Multi-b-valued diffusion-weighted imaging (DWI) of the breast is highly susceptible to image and fitting noise. A multi-compartment approach was developed to denoise multi-b-value breast DWI without spatial smoothing. In human subject exams (N=12), the denoising approach resulted in a significant reduction in variability of all perfusion and diffusion maps in breast tumor and normal fibroglandular tissue with minimal bias to the mean values, and increased statistical separation of diffusivity metrics between tumor and normal tissue. The denoising algorithm provides compartment fractions for tumor, tissue, and vascularity, which may improve visualization of tissue compartments in DWI.
The model-based diffusion denoising method5was proposed with multi-directional-shell neuro-diffusion to provide 3-4 times acceleration and improved diffusivity maps. The method used a generalized, multi-compartment signal model with anisotropic and isotropic tissue compartments, and Richardson-Lucy dampening for optimization:
$$\widehat{s}(b,q)=\sum_{j=1}^J\sum_{n=1}^Nf_{aniso,j,n}exp(-(\lambda_{||}(bq^Tu_n)^2)(-\lambda_{\bot}b(1-(q^Tu_n)^2))+\sum_{j=1}^Jf_{iso}exp(-\lambda_jb)$$
To adapt the method for breast multi-b diffusion where tissue anisotropy is less and diffusion directions are fewer, a model with far fewer compartments and reduced anisotropy was proposed (Table 1), with compartment values approximating normal fibroglandular tissue (NFT), tumors, vascular and fat components as surveyed in the literature1,6-7. As a by-product of the model, the fractional values belonging to each component (ffibroglandular, ftumor, fvascular) could provide visualization of the image signal from each component, as shown in Fig. 1.
Twelve serial multi-b DWI acquisitions from subjects undergoing neoadjuvant chemotherapy for invasive breast cancer (three subjects x 4 time-points) were selected retrospectively for processing. Studies were deidentified per DICOM standard and all subjects gave written informed consent. The acquisitions were obtained from a 1.5T scanner (GE Healthcare, Waukesha WI) with one b=0 acquisition and three-directions per b-value={100,600,800}sec/mm2, FOV=34-35cm, slice thickness=4mm, TR/TE=7500/67.6-67.9ms. Tumor and contralateral NFT were manually segmented on the un-denoised ADC maps. To reduce spatial-bias of diffusivity, gradient nonlinearity correction was applied8-9. The analyzed metrics included: ADC (utilizing b=0 and all b-values), slow ADC (utilizing non-b=0 data), b=100 ADC, and perfusion component fraction10 of b=0. In comparing standard vs denoising, pair-wise, non-parametric Wilcoxon signed-rank test (P<0.05 for statistical significance) was performed on the mean difference and coefficient of variation (CV) of each metric. Two-sample t-test was used to compare metrics between tumor and NFT.
As summarized in Table 2, denoising resulted in very small changes (<±1%) to mean ADC and slow ADC for tumor and NFT; denoising increased b=100 ADC by 5.3-10.2% for both tumor and NFT; denoising increased perfusion fraction as well without statistical significance. In all metrics and in both tumor and NFT, the coefficient of variation (CV) within the ROIs were significantly reduced. In particular, the CV reduction was substantial (15.3-21.2%) in both b=100 and perfusion fraction. These quantitative results could be visualized in Fig. 2, wherein denoising resulted in subtle changes to slow ADC, but dramatic reduction in image texture in both the b=100 ADC and perfusion maps; vascular structures became better visualized.
The differences in diffusivity measures between tumor and NFT were overall increased; t-statistic increased, on average by +1.5 for ADC, +3.2 for slow ADC, +3.8 for b=100 ADC and +0.6 for perfusion fraction. The number of instances with significant differences between tumor and normal tissue was unchanged in ADC (12/12) and in slow ADC (11/12); these were increased in b=100 ADC (from 10/12 to 11/12) and perfusion fraction (from 8/12 to 9/12). These effects are shown on the one subject in Fig. 3.
1. Young Cho, G, Moy L, Kim SG et al. Eur Radiol 2016. 26(8):2547-2558.
2. Jensen JH, Helpern JA, Ramani A et al. Magn Reson Med 2005. 53(6):1432-1440.
3. Haldar JP, Wedeen VJ, Nezamzadeh M et al. Magn Reson Med 2013. 69(1)277-289.
4. Lam F, Babacan SD, Haldar JP et al. Magn Reson Med 2014. 71(3):1272-1284.
5. Sperl JI, Sprenger T, Tan ET et al. Magn Reson Med 2017. 78(6):2428-2438.
6. Partridge SC, Ziadloo A, Murthy R et al. JMRI 2010. 31(2):339-347.
7. Jiang R, Ma Z, Dong H et al. Brit J Radiol 2016. 89:20160076.
8. Malyarenko DI, Ross BD, Chenevert TL. Magn Reson Med 2014. 71(3):1312-1323.
9. Newitt DC, Tan ET, Wilmes LJ et al. JMRI 2015. 42(4):908-919.
10. Lewin M et al. European Radiology 2011.21(2):281-290.