The NAA/mIns ratio has been proposed as a marker of unhealthy ageing. We found that the NAA/mIns ratio is decreased in the hippocampus for middle-aged and old individuals compared to young individuals. For old individuals, declining NAA/mIns ratios are correlated with poorer performance on a spatial working memory task but this relationship is not found in middle-aged and young individuals. Our results suggest that the NAA/mIns ratio decrease in the hippocampus is observable before the negative functional effects.
The hippocampus is a key age-sensitive brain region, affecting cognitive functions such as spatial working memory1. The N-acetyl aspartate (NAA) /myo-inositol (mIns) ratio has been proposed as a possible biomarker for unhealthy ageing but this has never been investigated in the hippocampus2. The ratio reflects the relationship in the brain between neuronal (NAA) and glial (mIns) integrity and lower levels are suggestive of negative biological developments such as neuroinflammation3. We hypothesised that the NAA/mIns ratio will be lower in the hippocampus with higher age. We further expected this ratio to correlate negatively with spatial working memory.
The preliminary analyses were based on data from 19 young (10 females), 20 middle-aged (10 females) and 19 old (9 females) participants. All measured spectra were of sufficient quality to be included and all CRLBs were below 16. the NAA/mIns ratio significantly differed between groups (F(2,55)=19.77, p<3*10-7) and the posthoc test showed that NAA/mIns ratio was lower in both the old (m=1.07, SD=0.04) and the middle-aged (m=1.11, SD=0.04) individuals as compared to the young individuals (m=1.38, SD=0.04) (Fig. 3). The concentration of mIns was also significantly different across groups (F(2,55)=18.65, p=6*10-7) with higher concentrations in old individuals (m=11.2, SD=0.32) as compared to middle-aged (m=9.98, SD=0.31) and young individuals (m=8.43, SD=0.32); mIns concentrations were also significantly increased in middle-aged individuals as compared to young individuals. No significant between group difference was observed for NAA (F(2,55)=2.38, p=0.1).
The number of errors made in SWM was used as the outcome score. For old individuals, a negative correlation was observed between the NAA/mIns ratio and the SWM error score (p=0.021, R=-0.52) (Fig. 4). No correlations between the NAA/mIns ratio and the SWM error score were observed for the other groups (p>0.05).
1. Valenzuela, M. J. et al.Posterior compensatory network in cognitively intact elders with hippocampal atrophy. Hippocampus25,581–593 (2015).
2. Waragai, M., Moriya, M. & Nojo, T. Decreased N-Acetyl Aspartate/Myo-Inositol Ratio in the Posterior Cingulate Cortex Shown by Magnetic Resonance Spectroscopy May Be One of the Risk Markers of Preclinical Alzheimer’s Disease: A 7-Year Follow-Up Study. J. Alzheimer’s Dis.60,1411–1427 (2017).
3. Chang, L., Munsaka, S. M., Kraft-Terry, S. & Ernst, T. Magnetic resonance spectroscopy to assess neuroinflammation and neuropathic pain. J. Neuroimmune Pharmacol.8,576–593 (2013).
4. Ashburner, J. SPM: A history. Neuroimage62,791–800 (2012).
5. Provencher, S. W. Automatic quantitation of localized in vivo 1H spectra with LCModel. NMR Biomed.14,260–64 (2001).
6. Gasparovic, C. et al.Neurometabolite Concentrations in Gray and White Matter in Mild Traumatic Brain Injury: An 1H–Magnetic Resonance Spectroscopy Study. J. Neurotrauma26,1635–1643 (2009).