosis of several neurological conditions including acute stroke (16-18) and the evaluation of brain tumors (19, 20) and traumatic brain injury (21, 22). DW-MRI is also used to map the orientation of white matter tracts, which can be achieved by measuring diffusion along multiple orientations and applying an analysis scheme such as diffusion tensor imaging (23), high-angular resolution diffusion imaging (HARDI) (24), q-ball (25) or diffusion spectrum imaging (26). It is only more recently, however, that there has been a heightened focus on using DW-MRI measurements to quantify the size of restrictive spaces in brain tissue.

A number of diffusion MRI techniques have emerged in the last decade that focus on the quantification of axon diameter and packing density in white matter (3-15). This information is obtained by taking a series of diffusion-weighted MR signals measured over a wide range of diffusion weightings (q-values or diffusion-encoding gradient areas) and diffusion times (time between diffusion-encoding gradients). At different diffusion times, different populations of axons exhibit restricted diffusion, thereby allowing axons of varying sizes to be probed. A model of restricted diffusion within axons and hindered diffusion outside the axons is then fit to the data. The sensitivity of axon diameter mapping techniques to small diameter axons is limited by the maximum gradient strength of clinical MR scanners (27, 28). The recent availability of higher maximum gradient strengths on human MRI scanners (29-32) has enabled the translation of these methods from animal (8, 9, 27, 33) and ex vivo studies (6, 10, 11, 13, 15) to the in vivo human brain (10, 12, 14, 28, 34). Such technological advancements offer the possibility of in vivo diffusion microscopy with unprecedented resolution of fine white matter structures (32) and micron-sized axons (28, 34) in the living human brain for the study of multiple sclerosis (MS) (35) and other neurological disorders affecting white matter. Related techniques such as NODDI measure orientational dispersion and neurite density (36), which has been shown to provide better distinction of microstructural disruption in MS lesions and normal-appearing white matter compared to conventional DTI metrics (37).

Quantification of myelin and the g-ratio

Myelination increases conduction velocity along an axon through a mechanism known as saltatory conduction. For myelinated axons, ion channels and action potentials occur only at the gaps between the myelin, known as the nodes of Ranvier. Between these nodes of Ranvier, the current flows passively through the insulating myelinated portions, leading to an increased rate of conduction. Larger axons and thicker myelin sheaths contribute to faster conduction, but there is a trade-off between axon size and myelin thickness due to spatial constraints imposed by the size of the brain. The relationship between axon size and myelin thickness is captured in a parameter known as the myelin g-ratio, defined as the ratio of the inner (axon) to the outer (axon plus myelin) diameter of the fiber. The optimal g-ratio for maximizing conduction speed is around 0.6 (38).

Determining the g-ratio noninvasively by MRI could provide important information regarding the macromolecular structure of white matter not available through other imaging approaches. The g-ratio can be calculated based on knowledge of the myelin volume fraction and the axon volume fraction (33, 39). Information regarding the axon volume fraction is available through the diffusion MRI methods described above, whereas myelin volume fraction can be determined through a number of MRI biomarkers, including those based on T1/T2 relaxometry measurements (40, 41) and magnetization transfer (MT) (42, 43). A number of quantitative MT methods have been developed in recent years (44, 45) based on the magnetization transfer ratio (MTR) (46, 47). The goal of quantitative MT measurements is to determine the proportion of protons that are bound to macromolecules and to quantify the rate of exchange of magnetization between bound and free protons. Quantitative MT methods will be surveyed and their strengths, weaknesses and applications to quantifying myelin content in white matter will be discussed.

Acknowledgements

This work was supported by NIH U01MH093765, NCRR P41EB015896, NINDS 1K23NS096056, and an RSNA Research Resident Grant. The author thanks Drs. Lawrence Wald, Jennifer McNab, Thomas Witzel and Eric Klawiter for helpful discussions on this material.

References

1. Hoffmeister B, Janig W, Lisney SJ. A proposed relationship between circumference and conduction velocity of unmyelinated axons from normal and regenerated cat hindlimb cutaneous nerves. Neuroscience. 1991;42(2):603-11.

2. Hursh JB. The properties of growing nerve fibers. American Journal of Physiology. 1939;127(1):140-53.

3. Stanisz GJ, Szafer A, Wright GA, Henkelman RM. An analytical model of restricted diffusion in bovine optic nerve. Magn Reson Med. 1997;37(1):103-11.

4. Assaf Y, Freidlin RZ, Rohde GK, Basser PJ. New modeling and experimental framework to characterize hindered and restricted water diffusion in brain white matter. Magn Reson Med. 2004;52(5):965-78.

5. Assaf Y, Basser PJ. Composite hindered and restricted model of diffusion (CHARMED) MR imaging of the human brain. Neuroimage. 2005;27(1):48-58.

