Zheng Zhong1,2, Muge Karaman1,2, Kaibao Sun1, and Xiaohong Joe Zhou1,2,3
1Center for MR Research, University of Illinois at Chicago, Chicago, IL, United States, 2Department of Bioengineering, University of Illinois at Chicago, Chicago, IL, United States, 3Department of Radiology and Neurology, University of Illinois at Chicago, Chicago, IL, United States
Synopsis
Aging is
considered a major factor in the development of neurodegenerative disease. The aging
process can result in brain tissue microstructural alterations, particularly in
regions relevant to neurodegeneration, such as the substantia nigra (SN). In
this study, we employed a non-Gaussian diffusion model – the continuous-time
random-walk (CTRW) model – together with a high-resolution diffusion
acquisition technique to investigate the possible microstructural changes in
the SN in normal aging. Two CTRW model parameters have exhibited significant
differences in the SN between young and elderly healthy human subjects.
Introduction
Aging
is considered a major factor in the development of neurodegenerative diseases such as
Parkinson’s Disease, which is characterized by the progressive degeneration of dopaminergic
neurons in the substantia nigra (SN). Studies on adult mice as well as on human have suggested
structural changes in the SN with aging1,2. The structural changes can be
potentially detected using diffusion-weighted imaging (DWI), which has emerged
as an important tool to study tissue microstructures with both Gaussian and
non-Gaussian models. Although the Gaussian model is widely used, its assumption
of Gaussian distribution of diffusion displacement breaks down when b-values become higher (e.g. >1500
s/mm2
in human brain tissues). As a result, a number of non-Gaussian diffusion models
have been proposed, each with its own potential in revealing different aspects
of tissue microstructure and
microenvironment3–5. In this study, we employ a non-Gaussian
diffusion model – the continuous-time random-walk (CTRW) model – together with
a high-resolution diffusion acquisition technique to investigate the possible
microstructural changes in the SN in normal aging.Materials and Methods
Subjects:
With IRB approval, 18 elderly and 15 young
healthy subjects were recruited in the study. The characteristics of all subjects
are summarized in Table 1.
Image Acquisition:
All subjects underwent diffusion MRI on a
3T GE MR750 scanner. Diffusion images were acquired from the brainstem using a
customized reduced field-of-view sequence6 with seven b-values: 04, 502,
2002, 5004, 10004, 20004, and 30004
s/mm2
(the subscript denotes NEXs). The other
parameters were: TR/TE=3080/86ms, slice thickness=3mm, FOV=10cm×6cm, and matrix
size=160×96, producing a voxel size of 0.6×0.6×3mm3. At each non-zero b-value, trace-weighted images were obtained to minimize the effect
of diffusion anisotropy.
Image and Statistical Analysis:
The
CTRW model describes the diffusion-weighted (DW) signal using a Mittag-Leffler
function Eα:
$$M(b)=M_{0}E_{\alpha}(-(bD_{m})^{\beta}), [1] $$ where Dm
is an anomalous diffusion coefficient, and α and β are the temporal
and spatial diffusion heterogeneity parameters, respectively7. Eq. [1] was used to fit
the multi-b-value diffusion images
voxel-by-voxel, producing maps of Dm,
α and β. For comparison, apparent diffusion coefficient (ADC) was also
computed using b-values of 50 and
1000 s/mm2.
For
each subject, regions-of-interest (ROIs) were drawn on each side of the SN as
well as in the
tegmentum which served as an internal reference to
exclude other confound factors8. The mean value of each
diffusion parameter within the ROIs was calculated and compared between the young
and the elderly groups using a 2-tailed Student’s t-test.
Results
The first row of Figure 2 shows representative
diffusion-weighted images at b=1000 s/mm2
from a young (left) and an elderly subject (right), where structures in the
brainstem were resolved with high spatial resolution (voxel size=0.6×0.6×3 mm3).
The remaining rows in Figure 2 illustrate a set of maps of Dm, α, β, and ADC obtained from the same subjects. In group analysis, the tegmentum
(i.e, the reference) did not exhibit significant difference between the young
and elderly groups in any of the four diffusion parameters (Figure 3, p>0.1). In contrast, in the SN significant
differences were observed in α and
Dm (p=0.03) between the two groups,
as shown in Figure 4. Discussion and Conclusion
With the use
of a non-Gaussian diffusion model together with a high-resolution DWI
acquisition technique, we have found significant differences in the SN between
the young and elderly healthy subjects. The difference was observed in Dm, but not in ADC, possibly due to the increased sensitivity
when non-Gaussian diffusion behaviors of water molecules accounted for. The
decreased Dm can be explained by hypertrophy of
the neurons in the SN with aging2 as well as the presence of age-related
gliosis and microglia9. α and β have been associated with the temporal and spatial diffusion
heterogeneity, respectively7,8. The decreased α in the SN observed in the elderly group suggests that water
molecules are diffusing through an increasingly more heterogeneous environment.
Both decreased Dm and α likely reflect specific aspects of tissue microstructural changes
associated with aging. With histology correlation in future studies, the exact
microstructural change may be identified. In conclusion, high b-value DWI with the CTRW model has
provided evidence of age-related alterations in the SN of healthy human subjects.Acknowledgements
This work was supported in part by NIH 1S10RR028898 and
R01EB026716. We thank Michael Flannery and Hagai Ganin for technical
assistance. References
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