Alexander Funk1, Chalermchai Khemtong1, Nesmine Maptue1, Dean Sherry1,2, and Craig Malloy1,3
1UT Southwestern Medical Center, Dallas, TX, United States, 2UT Dallas, Richardson, TX, United States, 3Veterans Affairs North Texas Healthcare System, Dallas, TX, United States
Synopsis
The
effects of deuteration in pyruvate on conversion to alanine and lactate, and
flux into the TCA cycle were investigated by 13C NMR of tissue
extracts from isolated hearts and studies of hyperpolarized [1-13C1,
3,3,3-2H3]pyruvate. No kinetic isotope effects were
noted, but substantial exchange of water protons with deuterium was observed in
alanine but not lactate or glutamate. Deuteration of pyruvate had little effect
on metabolism in heart tissue, and the data suggests that formation of a Schiff
base in alanine transaminase may occur without net interconversion of pyruvate
and alanine.
Introduction
In
recent years, deuteration of metabolites for hyperpolarization (HP) experiments
has become increasingly popular. Since the T1 decay of 13C
polarization limits the duration of data acquisition, incorporation of 2H
prolongs the T1 of 13C in some molecules, enabling a
longer period of observation.[1,2] In particular,
perdeuterated glucose and perdeuterated pyruvate have been of interest.[3,4] However, 2H
can influence fluxes in some metabolic pathways. The addition of the 2H
label introduces further isotopomers that allows observation of more additional
biochemical information, including the exchange processes between pyruvate,
lactate (via LDH) and alanine (via ALT) as well as the kinetic isotope effects
into pyruvate dehydrogenase (via PDH) (Figure 1). The 2H isotopomers
are distinguishable due to chemical shift isotope effects in the 13C
NMR spectrum. The conversion of pyruvate to alanine has shown a de-deuteration
reaction in previous experiments of isolated cells, which is also expected
here.[5]Methods
Three set of isolated rat heart perfusions
were performed to investigate the behavior of per-deuteration on metabolism.
Set 1 contained a 1:1 mixture of [2-13C1,3,3,3-2H3]pyruvate
and [U-13C3]pyruvate as an internal control and were
perfused for 30 minutes to steady state. A control set contained [2-13C1]pyruvate.
Set 2 were perfused with just [2-13C1,3,3,3-2H3]pyruvate
for 3 or 6 minutes respectively. Set 3 were injected with hyperpolarized [1-13C1,3,3,3-2H3]pyruvate
or [1-13C1]pyruvate as a control. Set 1 and 2 were
analyzed as tissue extracts by 13C{1H} NMR, while set 3
were obtained directly over time after injection.Results
The first set of perfusions showed no
difference in the relative rates between metabolites derived from [2-13C1,3,3,3-2H3]pyruvate
and [U-13C3]pyruvate, indicating no kinetic isotope
effect is present in the metabolism of pyruvate to lactate, alanine or
glutamate (Table 1). However, a substantial de-deuteration is observed for
alanine but not lactate (Figure 2). The second set of perfusions further
highlighted the de-deuteration of alanine. By supplying the heart with [2-13C1,3,3,3-2H3]pyruvate
on the time scale of a hyperpolarization experiment, it was possible to observe
the rate of de-deuteration between 3 and 6 minutes. In fact, the stage of
de-deuteration of alanine at 6 minutes is almost identical to the distribution
at 30 minutes, indicating that the de-deuteration happens mostly during the HP
time course. A final set of experiments was performed to investigate the direct
effect of per-deuteration on metabolism by injection of hyperpolarized [1-13C1,3,3,3-2H3]pyruvate.
Even though the C1 resonances observed in the HP experiment is 3 bonds away,
there is a small chemical shift isotope effect (Figure 3). The alanine
resonance is broadened and actually shows a small shift change over time, due
to the distribution of 2H isotopomers changing with the
de-deuteration reaction happening in real time. Furthermore, the lactate to
bicarbonate ratio was unaffected compared to a control, indicating no change in
PDH flux and therefore, confirming the lack of kinetic isotope effects that
were observed in the tissue extract experiments.Discussion
Previous studies using per-deuterated
glucose found significant kinetic isotope effects into alanine and glutamate. Since
pyruvate is the product of glycolysis, our finding that there is little kinetic
isotope effect of deuteration of exogenous pyruvate is somewhat unexpected.
However, earlier studies have reported compartmentation of glycolytic
metabolism separate from metabolism of exogenous pyruvate in the cytosol.[5,6] The de-deuteration of the metabolites
observed here allowed insight into the mechanism of ALT. Traditionally; it is
assumed that there is a linear mechanism from pyruvate to alanine, via a Schiff
base intermediate. These data suggest, however, that the Schiff base reaction allows
multiple de-deuteration per conversion of pyruvate to alanine (Figure 4).
Additionally, it is believed that the availability of just one iso-enzyme of
ALT in heart tissue allows the de-deuterated species to be limited to alanine
and not exchange back to pyruvate and lactate.[4,7]Conclusions
Overall,
per-deuteration of pyruvate does not affect the relative rates of metabolism in
isolated hearts, and, therefore, HP experiments with per-deuterated pyruvate
are a valid alternative. However, de-deuteration can complicate analysis and broaden
resonances in hyperpolarization experiments and other tissue types might
experience Acknowledgements
This project was supported by NIH P41EB015908.References
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