6. Assaf Y, Blumenfeld-Katzir T, Yovel Y, Basser PJ. AxCaliber: a method for measuring axon diameter distribution from diffusion MRI. Magn Reson Med. 2008;59(6):1347-54.

7. Alexander DC. A general framework for experiment design in diffusion MRI and its application in measuring direct tissue-microstructure features. Magn Reson Med. 2008;60(2):439-48.

8. Ong HH, Wright AC, Wehrli SL, Souza A, Schwartz ED, Hwang SN, et al. Indirect measurement of regional axon diameter in excised mouse spinal cord with q-space imaging: simulation and experimental studies. Neuroimage. 2008;40(4):1619-32.

9. Barazany D, Basser PJ, Assaf Y. In vivo measurement of axon diameter distribution in the corpus callosum of rat brain. Brain. 2009;132(Pt 5):1210-20.

10. Alexander DC, Hubbard PL, Hall MG, Moore EA, Ptito M, Parker GJ, et al. Orientationally invariant indices of axon diameter and density from diffusion MRI. Neuroimage. 2010;52(4):1374-89.

11. Ong HH, Wehrli FW. Quantifying axon diameter and intra-cellular volume fraction in excised mouse spinal cord with q-space imaging. Neuroimage. 2010;51(4):1360-6.

12. Zhang H, Hubbard PL, Parker GJ, Alexander DC. Axon diameter mapping in the presence of orientation dispersion with diffusion MRI. Neuroimage. 2011;56(3):1301-15.

13. Panagiotaki E, Schneider T, Siow B, Hall MG, Lythgoe MF, Alexander DC. Compartment models of the diffusion MR signal in brain white matter: a taxonomy and comparison. Neuroimage. 2012;59(3):2241-54.

14. Ferizi U, Schneider T, Tariq M, Wheeler-Kingshott CA, Zhang H, Alexander DC. The importance of being dispersed: A ranking of diffusion MRI models for fibre dispersion using in vivo human brain data. Med Image Comput Comput Assist Interv. 2013;16(Pt 1):74-81.

15. Morozov D, Bar L, Sochen N, Cohen Y. Modeling of the diffusion MR signal in calibrated model systems and nerves. NMR Biomed. 2013;26(12):1787-95.

16. Moseley ME, Kucharczyk J, Mintorovitch J, Cohen Y, Kurhanewicz J, Derugin N, et al. Diffusion-weighted MR imaging of acute stroke: correlation with T2-weighted and magnetic susceptibility-enhanced MR imaging in cats. AJNR Am J Neuroradiol. 1990;11(3):423-9.

17. Warach S, Gaa J, Siewert B, Wielopolski P, Edelman RR. Acute human stroke studied by whole brain echo planar diffusion-weighted magnetic resonance imaging. Ann Neurol. 1995;37(2):231-41.

18. Gonzalez RG, Schaefer PW, Buonanno FS, Schwamm LH, Budzik RF, Rordorf G, et al. Diffusion-weighted MR imaging: diagnostic accuracy in patients imaged within 6 hours of stroke symptom onset. Radiology. 1999;210(1):155-62.

19. Tsuruda JS, Chew WM, Moseley ME, Norman D. Diffusion-weighted MR imaging of the brain: value of differentiating between extraaxial cysts and epidermoid tumors. AJR Am J Roentgenol. 1990;155(5):1059-65; discussion 66-8.

20. Maier SE, Sun Y, Mulkern RV. Diffusion imaging of brain tumors. NMR Biomed. 2010;23(7):849-64.

21. Liu AY, Maldjian JA, Bagley LJ, Sinson GP, Grossman RI. Traumatic brain injury: diffusion-weighted MR imaging findings. AJNR Am J Neuroradiol. 1999;20(9):1636-41.

22. Hergan K, Schaefer PW, Sorensen AG, Gonzalez RG, Huisman TA. Diffusion-weighted MRI in diffuse axonal injury of the brain. Eur Radiol. 2002;12(10):2536-41.

23. Basser PJ, Mattiello J, LeBihan D. MR diffusion tensor spectroscopy and imaging. Biophys J. 1994;66(1):259-67.

24. Tuch DS, Reese TG, Wiegell MR, Makris N, Belliveau JW, Wedeen VJ. High angular resolution diffusion imaging reveals intravoxel white matter fiber heterogeneity. Magn Reson Med. 2002;48(4):577-82.

25. Tuch DS. Q-ball imaging. Magn Reson Med. 2004;52(6):1358-72.

26. Wedeen VJ, Hagmann P, Tseng WY, Reese TG, Weisskoff RM. Mapping complex tissue architecture with diffusion spectrum magnetic resonance imaging. Magn Reson Med. 2005;54(6):1377-86.

