Agnese Tamanti1, Anna Isabella Pisani1, Alberto De Luca2, Francesca Benedetta Pizzini3, Marco Castellaro4, Carmela Zuco1, Damiano Marastoni1, Francesco Crescenzo1, Alessandra Bertoldo4,5, Marco Pitteri1, Roberta Magliozzi1,6, and Massimiliano Calabrese1
1Department of Neurosciences, Biomedicine and Movement sciences, University of Verona, Verona, Italy, 2University Medical Center, Image Sciences Institute, Utrecht, Netherlands, 3Neuroradiology Unit, Department of Diagnostics and Pathology, Verona University Hospital, Verona, Italy, 4Padova Neuroscience Center, University of Padua, Padova, Italy, 5Department of Information Engineering, University of Padua, Padova, Italy, 6Division of Brain Sciences, Faculty of Medicine, Imperial College London Hammersmith Hospital, London, United Kingdom
Synopsis
Neurite Orientation
Dispersion and Density Imaging (NODDI) is a diffusion weighted MRI technique
introduced to asses neuronal microstructure. NODDI is used to retrieve indexes
such as neurite density and fibers orientation dispersion that might be useful
to assess neuronal damage and demyelination in multiple sclerosis (MS) MRI
images. For this reason, we investigate its ability to differentiate to
different MS phenotypes and clinical features in white matter and in multiple
areas of the grey matter.
Introduction
Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease
causing neurological disability1. There
are several phenotypes of MS depending on the symptoms and the pattern of
progression of the disease: relapsing-remitting (RR) MS is characterized by the
unpredictable alternance of periods of high disease activity and of relative
quiescence while primary progressive (PP) MS, instead, presents disability
progression from the first symptoms appearance without remission periods. Differences
in the disease phenotype and in the clinical outcome of the patients might be
related to microstructural changes in brain tissue. Neurite Orientation
Dispersion and Density Imaging (NODDI)2 is a novel diffusion weighted (DW) MRI technique
introduced to assess the neuronal microstructure morphology changes in a
clinically feasible data acquisition setting. Since only a few studies3 investigated the use of this method principally
in spinal cord3 and white matter4 (WM) of multiple sclerosis (MS) patients, we explored
the differences between NODDI values in the grey matter (GM) and WM of RRMS and
PPMS as well as their association with clinical features such as disease
duration and Expanded Disability Status Scale (EDSS).Methods
NODDI models the
diffusion signal in each voxel as the contribution of three independent
compartments: isotropic, intra-neurite and extra neurite compartment as in the
following formula:
$$A=(1-F_{iso})(F_{ic}A_{ic}+(1-F_{ic})A_{ec})+F_{iso}A_{iso}$$
Where $$$A_{ic}$$$, $$$A_{iso}$$$ and $$$A_{ec}$$$ are the intra-cellular, isotropic and
extracellular normalized signal fractions and $$$F_{ic}$$$,$$$F_{iso}$$$ and $$$F_{ec}$$$ the corresponding
volume fractions2.
This model was fitted
on data acquired from 169 RR and 21 PP patients of the MS Centre of Verona University
Hospital (dataset described in fig.1) to retrieve the
images of three tissue indices: Neurite
Density Index (NDI), Orientation Dispersion Index (ODI), and isotropic
diffusion volume fraction (Fiso), where:
- Fiso accounts for the water
free to move such as the interstitial water and Cerebrospinal Fluid (CSF)
- NDI represents the density of neurites
estimated from the Fic which represents the water contained in
highly anisotropic structures, such as dendrites and axons.
- ODI indicates the level of dispersion of
the neurites, low in areas of very coherent fibers, such as in the white
matter, and close to 1 where the fibers are very dispersed.
Data acquisition
was based on a double shell DW-MRI sequence with 8 non-weighted volumes, 32
gradient directions at b=700s/mm2 and 64 gradients at b=2000s/mm2 for an acquisition time of 20
min. DW-MRI images were preprocessed with Tortoise using a T2w sequence for reference to reduce motion, eddy
currents and EPI artifacts.
83 Regions Of Interest
(ROIs) were delineated from the T1w data using the Multi Atlas Segmentation
with joint Label Fusion (MALF) technique and the Advanced Normalization
Tool (ANTs). The T1w data were acquired using a 3D Fast Field Echo with TE/TR
3.7/8.4ms and resolution 1x1x1mm3.
Results
PPMS differed from RRMS in several
NODDI parameters: ODI was significantly greater in the PPMS than in the RRMS in
both the left and the right WM (p<0.02), in the left and the right Hippocampus
(p<0.001, p<0.05, respectively) and in the left and the right Insula
(p<0.03, p<0.001, respectively). In addition, ODI was significantly
lower in the left Putamen (p<0.01) among PPMS patients, compared to the
RRMS (fig.2). $$$F_{iso}$$$ was found to be significantly greater in several cortical regions in
the PPMS, compared to the RRMS and in RRMS with long disease duration (>10
years) compared to early RRMS (< 5 years), including in the Cingulate Gyrus,
the Hippocampus, the Thalamus and the Insula (p<0.001) (fig.3). Finally, among RR
patients, stratified by age (≤25,25-39,>39
years), we observed a negative correlation between EDSS and NDI of the left WM,
in the last two age groups only(fig.4).Discussion and Conclusion
Alterations in PPMS
compared to RRMS were observed both in WM and in GM.
Furthermore,
significant difference between RRMS with long disease duration compared to
early RRMS have been detected. Even if further studies are required to
investigate the relation between NODDI and clinical parameters, these data show
that NODDI may represent a sensible tool to assess differential brain microstructure
alterations related to different disease course and evolution.Acknowledgements
No acknowledgement found.References
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