Olli Juhani Nykänen1, Henri Leskinen1, Mikko Finnilä1,2, Sakari Karhula2, Simo Saarakkala2,3, and Mikko J Nissi1,2
1Department of Applied Physics, University of Eastern Finland, Kuopio, Finland, 2Research Unit of Medical Imaging, Physics and Technology, University of Oulu, Oulu, Finland, 3Department of Diagnostic Radiology, Oulu University Hospital, Oulu, Finland
Synopsis
In this study, we investigated the bright signal
feature that has been observed at the bone-cartilage interface using ultrashort
echo time imaging sequences. We used micro-CT imaging of the same specimens to
locate the origin of the signal in SWIFT-MRI images. The results indicated that
the bright signal originates from the deep non-calcified cartilage region
instead of the calcified cartilage, which has been previously hypothesized to be
part of the signal location. The physiological origin of this signal as well as
its role in the diagnosis of osteoarthritis remains to be evaluated.
Introduction
In this study, we investigated the bright signal
that has been observed at the osteochondral interface, i.e. the interface between articular cartilage and subchondral bone,
when imaging specimens with ultra-short echo time sequences (1,2). To investigate
the origins of this signal feature, we obtained high-resolution ultra-short
echo time SWIFT (3) scans
of human osteochondral samples at 9.4T and compared the resulting 3-D MRI data with
native high-resolution 3-D micro-CT data as well as with histologic images of
the same samples.Methods
Cylindrical (dia. = 4 mm) osteochondral
samples (n=8) were obtained from
human cadaver tibia under existing ethical permissions. MRI was performed at
9.4 T Varian/Agilent scanner using a 10-mm-diameter quadrature RF volume
transceiver (Rapid Biomedical GmbH, Rimpar Germany). The samples were immersed
in 1HMRI-signal-free perfluoropolyether inside a plastic test tube. Samples
were oriented in such a way that the surface of the cartilage was approximately
perpendicular to the main magnetic field of the scanner. SWIFT data were
acquired using a bandwidth of 62.5 kHz and a field of view of 3 cm and image
dimensions of 384x384x384 voxels. Number spokes per image was 262144 and the
images were reconstructed using re-gridding on a Cartesian grid. SWIFT images
were obtained by separately applying fat saturation, water saturations and no saturation.
This allowed reconstructing images with both water and fat signal without
radial blurring and images of short T2*-spins. After the MRI scans,
high-resolution micro-CT images were acquired using Skyscan 1272 device (Bruker
microCT, Kontich Belgium) by setting x-ray tube to 50kV and 200 µA and after
filtering with 0.5mm aluminium filter 1200 projections with 2200ms exposure time
and three times frame averaging were collected. Projections were reconstructed
to images with 2.75 µm pixel size with NRecon-software (v.1.6.10.4, Bruker
microCT) with beam hardening and ring corrections. The 3-D MRI images were
co-registered with the 3-D micro-CT images using Elastix –software (4) to allow precise localization of the bright signal feature in the
SWIFT images with respect to the bony features of the specimens. Depth-wise signal
intensities through the cartilage and subchondral bone of the co-registered MRI
and CT images were calculated in Matlab for further analyses. MRI and micro-CT
images were also compared with Masson’s trichrome stained histological slices
to further assess the different structural features of the osteochondral
samples and to evaluate the sources of the bright signal.Results
Out of the eight samples, six exhibited
bright signal at the osteochondral interface and two did not (Fig. 1). All of
the 3-D geometries measured by MRI or micro-CT were successfully co-registered
as indicated by the visually perfect match of the trabecular features between the
3-D MRI and micro-CT datasets (Fig 2). In all of the six samples that exhibited
the bright signal, it was located in the deep articular cartilage, immediately above
the layer of calcified cartilage, but
not in the calcified cartilage (Fig 2).
The layer of calcified cartilage was clearly identified in the histological
slices and was faintly detectable also in the micro-CT images. In general, the layer
of calcified cartilage was seen as tightly interconnected with the subchondral
plate in the micro-CT images (Fig 2).Disscussion and Conclusion
We observed that the bright signal at the osteochondral
interface is not located in the calcified cartilage, contrary to what has been
previously suggested (1,2). Instead,
the bright signal seems to originate from the deep radial layer of articular
cartilage. However, the exact source of the bright signal feature remains yet unsolved.
Possible causes include signal pileup resulting from the susceptibility
difference between the non-calcified cartilage and the subchondral plate
(calcified cartilage and subchondral bone), or very short T1 relaxation time
within the deep layers of non-calcified cartilage. Regardless, it appeared that
even in samples that are spatially close to each other, there are changes in
the appearance of this signal and the potential role of the signal in
osteoarthritis remains to be evaluated. It is also worth noting that in the
samples that did not exhibit the bright signal in deep layer, there was still a
detectable increase in short-T2* signal, located exactly at the interface.Acknowledgements
Support from the Academy of Finland (grants
#285909, #293970 and #319440) and European Research Council (grant # 336267) are
gratefully acknowledged.References
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