27. Dyrby TB, Sogaard LV, Hall MG, Ptito M, Alexander DC. Contrast and stability of the axon diameter index from microstructure imaging with diffusion MRI. Magn Reson Med. 2012.

28. Huang SY, Nummenmaa A, Witzel T, Duval T, Cohen-Adad J, Wald LL, et al. The impact of gradient strength on in vivo diffusion MRI estimates of axon diameter. Neuroimage. 2015;106:464-72.

29. Setsompop K, Kimmlingen R, Eberlein E, Witzel T, Cohen-Adad J, McNab JA, et al. Pushing the limits of in vivo diffusion MRI for the Human Connectome Project. Neuroimage. 2013;80:220-33.

30. Van Essen DC, Ugurbil K, Auerbach E, Barch D, Behrens TE, Bucholz R, et al. The Human Connectome Project: a data acquisition perspective. Neuroimage. 2012;62(4):2222-31.

31. Van Essen DC, Smith SM, Barch DM, Behrens TE, Yacoub E, Ugurbil K, et al. The WU-Minn Human Connectome Project: an overview. Neuroimage. 2013;80:62-79.

32. Fan Q, Nummenmaa A, Witzel T, Zanzonico R, Keil B, Cauley S, et al. Investigating the capability to resolve complex white matter structures with high b-value diffusion magnetic resonance imaging on the MGH-USC Connectom scanner. Brain Connect. 2014;4(9):718-26.

33. Stikov N, Campbell JSW, Stroh T, Lavelee M, Frey S, Novek J, et al. In vivo measurement of the myelin g-ratio with histological validation. NeuroImage 2015;118:397–405.

34. McNab JA, Edlow BL, Witzel T, Huang SY, Bhat H, Heberlein K, et al. The Human Connectome Project and beyond: initial applications of 300 mT/m gradients. Neuroimage. 2013;80:234-45.

35. Huang SY, Tobyne SM, Nummenmaa A, Witzel T, Wald LL, McNab JA, et al. Characterization of Axonal Disease in Patients with Multiple Sclerosis Using High-Gradient-Diffusion MR Imaging. Radiology. 2016:151582.

36. Zhang H, Schneider T, Wheeler-Kingshott CA, Alexander DC. NODDI: practical in vivo neurite orientation dispersion and density imaging of the human brain. Neuroimage. 2012;61(4):1000-16.

37. Schneider T, Brownlee W, Zhang H, Ciccarelli O, Miller DH, Wheeler-Kingshott CA. Application of multi-shell NODDI in multiple sclerosis. Proceedings of the 22nd Annual Meeting of the ISMRM., 2014. Milan, Italy.

38. Waxman SG, Kocsis JD, Stys PK. The Axon: Structure, Function and Pathophysiology. New York: Oxford University Press; 1995.

39. Stikov N, Perry LM, Mezer A, Rykhlevskaia E, Wandell BA, Pauly JM, et al. Bound pool fractions complement diffusion measures to describe white matter micro and macrostructure. Neuroimage. 2011;54(2):1112-21.

40. Stuber C, Morawski M, Schafer A, Labadie C, Wahnert M, Leuze C, et al. Myelin and iron concentration in the human brain: a quantitative study of MRI contrast. Neuroimage. 2014;93 Pt 1:95-106.

41. Mezer A, Yeatman JD, Stikov N, Kay KN, Cho NJ, Dougherty RF, et al. Quantifying the local tissue volume and composition in individual brains with magnetic resonance imaging. Nat Med. 2013;19(12):1667-72.

42. Edzes HT, Samulski ET. Cross relaxation and spin diffusion in the proton NMR or hydrated collagen. Nature. 1977;265(5594):521-3.

43. Wolff SD, Balaban RS. Magnetization transfer contrast (MTC) and tissue water proton relaxation in vivo. Magn Reson Med. 1989;10(1):135-44.

44. Schmierer K, Scaravilli F, Altmann DR, Barker GJ, Miller DH. Magnetization transfer ratio and myelin in postmortem multiple sclerosis brain. Ann Neurol. 2004;56(3):407-15.

45. Filippi M, Campi A, Dousset V, Baratti C, Martinelli V, Canal N, et al. A magnetization transfer imaging study of normal-appearing white matter in multiple sclerosis. Neurology. 1995;45(3 Pt 1):478-82.

46. Sled JG, Pike GB. Quantitative imaging of magnetization transfer exchange and relaxation properties in vivo using MRI. Magn Reson Med. 2001;46(5):923-31.

47. Henkelman RM, Stanisz GJ, Graham SJ. Magnetization transfer in MRI: a review. NMR Biomed. 2001;14(2):57-64.



Proc. Intl. Soc. Mag. Reson. Med. 24 (2016)