Yongxian Qian1, Tiejun Zhao2, Kathik Lakshmanan1, Timothy Shepherd1, Yulin Ge1, Yvonne W. Lui1, and Fernando E. Boada1
1Radiology, New York University, New York, NY, United States, 2Siemens Healthineers USA, New York, NY, United States
Synopsis
The literature reports a wide variance in CSF T2*
values (46-64ms). This variance may suggest T2* heterogeneity of CSF.
Here we explore the possibility of CSF T2* heterogeneity among healthy and neurologically-disordered
brains.
INTRODUCTION
In sodium (23Na) MRI of the brain, cerebrospinal
fluid (CSF) is considered a major source of signal contamination that degrades quantification
of sodium concentration in the brain parenchyma and limits potential clinical
utility in a host of neurological disorders with ionic abnormalities such as
concussion (or mild traumatic brain injury, mTBI), epilepsy, bipolar disorder, and
multiple sclerosis (MS). Current methods are limited in eliminating CSF signal when
applied in a clinical setting at 3T due to the difficulty in reliably distinguishing
between CSF and brain signals. Triple quantum filtering (TQF)1,2 was
pursued as an ideal technique that eliminates signals from sodium ions at free-motion,
including those in CSF. However, TQF approach suffers from low signal-to-noise
ratio (SNR) when applied to human brains at 3T. Inversion recovery3,4,
an alternative to TQF technique, takes advantage of difference in T1 relaxation
time between CSF and brain to null CSF signal. However, it inevitably decreases
brain signal as well and complicates sodium quantification. A new technique was
recently reported that has the potential to separate CSF signal from the brain using
multiple-TE acquisitions5,6, however, it requires prior knowledge of
T2* value in CSF. The literature reports a wide variance in CSF T2* values (46-64ms)7-9.
This variance may suggest T2* heterogeneity of CSF. Here we explore the possibility
of CSF T2* heterogeneity among healthy and neurologically-disordered brains.METHODS
Sodium ions in CSF are in fast and free motion
and thus have a mono-exponential T2* relaxation (Eq. 1)10. If sodium
signal, y(t), is measured at two echo times of TE1 and TE2,
T2* value is estimated via a curve fitting in Eq. 2. The distractive variation
of T2* value with B0 field inhomogeneity was minimized through a correction in
Eq. 3. The B0 field map was attained using the phase difference between the two
TE images5. CSF was selected in lateral ventricles (left and right)
for the T2* measurement. Multiple neighboring slices were used to define the extension
of ventricles. T2* value was calculated voxel-by-voxel and then averaged over a
CSF volume of voxels 70±50 in a range of 16-169.
Eq. [1]. $$$ y(TE)=Aexp(-TE/T_2^*) $$$
Eq. [2]. $$$ T_2^*=(TE_2-TE_1)/ln(y_1/y_2) $$$
Eq. [3]. $$$ 1/T_{2c}^*=1/T_2^*-\gamma|\delta B_0| $$$
EXPERIMENTS
Sodium MRI scans were performed on a clinical
scanner at 3T (MAGNETOM Prisma, Siemens Healthineers, Erlangen, Germany) with a
custom-built 8-channel dual-tuned 1H-23Na Tx/Rx head coil11.
Nineteen subjects (6 mTBI, 3 epilepsy, 2 bipolar disorder,1 MS, and 7 healthy
controls; age 32±13 years in 18-70 years;
7/12 Male/Female) were scanned, with the approved IRB and written consent. A custom-developed
sequence, the twisted projection imaging (TPI)12, was used for the data
acquisition with FOV=220mm, matrix size=64, 3D isotropic, TR=100ms, TE1/TE2=0.3/5ms,
q=90°, averages=4, p=0.4, and TA =10min28sec per a TE
imaging. The image reconstruction was performed offline with custom-developed
programs in C++ (MS Visual Studio 2012, Redmond, WA), while the T2* and B0 map
calculations in MATLAB (MathWorks, Natick, MA).RESULTS AND DISCUSSION
Fig. 1 shows the two-TE sodium images, T2* maps,
and B0 field inhomogeneity for a typical patient with mTBI, where the ventricular
CSF has large T2* values (>30ms) under small inhomogeneity (~5Hz) in the B0 field.
Fig. 2 demonstrates that the studied subjects have a wide range of the
corrected T2* (or T2*c) values (38-56ms), consistent with what has been
reported in the literature7-9. More importantly, it demonstrates a
significant difference between the healthy and mTBI groups (p=0.02) as well as
between the mTBI and other (mixed) patient groups (p=0.04). However, biological
variables such as age and sex did not present any impact on the T2*c value (Figs.
3 and 4).CONCLUSION
Collectively, these results provide a piece of
evidence supporting heterogeneity of ventricular CSF T2* among subjects, especially
after mTBI, independent of age and sex. The physiological basis for this
heterogeneity is unclear at this early stage. In addition, the sample size of
subjects studied in this work is small (n=19) and thus the statistical
significance of the results needs to be further tested in a large pool of
subjects.Acknowledgements
This work was financially
supported in part by NIH grants R01 MH088370, R01 CA111996 and R01NS082436. This
work was also performed under the rubric of the Center for Advanced Imaging
Innovation and Research (CAI2R, www.cai2r.net), an NIBIB Biomedical Technology
Resource Center (NIH P41 EB017183).References
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