Differences in imaging protocols (IP) and post-processing methods (PM) may influence relative cerebral blood volume (rCBV). Our goal was to leverage a dynamic susceptibility contrast (DSC) DRO to characterize rCBV consistency across 12 sites, focusing on differences due to site-specific IPs and/or PMs. Our results indicate high agreement when one center processes rCBV despite slight variations in the IP. However, substantial disagreement was observed when site-specific software was applied for rCBV measurements. These results have important implications for comparing DSC-MRI data across sites/trials, where PM variability could confound the use of rCBV as a biomarker of therapy response.
Multiple IP and PM were reported by sites (Table 1). A variety of software platforms were used: IB Neuro, nordicICE, PGUI, 3D Slicer, Philips IntelliSpace Portal, and in-house processing scripts. Accuracy of each processed rCBV map was first assessed as shown in Figure 1. Phase I results indicate that 17 of 20 analyses demonstrate high rCBV accuracy when processed by the managing center. This illustrates that most of the sites’ IPs are able to accurately compute rCBV – most likely because most sites use a protocol similar to the SIP. Phase II results in 10 of 17 analyses demonstrated high rCBV accuracy when different software choices were used to process the SIP. These software choices included IB Neuro, nordicICE, and in-house processing. Phase III resulted in 12 of 25 combinations of site-specific IP and PM that were able to accurately compute rCBV.
A decrease in agreement was observed when leakage was introduced across all DROs. Phase I had the highest agreement in multisite rCBV (ICCintact-BBB = 0.97; ICCdisrupted-BBB = 0.88). On the other hand, both Phase II (ICCintact-BBB = 0.69; ICCdisrupted-BBB = 0.44) and Phase III (ICCintact-BBB = 0.64; ICCdisrupted-BBB = 0.38) had poor agreement in rCBV across sites. These results indicate that inconsistency in rCBV currently most likely is due to the post-processing methods. The poor agreement across rCBV from the intact-BBB simulations also indicate that variability in pre-processing (e.g. filtering, smoothing) affects rCBV.
For Phase I (Figure 2a), majority of sites have narrow 95% LOAs and results are centered around the mean rCBV compared to the reference. The exceptions to this likely arise from differences in the preload. Two sites showed larger 95% LOA because of differences in TE and injection dose. The LOA widen and exhibit bias for Phase II and III (Fig 2b and c) when PMs are varied. Software that demonstrate the narrowest 95% LOA and no bias are IB Neuro, nordicICE, ISP’s “model-free” option, and in-house processing.
Lastly, Figure 3 illustrates CV% calculated in all voxels for each DRO as a function of the rCBV. In general, the mean CV increases for each phase when more freedom is allowed for both IPs and PMs. The greatest CV% occurs at low rCBV values. This suggests standardization is necessary when voxel-wise analysis (versus hot spot analysis) is performed for detection of early therapeutic effects.
NIH/NCI R01CA213158 (LCB, NS, CCQ)
NIH/NCI U01CA207091 (AJM, MCP)
NIH/NCI U01CA166104 and P01CA085878 (DM, TLC)
NIH/NCI U01CA142565 (CW, AGS, TEY, NR)
NIH/NCI U01CA176110 (KMS, MAP)
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